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Explain every types of alopeica with their treatments.

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  1. 1. INTRODUCTION Hairs are keratinized elongated structures derived from invaginations of epidermis and project out from most of the body surface.
  2. 2. Types of Hair Morphologically:
  3. 3. LENGTH, WIDTH AND GROWTH RATE Length : range from <1mm to > 1 meter. Average uncut scalp hair : 25 – 100 cm. Width : from 0.005 to 0.06mm. Growth rate: about 1 cm/ month (terminal hair).
  4. 4. FUNCTIONS 1. Protects body surface from external injury. 2. Helps in sensory function. 3. Psycho – social importance. 4. Forensic importance:- i. Identification of race, sex, age and religion. ii. Cause of death can be determined. iii. Time of death can be determined. 5. Assist thermo regulation mainly in lower animals.
  6. 6. ANAGEN (GROWING PHASE) Lasts for about 1000 days. Follicular cells grow, divide and become keratinized. Darkly pigmented portion is evident just above the hair bulb.
  7. 7. CATAGEN (INVOLUTING PHASE) Lasts for about 10 days. Gradual thinning and decrease of the pigment. Melanocytes stop producing melanin. Matrix keratinocytes abruptly cease proliferating.
  8. 8. TELOGEN (RESTING PHASE) Lasts for about 100 days. Club-shaped proximal end shed from the follicle during telogen. Growth of a new anagen hair leads to shedding of any remaining telogen hair.  New hair does not “push out” the hair from the previous cycle.
  9. 9. EXOGEN (HAIR SHEDDING PHASE) Recently added phase. Describes relationship between hair shaft and base of telogen follicle. Hairs can be retained for more than one cycle. Shedding phase is most likely independent of anagen and telogen.
  10. 10. HAIR LOSS Hair loss is a common reason for male and female patients to consult the dermatologist. Natural shedding of hair accounts for normal daily hair loss. Recent measurements indicate that the average rate of hair loss is closer to 35 to 40 hairs per day.
  11. 11. MEDICAL CAUSES OF ALOPECIA • Physical stress: surgery, illness, anemia, lack of sleep. • Emotional stress: psychiatric illness, death of family member, job loss, anxiety etc. • Diet considerations: rapid weight loss or gain, unusual dieting habits, protein intake failure, prolonged fasting. • Hormonal causes: postpartum, oral contraceptives, menopause, ingestion of testosterone containing hormone supplements. • Endocrinopathy: hypothyroidism, hyperthyroidism.
  12. 12. CLASSIFICATION OF ALOPECIA • Alopecia areata. • Androgenetic alopecia. • Postpartum Alopecia • Trichotillomania. • Traction alopecia. • Syphilitic alopecia. • Triangular alopecia.
  13. 13. ALOPECIA AREATA Rapid and complete loss of hair in one or most often several round or oval patches. Usually occur on the scalp, bearded area, eyebrows, eye lashes and less commonly on other hairy areas of the body.
  14. 14. MEDICATIONS Corticosteroids. Cyclosporine. Sulfasalazine.  Methotrexate. Azathioprine.
  15. 15. ANDROGENETIC ALOPECIA Also known as Male pattern baldness or Common baldness. Reversible scalp hair loss that generally spares parietal and occipital areas of the scalp. Usually occurs in twenties or early thirties. Chiefly occurs in the vertex and front temporal regions.
  16. 16. MEDICATIONS Topical minoxidil (below 2% ). Finasteride (2.5 mg/daily). Spironolactone.
  17. 17. POSTPARTUM ALOPECIA Temporary hair loss at the conclusion of pregnancy. Growth cycle generally returns to normal within one year after the baby is delivered.
  18. 18. TRICHOTILLOMANIA Defined as a self-induced and recurrent loss of hair. It includes the criterion of an increasing sense of tension before pulling the hair and gratification or relief when pulling the hair.
  19. 19. MEDICATIONS Clomipramine. Fluoxetine and other selective serotonin reuptake inhibitors. Non-pharmacological interventions, including behavior modification programs, may be considered. Dual treatment (behavioral therapy and medication) may provide an advantage in some cases.
  20. 20. TRACTION ALOPECIA Is a form of alopecia, or gradual hair loss, caused primarily by pulling force being applied to the hair.
  21. 21. TRIANGULAR ALOPECIA also known as "Temporal alopecia” non-inflammatory, non-scarring form of hair loss. hair loss that may be congenital but usually appears in childhood as a focal patch of loss that may be complete or leaving fine vellus hairs behind. inherited by the autosomal dominant trait.
  22. 22. SYPHILITIC ALOPECIA Observe in syphilis patient. Hair loss can occur as patchy “moth- eaten” thinning occurring in small irregular areas.
  23. 23. CONCLUSION Alopecia cannot be cured totally. Treatment is a very lengthy. Researches are going on to invent a way to cure alopecia totally. Some peoples implant false hair nowadays for their mental satisfaction.
  24. 24. REFERENCES • Walker SA, Rothman S. Alopecia areata: a statistical study and consideration of endocrine influences. J Invest Dermatol 1950; 14:403–13. • Ikeda T. A new classification of alopecia areata. Dermatologica 1965; 131: 421–45. • Hamilton, J.B. (1951) Patterned loss of hair in man: types and incidence. Annal NY Acad Sci 53, 708-728. • Norwood, O.T. and Lehr, B. (2000) Female androgenetic alopecia: a separate entity. Dermatol Surg 26, 679-682, PubMed Label: 20345216.

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Explain every types of alopeica with their treatments.


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