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Dr Rupa Ananthasivan DMRD, DNB, FRCR
Consultant Radiologist, Manipal Hospital, Bangalore.
Vascular Emergencies
๏‚— Prompt diagnoses
๏‚— Accurate Diagnoses
๏‚— Timely Intervention
๏‚— Quick Decision Making-Surgical vs Non-surgical
Tools Available
๏‚— Ultrasound with Doppler
๏‚— CT Angiography
๏‚— MR Angiography
๏‚— DSA
๏‚— Intravascular USG
Advantages
๏‚— Readily Available
๏‚— Portable
๏‚— Speed
๏‚— Temporal Resolution
๏‚— Spatial Resolution
Disadvantages
๏‚— Acoustic Impedance-Air, Bone, Soft tissue, Bowel,
tissue oedema,
๏‚— Field of View
๏‚— Operator Dependence
Doโ€™s and Donโ€™ts !!!
๏‚— Do NO Harm !!!
๏‚— Be Prepared.
๏‚— Be Meticulous but quick.
๏‚— Know your Patient โ€“and your machine .
Acute Vascular Emergencies
๏‚— Ruptured Aortic Aneurysm
๏‚— Acute Carotid Thromboses
๏‚— Carotid and Vertebral dissection
๏‚— Pseudo-Aneurysm
๏‚— Acute Limb Ischemia
๏‚— Graft Failure
Ruptured Aortic Aneurysm
๏‚— Fatal if untreated. 50 % may not reach hospital alive
๏‚— Surgery=30-65 % survival rate
๏‚— Classical Triad- Back Pain, Hypotention, Pulsatile
Abdominal Mass- 50%
๏‚— Timely Intervention is the key
Propensity to Rupture
๏‚— F > M
๏‚— > 6 cm
๏‚— Increase in size > 5 mm / year (vs 2-3 mm/yr)
Ruptured Aortic Aneurysm
๏‚— Sensitivity= 98 %
๏‚— Specificity=95 %
( Setting of Abdominal Pain and Haemodynamic
Instability)
Findings
๏‚— Crescent of Intramural Hemorrhage
๏‚— Para- Aortic Haematoma
๏‚— Retroperitoneal Haematoma
๏‚— Haemoperitoeum
Acute Carotid Thrombosis
๏‚— Complication of endarterectomy/ stenting
๏‚— Acute progression of Carotid stenosis
โ€œ Culpritโ€ lesion in an acute stroke
Acute Carotid Thrombosis
๏‚— Thrombus is heterogenously echogenic/ or very
hypo-echoic
๏‚— Calibre of vessel is normal or expanded
๏‚— Pulsations in vessel wall but NO FLOW
๏‚— Swirling, sludge like flow in Carotid bulb
Thump Flow
Acute Carotid Thrombosis-
๏‚— Thump flow proximally
๏‚— No flow in the ICA
๏‚— Low resistance flow in the ECA
Free Floating Thrombus
๏‚— Broad Base towards vessel wall
๏‚— Tongue like projection
๏‚— Oscillation with blood flow
Carotid Dissection
๏‚— Due to hemorrhage into the intima with extension into
sub-intimal and sub-adventitial layers
๏‚— Type A Dissection of Aortic Arch
Carotid Dissection
๏‚— Ehler-Danlos Syndrome
๏‚— FMD
๏‚— Idiopathic
๏‚— Trauma- Hyper-extension, Penetrating
๏‚— Iatrogenic-Following Catheterization
Presentation
๏‚— Often delayed
๏‚— Neck pain, headache
๏‚— Tinnitus
๏‚— Focal neurological defects
๏‚— Horners Syndrome
๏‚— Blindness
Dissections
๏‚— 20% strokes in young patients
๏‚— 2.5% strokes in older patients
Carotid Dissection
๏‚— Intra-cranial- Rare-75% mortality -20-30 yrs
๏‚— Extra-cranial- Subtle Symptoms-40yrs
Commonly - proximal ICA just distal to bifurcation
Extra- cranial Carotid Dissection
๏‚— Patent Carotid Bifurcation
๏‚— Tapering of the proximal ICA
๏‚— Distal ICA stenosis /occlusion
๏‚— Intimal Flap
๏‚— Thrombosed False
Lumen bulging out
Carotid Dissection
๏‚— High resistance , Low velocity flow in Carotid artery
๏‚— If False lumen patent โ€“
low velocity to and fro flow
Carotid Dissection
Courtsey-Dr Ullas V Acharya
Vertebral Dissection
๏‚— Occlusion
๏‚— Dampened Flow
๏‚— Reverse flow
Accuracy
๏‚— Cervical Carotid artery=70 %
๏‚— Vertebral Artery=75-86%
Pseudo Aneurysm
๏‚— Contained Rupture of a blood Vessel
๏‚— Absence of three layers
๏‚— Causes
Iatrogenic- Catheterization, Post-Surgical following
Anastomosis, Arterial Repair
๏‚— Penetrating Trauma
Femoral Pseudo-aneurym
๏‚— O.2 % of diagnostic angios
๏‚— 8% 0f Interventional procedures
๏‚— CFA > EIA> SFA> DFA
Femoral Pseudo-Aneurysm
๏‚— Incidence=7-9 %
> Size of Catheter,
>Length of procedure,
>Use of anti-coagulants,
>Obesity,
>Hypertension
Femoral Pseudo-aneurysm
๏‚— Often 1-10 days post-procedure
๏‚— Swelling in Groin
๏‚— Pulsatile mass
๏‚— Thrill
Femoral Pseudoaneurysm
๏‚— โ€œBubble like Anechoic /hypoechoic structure attached
to artery
๏‚— Cylindrical neck along needle track
๏‚— Bubble measures 1-3 cms
๏‚— Neck variable length and width
Femoral Pseudoaneurysm
๏‚— Swirling pattern within pseudo-aneurysm-----Yin-
Yang
๏‚— To and Fro flow in the neck
๏‚— Rarely only neck show flow
Treatment
๏‚— USG guided compression- Upto 75% success
> Ihr compression
> painful
Thrombin Injection-99% success
0.5-1ml 0f 1000 IU/ml
Inadvertent injection into artery/vein very rare
Hematoma
๏‚— Natural Outcome of Vascular Disruption
๏‚— Due to poor Compression
๏‚— Ecchymosis and Discomfort
๏‚— Rarely large compressing the artery and nerve roots
๏‚— Retroperitoneal and pelvic extention, Compartment
syndrome
Haemotoma
๏‚— Initially Echogenic / hypo-echoic well/ill defined area
๏‚— Later becomes more well-defined and anechoic
๏‚— Initial scan โ€“exact measurements and even skin
marking
Acute Limb Ischemia
๏‚— Embolic- Heart, Aorta, Iliac arteries, Aneurysm
๏‚— Thrombosis over an existing plaque
Symptom onset is usually rapid-severe claudication, rest
pain, sensory loss and colour change
Depends upon collaterals
Acute Interventions
๏‚— Limb Saving-Embolectomy, Bypass, Thrombolysis
๏‚— Life Saving- Amputation
Acute Limb Ischemia
๏‚— Gray Scale
๏‚— Colour Doppler
๏‚— Spectral Tracing
Acute Limb Ischemia
๏‚— Case 1--- Gangrene left Foot
๏‚— Case 2- Acute Pain Right hand, Impending Gangrene
๏‚— Case 3- Gangrene right Thumb
Left Superficial Femoral Artery-1
Brachial Artery โ€“ Case 2
Acute Limb Ischemia
๏‚— Acute Thrombosis - hetero-echoic/ hypoechoic
๏‚— Expansion of Vessel
๏‚— Retrograde Propagation
๏‚— Use Accompanying veins to track artey
๏‚— Identify reconstituted area
Acute Limb Ischaemia
๏‚— Upstream- High Resistance, Biphasic, Reversed
Diastolic
๏‚— Downstream-Monophasic, Low resistance, Decreased
Velocity
Case 2-
Impending
Gangrene-Right
Hand
Case 2
Case 1- Gangrene
Left Foot
Case 3- Gangrene Left
Thumb
Case 1- Left
Foot
Gangrene
Leg arteries
Case 2- Impending
Gangrene-Right
Hand
Case 3-
Gangrene
Left Thumb
Diagnoses
๏‚— Case 1- Superficial Femoral artery occlusion due to
atherosclerosis with significant small vessel disease
๏‚— Case 2- Acute Distal Subclavian Artery Occlusion
๏‚— Case 3-Significant Radial Artery Compromise
Case 2- Acute
Subclavian
Occlusion
Failed Inguino Bypass Graft
๏‚— > 1 month ---------Surgical technique
๏‚— 1month-2 years---Fibrio-intimal Hyperplasia
๏‚— > 2 years-------------Atherosclerosis
Bypass Graft Failure
๏‚— Graft thrombosis
๏‚— Absent Flow
๏‚— Poor flow in run โ€“off vessels
Impending Failure
๏‚— Low flow velocity within the graft
๏‚— Average velocity < 45 cm/sec
๏‚— Interval decrease in ankle- brachial index
-Alert
-Quick
-Accurate
-Calm
The Eyes do not see what the
mind does not know !!!!
Dr Pramesh Reddy
Mr. Mahesh Mahadev
Carotid Stenosis
๏‚— Candidates for Acute Carotid Endarterectomy / Acute
Carotid Stenting
๏‚— Cresendo TIA
๏‚— Stroke In Evaluation
๏‚— Fluctuating defficiets
๏‚— Free floating thrombosis
Intra-operative USG
๏‚— Intimal flaps
๏‚— Ulcerative plaques
๏‚— Retained Thrombi
Acute Limb Ischaemia
๏‚— ? Segmental Pressure Gradients
๏‚— Entire limb artery Examination
AVF (Graft ) Thrombosis
๏‚— Loss of thrill
๏‚— Inability to dialyze
๏‚— Swelling
๏‚— Usually Venous portion
Other Complications
๏‚— A-V Shunting through unligated veins
๏‚— Anastomotic Pseudoaneurysm
๏‚— Perigraft Abcess
Role of doppler in acute vasclar emergencies dr.rupa
Role of doppler in acute vasclar emergencies dr.rupa
Role of doppler in acute vasclar emergencies dr.rupa
Role of doppler in acute vasclar emergencies dr.rupa
Role of doppler in acute vasclar emergencies dr.rupa
Role of doppler in acute vasclar emergencies dr.rupa

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