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Gamal Rabie Agmy, MD, FCCP
Professor of Chest Diseases, Assiut University
ERS National Delegate of Egypt
The definition of atelectasis is loss of air in the alveoli;
alveoli devoid of air (not replaced).
A diagnosis of atelectasis requires the following:
1-A density, representing lung devoid of air
2-Signs indicating loss of lung volume
Atelectasis
1-Absorption Atelectasis
When airways are obstructed there is no further
ventilation to the lungs and beyond. In the early
stages, blood flow continues and gradually the
oxygen and nitrogen get absorbed, resulting in
atelectasis.
Types of Atelectasis:
2-Relaxation Atelectasis
The lung is held close to the chest wall because of the
negative pressure in the pleural space. Once the
negative pressure is lost the lung tends to recoil due
to elastic properties and becomes atelectatic. This
occurs in patients with pneumothorax and pleural
effusion. In this instance, the loss of negative
pressure in the pleura permits the lung to relax, due
to elastic recoil. There is common misconception that
atelectasis is due to compression.
Types of Atelectasis:
3-Adhesive Atelectasis :
Surfactant reduces surface tension and keeps the
alveoli open. In conditions where there is loss of
surfactant, the alveoli collapse and become
atelectatic. In ARDS this occurs diffusely to both
lungs. In pulmonary embolism due to loss of blood
flow and lack of CO2, the integrity of surfactant
gets impaired.
Types of Atelectasis:
Types of Atelectasis:
4-Cicatricial Atelectasis
–Alveoli gets trapped in scar and
becomes atelectatic in fibrotic
disorders
.
5-Round Atelectasis
An instance where the lung gets trapped by
pleural disease and is devoid of air.
Classically encountered in asbestosis.
Types of Atelectasis:
Generalized
1-Shift of mediastinum: The trachea and heart gets shifted
towards the atelectatic lung.
2-Elevation of diaphragm: The diaphragm moves up and
the normal relationship between left and right side gets
altered.
3-Drooping of shoulder.
4-Crowding of ribs: The interspace between the ribs is
narrower compared to the opposite side.
Signs of Loss of Lung Volume:
Movement of Fissures
You need a lateral view to appreciate the movement of
oblique fissures. Forward movement of oblique fissure in
LUL atelectasis. Backward movement in lower lobe
atelectasis.
Movement of transverse fissure can be recognized in the
PA film.
Signs of Loss of Lung Volume:
Movement of Hilum
The right hilum is normally slightly lower than the left.
This relationship will change with lobar atelectasis.
Signs of Loss of Lung Volume:
Compensatory Hyperinflation
Compensatory hyperinflation as evidenced by increased
radiolucency and splaying of vessels can be seen with the
normal lobe or opposite lung.
Signs of Loss of Lung Volume:
Alterations in Proportion of Left and
Right Lung
The right lung is approximately 55% and left lung 45%. In
atelectasis this apportionment will change and can be a
clue to recognition of atelectasis. .
Signs of Loss of Lung Volume:
Hemithorax Asymmetry
In normals, the right and left hemithorax are equal in size.
The size of the hemithorax will be asymmetrical and
smaller on the side of atelectasis
Signs of Loss of Lung Volume:
Signs of Loss of Lung Volume:
Generalized
Shift of mediastinum: The trachea and heart gets shifted towards the atelectatic lung.
Elevation of diaphragm: The diaphragm moves up and the normal relationship between left
and right side gets altered.
Drooping of shoulder.
Crowding of ribs: The interspace between the ribs is narrower compared to the opposite side.
Movement of Fissures
You need a lateral view to appreciate the movement of oblique fissures. Forward movement of
oblique fissure in LUL atelectasis. Backward movement in lower lobe atelectasis.
Movement of transverse fissure can be recognized in the PA film.
Movement of Hilum
The right hilum is normally slightly lower than the left. This relationship will change with lobar
atelectasis.
Compensatory Hyperinflation
Compensatory hyperinflation as evidenced by increased radiolucency and splaying of vessels
can be seen with the normal lobe or opposite lung.
Alterations in Proportion of Left and Right Lung
The right lung is approximately 55% and left lung 45%. In atelectasis this apportionment will
change and can be a clue to recognition of atelectasis.
Hemithorax Asymmetry
In normals, the right and left hemithorax are equal in size. The size of the hemithorax will be
asymmetrical and smaller on the side of atelectasis
Atelectasis Right Lung
Homogenous density right hemithorax
Mediastinal shift to right
Right hemithorax smaller
Right heart and diaphragmatic silhouette are not identifiable
Atelectasis Left Lung
Homogenous density left hemithorax
Mediastinal shift to left
Left hemithorax smaller
Diaphragm and heart silhouette are not identifiable
Left Lower Lobe Atelectasis
• Inhomogeneous cardiac density
• Left hilum pulled down
• Non-visualization of left diaphragm
• Triangular retrocardiac atelectatic LLL
Atelectasis Left Lower Lobe
Double density over heart
Inhomogenous cardiac density
 Triangular retrocardiac density
Left hilum pulled down
Other findings include:
Pneumomediastinum
Atelectasis Left
Upper Lobe
Mediastinal shift to left
Density left upper lung field
Loss of aortic knob and left hilar
silhouettes
Herniation of right lung
Atelectatic left upper lobe
Forward movement of left
oblique fissure "Bowing sign"
Atelectasis Left Upper
Lobe
Hazy density over left
upper lung field
Loss of left heart
silhouette
Tracheal shift to left
Lateral
A: Forward movement of
oblique fissure
B: Herniated right lung
C: Atelectatic LUL
Lateral
Movement of oblique and transverse
fissures
Atelectasis Right Upper Lobe
Homogenous density right upper lung
field
Mediastinal shift to right
Loss of silhouette of ascending aorta
Lateral
Movement of oblique and transverse
fissures
Atelectasis Right Upper Lobe
Homogenous density right upper lung field
Mediastinal shift to right
Loss of silhouette of ascending aorta
RML Atelectasis
Vague density in right lower lung field, almost normal
RML atelectasis in lateral view, not evident in PA view
Vague density in right lower lung field (almost a normal film).
Dramatic RML atelectasis in lateral view, not evident in PA view. Movement of
transverse fissure.
Other findings include: Azygous lobe
Atelectasis Right Lower Lobe
Density in right lower lung field
Indistinct right diaphragm
Right heart silhouette retained
Transverse fissure moved down
Right hilum moved down
Adhesive Atelectasis
Alveoli are kept open by the integrity of surfactant. When there is loss
of surfactant, alveoli collapse. ARDS is an example of diffuse alveolar
atelectasis.
Plate-like atelectasis is an example of focal loss of surfactant.
Relaxation Atelectasis
The lung is held in apposition to the chest wall because of negative pressure
in the pleura. When the negative pressure is lost, as in pneumothorax or
pleural effusion, the lung relaxes to its atelectatic position. The atelectasis is
a secondary event. The pleural problem is primary and dictates other
radiological findings.
Round Atelectasis
Mass like density
Pleural based
Base of lungs
Blunting of costophrenic angle
Pleural thickening
Pulmonary vasculature curving
into the density
Esophageal surgical clips
Round Atelectasis
Mass like density
Pleural based
Base of lungs
Blunting of costophrenic angle, pleural thickening
Pulmonary vasculature curving into the density
RML Lateral Segment Atelectasis
Sub-segmental Atelectasis
Atelectasis
Segmental
Anterior sub-segment of RUL
"Bronchial wedge"
Hilar Displacement
Bronchiectasis
Left lung atelectasis due to mucus plugging
Mucus plugs suctioned with bronchoscopy
Bronchogram done after bronchoscopy
Saccular bronchiectasis in bronchogram below
Bronchogram
Bronchograms are rarely done nowadays. The need for it
disappeared with the invention of the fiberoptic
bronchoscopy and high resolution CT scan. View these
images to get a greater understanding of a three
dimensional view of a bronchial tree..
Bronchogram
Bronchograms are rarely done nowadays. The need for it disappeared with the
invention of the fiberoptic bronchoscopy and high resolution CT scan.
Calcification
Focal lung lesion: Ghon's complex
Miliary lung calcification:
Histoplasmosis
Tuberculosis
Alveolar microlithiasis
Chicken pox pneumonia
Solitary pulmonary nodule :
Central / Granuloma
Lamellar / Histoplasmosis
Pop corn / Hamartoma
Eccentric / Scar Cancer
Calcification
Nodes:
Homogenous / TB
Clumpy / Histoplasmosis
Egg shell / Silicosis, Sarcoidosis
Tracheal cartilage : Aging
Tumor:
Mediastinal mass / Teratoma
Healed lymphoma / Metstasis
Calcification
Vascular:
Aortic calcification
Pulmonary artery calcification
Pulmonary hypertension
Pleural:
Visceral / Hemothorax, TB, Empyema
Parietal / Asbestosis
Subcutaneous calcification:
Cysticercus
Broncholith
Subsegmental atelectasis
Calcified node
Broncholith obstructing bronchus
Silicosis
Egg shell calcification of lymph nodes
Other findings include:
Diaphragmatic pleural calcification
Multiple cavities with fluid levels
Histoplasmosis
Calcified nodes
Clumpy calcification Calcified nodules in lungs
Hamartoma
Popcorn calcification
Pleural Calcification
Visceral pleural
calcification
Parietal pleura appears
black because it is
sandwiched between
bony densities
Pleural Calcification
Visceral pleura
Old TB
Visceral pleural calcification
Open drainage with air fluid levels in pleural space
Subcutaneous calcification
Cavitary Lung Lesions
Number:
Multiple bilateral cavities would raise
suspicion for either bronchiogenous or
hematogenous process. You should consider:
Aspiration lung abscess
Septic emboli
Metastatic lesions
Vasculitis (Wegener's)
Coccidioidomycosis, tuberculosis
Location:
• Classical locations for aspiration lung abscess
are superior segment of the lower lobes
posterior segments of upper lobes.
• Tuberculous cavities are common in superior
segments of upper and lower lobes or posterior
segments of upper lobes.
• When a cavity in anterior segment is
encountered, a strong suspicion for lung cancer
should be raised. TB and aspiration lung
abscess are rare in anterior segments. Cancer
lung can occur in any segment.
Wall Thickness:
• Thick walls are seen in:
– Lung abscess
– Necrotizing squamous cell lung cancer
– Wegener's granulomatosis
– Blastomycosis
Wall Thickness:
• Thin walled cavities are seen in:
• Coccidioidomycosis
• Metastatic cavitating squamous cell
carcinoma from the cervix
• M. Kansasii infection
• Congenital or acquired bullae
• Post-traumatic cysts
• Open negative TB
Contents:
• The most common cause for air fluid level is
lung abscess. Air fluid levels can rarely be
seen in malignancy and in tuberculous
cavities from rupture of Rasmussen's
aneurysm.
• A fungous ball should make you consider
aspergillosis. A blood clot and fibrin ball will
have the same appearance.
• Floating Water Lily: The collapsed membrane
of a ruptured echinococcal cyst, floats giving
this appearance.
Lining of Wall:
The wall lining is irregular and nodular in
lung cancer or shaggy in lung abscess
Evolution of Lesion:
Many times review of old films to assess the
evolution of the radiological appearance of
the lesion extremely helpful. Examples
• Infected bullae
• Aspergilloma
• Sub acute necrotizing aspergillosis
• Bleeding from Rasmussen's aneurysm in a
tuberculous cavity
Associated Features:
Ipsilateral lymph nodes or lytic
lesions of the bone is seen
with malignancy
Bulla
<1mmwall
>1cmsize
Pneumatocele
<1mmwall
staph.infection
Honeycombing
<1cmsize
multipleequal
Cyst
1-3mmwall
1-10cmsize
Cavity
>3mmwall
Anysize
Cavitarylesionsoflung
Bulla
Definition
•Thin-walled–less than 1 mm
•Air-filled space
•In the lung> 1 cm in size and up to 75% of lung
•Walls may be formed by pleura, septa,
or compressed lung tissue.
•Results from destruction, dilatation and
confluence of airspaces distal to terminal
bronchioles.
•Bullous disease may be primary or associated
with emphysema or interstitial lung disease.
• Primary bullous lung disease may be familial
and has been associated with Marfan's, Ehler's
Danlos, IV drug users, HIV infection, and
vanishing lung syndrome.
•Bullae may occasionally become very large
and compromise respiratory function. Thus
has been referred as vanishing lung syndrome,
and may be seen in young men.
Upper lobe Bulla
Lower lobe Bulla
A: Xray shows bilateral bulla.
B: CT shows bilateral bulla.
C: CT after bullectomy.
Pneumatocele is a benign air containing cyst of lung, with
thin wall < 1mm as bulla but with different mechanism 
Infection with staph aureus is the commonest cause ( less
common causes are, trauma, barotrauma) lead to necrosis
and liquefaction followed by air leak and subpleural
dissection forming a thin walled cyst.
•Honeycombing is defined as multiple cysts < 1cm in diameter,with
well defined walls, in a background of fibrosis, tend to form
clusters and is considered as end stage lung .
•It is formed by extensive interstitial fibrosis of lung with residual
cystic areas.
A cyst is a ring
shadow > 1 cm in
diameter and up to
10 cm with wall
thickness from 1-3
mm.
Thin walled cysts of LAM
A cavity is > 1cm
in diameter, and its
wall thickness is
more than 3 mm.
•A central portion  necrosis and communicate to bronchus.
•The draining bronchus is visible (arrow). CT (2 mm slice thickness)
shows discrete air bronchograms in the consolidated area.
Mechanism
1. Site
A cavity in apicoposterior segment of left upper lobe
2.Number
Multiple cavities:
1. Aspiration.
2. TB
3. Fungal.
4. Metastatic.
5. Septic emboli.
6.Wegners granulomatosis
Multiple cysts of metastasis
from squamous cell
carcinoma.
Multiple thick wall cavities from
adenocarcinoma of right lung
Irregular , nodular inner lining of thick wall abscess
Malignant cavity.
3. Thickness and
irregularity
4. eccentric
Malignant
5. Relation to lymph
node enlargement
6. Contents
•Arrow head  Crescent sign.
•Black arrows  Fibrotic bands surrounding cavity
(Fibrocavitary TB).
Primary Lung Cancer
• Thick wall
• Shaggy lumen
• Eccentric cavitation
|
Squamous Cell Carcinoma Lung
LUL mass
Thick walled cavity
Eccentric location of cavity
Fungous Ball
Long standing cavity
Containing round density (A)
Mobile density
Adjacent pleural reaction (B) - characteristic of aspergilloma
Cavitating Metastasis
MultipleThin Walled Cavities
Cancer Cervix

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Chest radiology part 2

  • 1.
  • 2. Gamal Rabie Agmy, MD, FCCP Professor of Chest Diseases, Assiut University ERS National Delegate of Egypt
  • 3. The definition of atelectasis is loss of air in the alveoli; alveoli devoid of air (not replaced). A diagnosis of atelectasis requires the following: 1-A density, representing lung devoid of air 2-Signs indicating loss of lung volume Atelectasis
  • 4. 1-Absorption Atelectasis When airways are obstructed there is no further ventilation to the lungs and beyond. In the early stages, blood flow continues and gradually the oxygen and nitrogen get absorbed, resulting in atelectasis. Types of Atelectasis:
  • 5. 2-Relaxation Atelectasis The lung is held close to the chest wall because of the negative pressure in the pleural space. Once the negative pressure is lost the lung tends to recoil due to elastic properties and becomes atelectatic. This occurs in patients with pneumothorax and pleural effusion. In this instance, the loss of negative pressure in the pleura permits the lung to relax, due to elastic recoil. There is common misconception that atelectasis is due to compression. Types of Atelectasis:
  • 6. 3-Adhesive Atelectasis : Surfactant reduces surface tension and keeps the alveoli open. In conditions where there is loss of surfactant, the alveoli collapse and become atelectatic. In ARDS this occurs diffusely to both lungs. In pulmonary embolism due to loss of blood flow and lack of CO2, the integrity of surfactant gets impaired. Types of Atelectasis:
  • 7. Types of Atelectasis: 4-Cicatricial Atelectasis –Alveoli gets trapped in scar and becomes atelectatic in fibrotic disorders
  • 8. . 5-Round Atelectasis An instance where the lung gets trapped by pleural disease and is devoid of air. Classically encountered in asbestosis. Types of Atelectasis:
  • 9. Generalized 1-Shift of mediastinum: The trachea and heart gets shifted towards the atelectatic lung. 2-Elevation of diaphragm: The diaphragm moves up and the normal relationship between left and right side gets altered. 3-Drooping of shoulder. 4-Crowding of ribs: The interspace between the ribs is narrower compared to the opposite side. Signs of Loss of Lung Volume:
  • 10. Movement of Fissures You need a lateral view to appreciate the movement of oblique fissures. Forward movement of oblique fissure in LUL atelectasis. Backward movement in lower lobe atelectasis. Movement of transverse fissure can be recognized in the PA film. Signs of Loss of Lung Volume:
  • 11. Movement of Hilum The right hilum is normally slightly lower than the left. This relationship will change with lobar atelectasis. Signs of Loss of Lung Volume:
  • 12. Compensatory Hyperinflation Compensatory hyperinflation as evidenced by increased radiolucency and splaying of vessels can be seen with the normal lobe or opposite lung. Signs of Loss of Lung Volume:
  • 13. Alterations in Proportion of Left and Right Lung The right lung is approximately 55% and left lung 45%. In atelectasis this apportionment will change and can be a clue to recognition of atelectasis. . Signs of Loss of Lung Volume:
  • 14. Hemithorax Asymmetry In normals, the right and left hemithorax are equal in size. The size of the hemithorax will be asymmetrical and smaller on the side of atelectasis Signs of Loss of Lung Volume:
  • 15. Signs of Loss of Lung Volume: Generalized Shift of mediastinum: The trachea and heart gets shifted towards the atelectatic lung. Elevation of diaphragm: The diaphragm moves up and the normal relationship between left and right side gets altered. Drooping of shoulder. Crowding of ribs: The interspace between the ribs is narrower compared to the opposite side. Movement of Fissures You need a lateral view to appreciate the movement of oblique fissures. Forward movement of oblique fissure in LUL atelectasis. Backward movement in lower lobe atelectasis. Movement of transverse fissure can be recognized in the PA film. Movement of Hilum The right hilum is normally slightly lower than the left. This relationship will change with lobar atelectasis. Compensatory Hyperinflation Compensatory hyperinflation as evidenced by increased radiolucency and splaying of vessels can be seen with the normal lobe or opposite lung. Alterations in Proportion of Left and Right Lung The right lung is approximately 55% and left lung 45%. In atelectasis this apportionment will change and can be a clue to recognition of atelectasis. Hemithorax Asymmetry In normals, the right and left hemithorax are equal in size. The size of the hemithorax will be asymmetrical and smaller on the side of atelectasis
  • 16. Atelectasis Right Lung Homogenous density right hemithorax Mediastinal shift to right Right hemithorax smaller Right heart and diaphragmatic silhouette are not identifiable
  • 17. Atelectasis Left Lung Homogenous density left hemithorax Mediastinal shift to left Left hemithorax smaller Diaphragm and heart silhouette are not identifiable
  • 18. Left Lower Lobe Atelectasis • Inhomogeneous cardiac density • Left hilum pulled down • Non-visualization of left diaphragm • Triangular retrocardiac atelectatic LLL
  • 19. Atelectasis Left Lower Lobe Double density over heart Inhomogenous cardiac density  Triangular retrocardiac density Left hilum pulled down Other findings include: Pneumomediastinum
  • 20. Atelectasis Left Upper Lobe Mediastinal shift to left Density left upper lung field Loss of aortic knob and left hilar silhouettes Herniation of right lung Atelectatic left upper lobe Forward movement of left oblique fissure "Bowing sign"
  • 21. Atelectasis Left Upper Lobe Hazy density over left upper lung field Loss of left heart silhouette Tracheal shift to left Lateral A: Forward movement of oblique fissure B: Herniated right lung C: Atelectatic LUL
  • 22. Lateral Movement of oblique and transverse fissures Atelectasis Right Upper Lobe Homogenous density right upper lung field Mediastinal shift to right Loss of silhouette of ascending aorta
  • 23. Lateral Movement of oblique and transverse fissures Atelectasis Right Upper Lobe Homogenous density right upper lung field Mediastinal shift to right Loss of silhouette of ascending aorta
  • 24. RML Atelectasis Vague density in right lower lung field, almost normal RML atelectasis in lateral view, not evident in PA view
  • 25. Vague density in right lower lung field (almost a normal film). Dramatic RML atelectasis in lateral view, not evident in PA view. Movement of transverse fissure. Other findings include: Azygous lobe
  • 26. Atelectasis Right Lower Lobe Density in right lower lung field Indistinct right diaphragm Right heart silhouette retained Transverse fissure moved down Right hilum moved down
  • 27. Adhesive Atelectasis Alveoli are kept open by the integrity of surfactant. When there is loss of surfactant, alveoli collapse. ARDS is an example of diffuse alveolar atelectasis. Plate-like atelectasis is an example of focal loss of surfactant.
  • 28. Relaxation Atelectasis The lung is held in apposition to the chest wall because of negative pressure in the pleura. When the negative pressure is lost, as in pneumothorax or pleural effusion, the lung relaxes to its atelectatic position. The atelectasis is a secondary event. The pleural problem is primary and dictates other radiological findings.
  • 29. Round Atelectasis Mass like density Pleural based Base of lungs Blunting of costophrenic angle Pleural thickening Pulmonary vasculature curving into the density Esophageal surgical clips
  • 30. Round Atelectasis Mass like density Pleural based Base of lungs Blunting of costophrenic angle, pleural thickening Pulmonary vasculature curving into the density
  • 31. RML Lateral Segment Atelectasis
  • 35.
  • 36.
  • 37.
  • 38.
  • 39. Bronchiectasis Left lung atelectasis due to mucus plugging Mucus plugs suctioned with bronchoscopy Bronchogram done after bronchoscopy Saccular bronchiectasis in bronchogram below
  • 40. Bronchogram Bronchograms are rarely done nowadays. The need for it disappeared with the invention of the fiberoptic bronchoscopy and high resolution CT scan. View these images to get a greater understanding of a three dimensional view of a bronchial tree..
  • 41. Bronchogram Bronchograms are rarely done nowadays. The need for it disappeared with the invention of the fiberoptic bronchoscopy and high resolution CT scan.
  • 42. Calcification Focal lung lesion: Ghon's complex Miliary lung calcification: Histoplasmosis Tuberculosis Alveolar microlithiasis Chicken pox pneumonia Solitary pulmonary nodule : Central / Granuloma Lamellar / Histoplasmosis Pop corn / Hamartoma Eccentric / Scar Cancer
  • 43. Calcification Nodes: Homogenous / TB Clumpy / Histoplasmosis Egg shell / Silicosis, Sarcoidosis Tracheal cartilage : Aging Tumor: Mediastinal mass / Teratoma Healed lymphoma / Metstasis
  • 44. Calcification Vascular: Aortic calcification Pulmonary artery calcification Pulmonary hypertension Pleural: Visceral / Hemothorax, TB, Empyema Parietal / Asbestosis Subcutaneous calcification: Cysticercus
  • 46.
  • 47. Silicosis Egg shell calcification of lymph nodes Other findings include: Diaphragmatic pleural calcification Multiple cavities with fluid levels
  • 50. Pleural Calcification Visceral pleural calcification Parietal pleura appears black because it is sandwiched between bony densities
  • 52. Visceral pleural calcification Open drainage with air fluid levels in pleural space
  • 55. Number: Multiple bilateral cavities would raise suspicion for either bronchiogenous or hematogenous process. You should consider: Aspiration lung abscess Septic emboli Metastatic lesions Vasculitis (Wegener's) Coccidioidomycosis, tuberculosis
  • 56. Location: • Classical locations for aspiration lung abscess are superior segment of the lower lobes posterior segments of upper lobes. • Tuberculous cavities are common in superior segments of upper and lower lobes or posterior segments of upper lobes. • When a cavity in anterior segment is encountered, a strong suspicion for lung cancer should be raised. TB and aspiration lung abscess are rare in anterior segments. Cancer lung can occur in any segment.
  • 57. Wall Thickness: • Thick walls are seen in: – Lung abscess – Necrotizing squamous cell lung cancer – Wegener's granulomatosis – Blastomycosis
  • 58. Wall Thickness: • Thin walled cavities are seen in: • Coccidioidomycosis • Metastatic cavitating squamous cell carcinoma from the cervix • M. Kansasii infection • Congenital or acquired bullae • Post-traumatic cysts • Open negative TB
  • 59. Contents: • The most common cause for air fluid level is lung abscess. Air fluid levels can rarely be seen in malignancy and in tuberculous cavities from rupture of Rasmussen's aneurysm. • A fungous ball should make you consider aspergillosis. A blood clot and fibrin ball will have the same appearance. • Floating Water Lily: The collapsed membrane of a ruptured echinococcal cyst, floats giving this appearance.
  • 60. Lining of Wall: The wall lining is irregular and nodular in lung cancer or shaggy in lung abscess
  • 61. Evolution of Lesion: Many times review of old films to assess the evolution of the radiological appearance of the lesion extremely helpful. Examples • Infected bullae • Aspergilloma • Sub acute necrotizing aspergillosis • Bleeding from Rasmussen's aneurysm in a tuberculous cavity
  • 62. Associated Features: Ipsilateral lymph nodes or lytic lesions of the bone is seen with malignancy
  • 64. Bulla Definition •Thin-walled–less than 1 mm •Air-filled space •In the lung> 1 cm in size and up to 75% of lung •Walls may be formed by pleura, septa, or compressed lung tissue. •Results from destruction, dilatation and confluence of airspaces distal to terminal bronchioles.
  • 65. •Bullous disease may be primary or associated with emphysema or interstitial lung disease. • Primary bullous lung disease may be familial and has been associated with Marfan's, Ehler's Danlos, IV drug users, HIV infection, and vanishing lung syndrome. •Bullae may occasionally become very large and compromise respiratory function. Thus has been referred as vanishing lung syndrome, and may be seen in young men.
  • 68. A: Xray shows bilateral bulla. B: CT shows bilateral bulla. C: CT after bullectomy.
  • 69.
  • 70. Pneumatocele is a benign air containing cyst of lung, with thin wall < 1mm as bulla but with different mechanism  Infection with staph aureus is the commonest cause ( less common causes are, trauma, barotrauma) lead to necrosis and liquefaction followed by air leak and subpleural dissection forming a thin walled cyst.
  • 71. •Honeycombing is defined as multiple cysts < 1cm in diameter,with well defined walls, in a background of fibrosis, tend to form clusters and is considered as end stage lung . •It is formed by extensive interstitial fibrosis of lung with residual cystic areas.
  • 72.
  • 73. A cyst is a ring shadow > 1 cm in diameter and up to 10 cm with wall thickness from 1-3 mm.
  • 74.
  • 76. A cavity is > 1cm in diameter, and its wall thickness is more than 3 mm.
  • 77. •A central portion  necrosis and communicate to bronchus. •The draining bronchus is visible (arrow). CT (2 mm slice thickness) shows discrete air bronchograms in the consolidated area. Mechanism
  • 79. A cavity in apicoposterior segment of left upper lobe
  • 80. 2.Number Multiple cavities: 1. Aspiration. 2. TB 3. Fungal. 4. Metastatic. 5. Septic emboli. 6.Wegners granulomatosis
  • 81. Multiple cysts of metastasis from squamous cell carcinoma. Multiple thick wall cavities from adenocarcinoma of right lung
  • 82.
  • 83.
  • 84. Irregular , nodular inner lining of thick wall abscess Malignant cavity. 3. Thickness and irregularity
  • 86. 5. Relation to lymph node enlargement
  • 88. •Arrow head  Crescent sign. •Black arrows  Fibrotic bands surrounding cavity (Fibrocavitary TB).
  • 89.
  • 90. Primary Lung Cancer • Thick wall • Shaggy lumen • Eccentric cavitation
  • 91. | Squamous Cell Carcinoma Lung LUL mass Thick walled cavity Eccentric location of cavity
  • 92. Fungous Ball Long standing cavity Containing round density (A) Mobile density Adjacent pleural reaction (B) - characteristic of aspergilloma