2. What is Periodontitis ?
Periodontitis is
Inflammatory disease of the supporting tissue of the teeth
Caused by specific microorganism
Resulting in progressive destruction of the periodontal
legament and alveolar bone with pocket formation ,
recession or both
5. What is Juvenile or Aggressive
Periodontitis?
Juvenile or Aggressive periodontitis is
Rapid loss of attachment and bone loss
Occurring in an otherwise clinicially healthy patients with
the amount of microbial deposits in consistent with disease
severity
Familial aggregation of diseased individual
8. • Degenaration of principle fibers of PDL
• Cessation of cementum formation
• Resorption of alveolar bone
• Tooth migration without detectable inflammation
Stage I
• Rapid proliferation of the junctional epithelium
along the root
• Earliest sings of inflammation appear
Stage II
• Progressive inflammation and development of
deep, infrabony periodontal pocket
Stage
III
9. Etiology of juvenile periodontitis
Micro organisms
Actinobacillus actinomycetemcomitans
Porphyromonas gingivalis
Eikenella corrodens
Caprocytophaga
Spirocheates
Bacillus
Defective neutrophil or monocyte function
Poor serum antibody response to infecting agent
11. What is localized juvenile
periodontitis ?
Localized juvenile periodontitis is
Occuring in otherwise healthy individual under the
age of 30 years
Destructive periodontitis localized to the 1st
permanent molars and incisors not involving more
than two other teeth
13. Age and sex distribution
Both sexes ( slight female predilection )
Seen mostly between 20 years
Distribution of lesion
1st molar and/or incisors
1st molar and/or incisors + additional teeth ( not more than 2
teeth other than 1st molars and incisors )
14. Lack of clinical inflammation despite the presence of deep
periodontal pocket
Small amount of plaque, forms a thin film rarely
mineralized
Mobility and migration of 1st molars and incisors
Classically distolabial migration of maxillary incisors with
diastema
15. Root surface sensitivity
Deep dull radiating pain
Periodontal abscess
Enlargement of regional lymphnode
17. Vertical or angular bone loss around the 1st molars and incisors
The pattern appears to be “Arc shaped” loss of alveolar bone
extending from distal surface of 2nd premolar to mesial surface of
2nd molar
Frequently bilaterally symmetrical pattern of bone loss occurs
called as “ mirror image pattern”
Fig : Radiograph showing localized juvenile
periodontitis
19. Ulcerated pocket epithelium
Accumulation of various inflammatory cells in the
connective tissue mainly leukocytes, plasma cells and
small number of lymphocytes and macrophages
Bacterial invasion of connective tissue
The flora involves A. actinomycetemcomitans ,
Capnocytophaga sputigena and others
21. Leucotoxin Destroys polymorphonuclear
leukocytes and macrophages
Endotoxin Activates host cells to secret
inflammatory mediators
Bacteriocin Inhibit IgG and IgM production
Collagenase Degradation of collagen
Chemotactic
Inhibition factor
Inhibit neutrophil chemotaxis
Virulence factor associated with
A. actinomycetemcomitans
22. Immunology
Functionals defects of polymorphoneuclear leukocytes
or monocytes, impairs the chemotactic attraction of
these cells to the site of infection
24. What is Generalized juvenile
periodontitis ?
Generalized juvenile periodontitis is
Generalized interproximal attachment loss
Affecting atleast three permanent teeth other than the
1st molar and incisors
26. Age and sex distribution
Affects between puberty and 35 years
No sex discrimination
Distritubiton of lesion
All or most of the teeth are affected, no specific pattern is
observed
There are two phases of lesion
27. Destructive phase Non destructive phase
Tissue appears severely
inflammed, ulcerated
and fiery red
Bleeding with or
without stimulation
Suppuration
Attachment and bone
loss
Tissue appears pink with some
stippling
Lack of inflammation
Probling will reveal deep pocket
Bone attachment levels relatively
stable
28.
29. Some patients may exhibit
Weight loss
Mental depression
General malaise
Systemic condition may predispose patient to
generalized juvenile periodontitis, these includes
chornic neutrophil defect
Leukocyte adherence deficiency
31. No define pattern of distribution
Range from severe bone loss associated with
minimal number of teeth to advanced bone loss
affecting the majority of teeth in the dentition
Fig : showing radiograph of GJP
32. Treatment
Extraction Standard
periodontaltherapy
Antibiotic therapy
Extraction of
involved teeth,
specially 1st molar
Transplantation
of developing 3rd
molar into the
sockets of
previously
extracted 1st molar
Scaling
Root planning
Curettage
Flap surgery with /
without bone grafts
Root amputation
Hemisection
Occlusal
adjustment
Tetracycline
hydrochloride 250mg
q.i.d + local
mechanical therapy
Doxycycline 100mg
per day
Combination of
amoxicilline and
metronidazole
34. Periodontal disease accounts for a majority of
missing teeth in adults and results in tremendous
economic and social burdens both to the individual
and the society
Periodontal disease is so prevalent that only
possible solution to the problem is its prevention by
maintaining the good oral hygiene
Conclusion
35. Reference
Essential of Clinical Periodontology And Periodontics
; 4th edition ; Shantipriya Reddy
Carranza’s Clinical Periodontology ; 11th edition ;
Newman ;Takei ; Klokkevold ; Carranza
Cawson’s Essentials of Oral Pathology And Oral
Medicine ; 8th edition ; R. A. Cawson ; E. W. Odell