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Name of the Student : Ranjitha .N.
ID No. : UHS15PGM664
Course : HPP 507
Degree Programme and Subject : M.Sc., (Hort.)
Plant pathology
College : College of Horticulture, Bengaluru.
Principles Of Integrated Diseases
Management
Exclusion
Eradication
Protection
Resistance
Therapy
Avoidence
METHODS TO CONTROL PLANT DISEASES
INTRODUCTION
• Chilli (Capsicum annum and Capsicum fruticence) belongs to the family
solanaceae and genus capsicum.
• Leading state AP (25% of the total area and over 40-50%of the total world
production).
• Chilli is grown in tropical as well as in temperate zones.
• It is widely used in the manufacture of curry powder, curry paste and all kinds of
pickles and preparing soups and salads etc, the pungency in chillies due to an
alkaloid capsisin.
• A major contributor to this lower productivity are various diseases ,which are
damping off, anthracnose ,bacterial leaf spot and leaf curl of chillies
• Damping off - Pythium spp
• Anthracnose- Colletotrichum goleosporides
• Bacterial leaf spot –Xanthomnas campestries pv.vesicatoria
• Leaf curl – Tobacco leaf curl
• Phytophthora – Phytopthora capsici
• Alternaria blight – Alternaria capsici
• Leaf spot - Cercospora capsici
• Powdery mildew – Levilulla taurica
• Chilli mosaic – CMV( cucumber mosaic virus )
• The diseases prevently occurs and greatly damages many plants such as chilli
,tomato , brinjal belonging to solanaceae family.
SYMPTOM:
• Symptom of this diseases generally manifest into two stages pre emergence and
post emergence stages.
• Pre emergence stage symptom occur when the seeds are still to grow in soil or the
developing seedlings are still to come over the soil surface.
• Seed generally fail to germinate ,become soft and mushy then turn to brown ,
shrink and finally degenerate.
• Post emergence symptom occur when the seedling are out on the surface of the
soil.
• The pathogen attack the young tissues at or below the ground level. The infected
tissues become discoloured ,water soaked and soon collapse.
• The infected part of the seedlings looks much thinner softer than healthy part.
• The pathogen continues to invade the seedling tissues after it has fallen on the
ground and seedling quickly withers and dies.
IN NURSERY(DAMPING OFF)
DAMPING OFF(POST EMERGENCE)
ETIOLOGY
• Diseases caused by pythium spp (Pythium ultimum ,Pythium debaryanum
,Pythium aphanidermatum)
• The mycelium is colourless ,slender , coenocytic ,profusely branching and rapid
growing.
• Mycelium produces terminal or intercalary sporangia, which are globose to oval.
• The zoospores are produced within a vesicle.
• Zoospores are reniform and biflagellate.
• The size of the resting zoospores is upto 8µm in diameter.
• Sexual spores are oospores ,the oospore develops into a smooth and thick walled
oospore. It measures about 12-20µm.
• Primary source :oospores in soil or plant debries.
• Secondary source: zoospores through irrigation water.
.
Epidemology:
• High soil moisture
• higher soil temperature.
• The disease is further aggravated in ill-aerated soils with poor drainage
having thick stand of the seedlings
CULTURAL PRACTICES:
• Field sanitation:
1. Collect and burn the infected plant debries of chilli.
2. Destroy self-grown solanaceous plant, other volunteer plants from the field and
surrounding areas.
Deep ploughing:
1. Post harvest ,plough the left over inoculum deep into the soil
2. Practice summer ploughing
3. solarization
Cropping system:
• Follow crop rotation.
• Avoid growing of two solanaceous crop in a single year in and around the same
field.
Seed:
• Use certified seed of varieties for different areas.
2.Use Seeds obtained from a diseases free crop.
3. Seed treatment with Bavistin or Agrosan GN @2g/kg of seeds
Sowing:
• Thin sowing to avoid crowding ,
• light sandy soils for nurseries or use of pure time sand dust mixture for raising
the seedlings .
• Under well drained conditions seedling can be transplanted into loamy soil also.
NUTRITION
Apply fully decomposed FYM or compost to balance the nutrient supply .
Add NPK fertilizer where necessary to make up for a 90:60:60 kg/ha.
Thinning and weeding:
• Remove the weeds .
• Rouge out the mixture and stay diseased plant as their serve as foci of infection.
IRRIGATION
• Provide only need based light irrigations
VARIETAL:
• Resistant/ tolerant genotypes against disease G-5,Pusa Jwala ,PantC-1 ,NP-46
CHEMICAL:
• seed treatment such as Thiram or captan @ 3g/kg of seeds for the
better control of damping off .
• Soil drenching with Captan or Thiram @0.2-0.5%
• Also called dieback or ripe fruit rot of chillies.
• The disease is one of the most serious disease of chilli . It occurs throughout India
SYMPTOMS:
• The pathogen cause two different types of symptoms on two different parts of the
host.
• Called die back because the disease cause necrosis of the tender twigs from the tip
back wards.
• The entire branch of the plant may wither away.
• The twigs are water soaked to brown , become grayish white or straw coloured in
advance stage of the disease.
• Large number of black dots called acervuli are formed on the affected twigs.
• The disease usually occurs on mature fruits as circular to elliptical sunken black
margins and marketed with concentric rings.
• Badly diseasd fruits turn straw coloured from normal red.
• On this discoloured area ,numerous black dots are present. The diseased fruit may
drop off prematurely.
ACERVULLI
ETIOLOGY
• Disease is caused by Colletotrichum capsici .
• Mycelium septate ,colourless and inter and intracellular.
• Hyphae develop the fruiting body called acervulli.
• Acervulli consists of septate ,conidiophore and conidia.
• The setae are septate and dark brown with light brown tip.
• Conidiophores are mostly clubshaped ,under- branched, small and unicellular.
• The conidia are hyaline ,unicellular ,sickle shaped and are produced singhly at the
tip of the conidiophores.
Primary source: Infected seeds and diseased crop debries .
Secondary source :Conidia dispersed by rain splash and wind .
It also infect tomato ,potato ,brinjal ,wild bringal are the other hosts.
Epidemology:
• Ideal temperature is 28-30°.the pathogen infects the host at a minimum
temperature of 22-25°c.
• Relative humidity : An RH of above 80% is ideal. Heavy prolonged dew
deposition is important for dieback development.
• Integrated of following practices manage the diseases effictely economically and
in a most environment friendly manner.
CULTURAL PRACTICES:
• Field sanitation:
1. Collect and burn the infected plant debries of chilli
2. Destroy self-grown solanaceous plant, other volunteer plants from the field and
surrounding areas.
• Seeds obtained from a diseases free crop.
• Treat the seeds with 2g of Thiram per kg of seeds before sowing for anthracnose
of chillies.
Thinning and weeding:
• Weeds serve carriers of plant pathogens and high plant density favour
development .
• Remove the weeds once before and then after the irrigation.
• Rouge out the mixture and stay diseased plant as their serve as foci of
infection.
IRRIGATION
• Provide only need based light irrigations, high humidity, and frequent irrigation
favour disease development.
VARIETAL:
• Resistant/ tolerant genotypes against major disease G-4 ,Pusa Jwala ,PantC-1 ,JCA
196.
Chemical method:
• For the management of Anthracnose chilli –
• spray of Captafol @ 0.2% followed by copper oxy chloride @ 0.25% and
Caebendizim @0.1% , two sprays at 10-15 days interval ,35-45 days after
transplanting .
Bacterial spots
• Major disease of chillis in tropical and subtropical climates. This appears on
leaves and fruits.
• In Rajasthan the disease cause 8-16% loss in yield of fruits.
SYMPTOM:
• On leaves ,the first indication of the disease is appear of small , circular to
irregular, water soaked area which appear as definite spots on the lower surface of
the leaf.
• As the colour changes from dark green to purplish grey with a black centre.
• A narrow yellow halo may surround the spot.
• In the wet weather , the leaves are too many ,the intervening tissues become dry
and brown and the whole leaf dies.
INFECTED FIELD(BACTERIAL DISEASE)
Etiology
• Diseases caused by Xanthomonas campestris pv. Vesicatoria.
• The bacterial cells are single ,straight rods 0.2-0.8*0.6-2.0 µm in size.
• They are gram negative and motile by a single polar flagellum.
• These bacterial are strict aerobes and optimum temperature for growth is 25-27°C.
Disease cycle
• It is a seed borne bacteria .10-15% seeds carry the bacteria which is enough to
initiate the disease through seedlings.
• The bacterium also subsists on infected plant debries, weeds and volunteer plants.
• Spattering rains are the chief means of local dissemination of bacterial cells from
the ooze developed on the affected parts.
• The bacteria are also rapidly spread throughout the chilli.
• movement of equipments and workers having come in contact with the diseased
plants.
• Field sanitation
• Use diseases free seedlings and seeds.
• Use of resistant varieties.
• Avoid sprinkler irrigation.
• Seed treatment with Agronson GN @2.5g/kg of seeds and Cersan @2.5g/kg of
seeds were used for bacterial leaf spot of chilli .
• Application of streptomycin sulphate 500ppm+COC 0.3%
Leaf Curl of Chilli:
• The leaf curl is very common wherever the crop is grown.it is a major initiation in
successful cultivation of both hot and bell pepper.
SYMPTOM:
The most characteristics symptom are
• the curling of leaves , small size , shortened internodes and general dwarfing of
the plant which assumes a bushy appearance.
• Leaves are pale color and roll downwards . Fruiting is stopped, if fruit are formed
, they are small and deformed.
• Causal agent: Tobacco leaf curl virus
• The genome consists of a single circular ssDNA.
• Whereas the genome of the other known whitefly transmitted Gemini virus
consists of two ssDNA.
• Gemini particles are paired and polyhedral .The member of this group are found
predominantly in tropical area.
INFECTED PLANT
Transmission:
•The disease is transmitted by whitefly , Bemisia tobasci.
• This vector spread the maximum diseases during the month of august to October.
• The vector acquires the virus after feeding on an infected plant for at least 15-30
minutes.
•There latent period of several hours after which the virus can be inoculated into a
healthy plant.
Management
Cultural control
• Nursery beds should be covered with nylon net or straw to protect the seedlings
from viral infection.
• Raise 2 -3 rows of maize or sorghum as a border crop to restrict the spread of
vector.
• Rogue out the infected plants as soon as they are noticed in the field.
Bio-control agents
• Different botonicals , animal products and other materials such as ash ,cow urine
,and dung ,fish meal,and milk etc.
• Rhizobacteria(PGPR),leaf proteins,proline,and Trichoderma spp.reduce diseases
in many plant species manipulating host plant’s physical and biochemical
properties.
• Use of milk in plant diseases management – it is the indigenous ancient tool
against plant diseases.
• Amino acids ie, proline, isoleucine and phenyl alanine in milk induce resistance
against plant diseases.
• potassium phosphate content strengthens host immune system against diseases.
Chemical method:
• Protect the crop in nursery bed from insect vectors(whitefly)by spraying
Metasystox 1ml/lt of water at 10days interval.
OTHER MINOR DISEASES
• BACTERIAL SOFT ROT
Erwinia carotovora pv. carotovora
Symptom:
• The internal tissue softens before eventually turning into a watery mass with a foul
smell.
• This problem is worst in wet weather beacouse the bacteria are rain splashed from
the ground and on to the fruit.
• Also started by insect injury.
Management :
• Keep plants off the ground
• Controlling insects can help reduce the threat
• BACTERIAL WILT:
Pseudomonas solanacearum
Symptom:
• Start with wilting of the leaves.
• After a few days a permanent wilt of the entire plant results,with no leaf
yellowing.
• Cutting roots or stems show milky streams of bacteria when they suspended in
water.
Management
Plant clean and transplants and to remove diseased plants.
• CERCOSPORA LEAF SPOT
Crecospora capsici
Symptom:
• Worst under extended warm, wet conditions.
• Characterized by small brown circular leaf lesions that have a watery appearance.
• Excessive leaf drop may occur in common infestations.
Management:
• Use clean seed and crop rotation
• Good air flow round plantsin sheltered areas will also help minimize
this problem.
• Fungicides like Hexaconazole 0.1% etc.
• PHYTOPTHORA BLIGHT
PHYTOPTHORA CAPSICI
SYMPTOM:
• Plants suffering from this condition often wilt and die, leaving brown stalks and
leaves ,small
• Poor quality fruits
• If the fungus enters the roots .
• Symptoms of the less serve leaf blight include brown or black spots that may kill a
localized portion of the plant.
• Affected areas are often borded with a white mold.
• Wind borne fungus ,observed in wet water logged condition.
• It is promoted by warm , wet weather
Management:
• Avoid excess watering and poorly drained soil.
• Fungicides : Mancozeb@0.2%
• The cultivated chilli suffers from number of fungal ,bacterial ,and viral diseases.
• Among the fungal diseases damping off and anthracnose are the major devastring
diseases causing considerable yield loss .
• Environment play an effective role for development of these diseases.
• Diseases perpetuate through plant debries in soil ,special fruiting body ,volunteer
host and weeds year to year .
• The diseases can be managed effectively and eco friendly by cultural practices and
applying economic chemicals.
Idm

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Idm

  • 1. Name of the Student : Ranjitha .N. ID No. : UHS15PGM664 Course : HPP 507 Degree Programme and Subject : M.Sc., (Hort.) Plant pathology College : College of Horticulture, Bengaluru.
  • 2. Principles Of Integrated Diseases Management Exclusion Eradication Protection Resistance Therapy Avoidence
  • 3. METHODS TO CONTROL PLANT DISEASES
  • 4. INTRODUCTION • Chilli (Capsicum annum and Capsicum fruticence) belongs to the family solanaceae and genus capsicum. • Leading state AP (25% of the total area and over 40-50%of the total world production). • Chilli is grown in tropical as well as in temperate zones. • It is widely used in the manufacture of curry powder, curry paste and all kinds of pickles and preparing soups and salads etc, the pungency in chillies due to an alkaloid capsisin. • A major contributor to this lower productivity are various diseases ,which are damping off, anthracnose ,bacterial leaf spot and leaf curl of chillies
  • 5. • Damping off - Pythium spp • Anthracnose- Colletotrichum goleosporides • Bacterial leaf spot –Xanthomnas campestries pv.vesicatoria • Leaf curl – Tobacco leaf curl
  • 6. • Phytophthora – Phytopthora capsici • Alternaria blight – Alternaria capsici • Leaf spot - Cercospora capsici • Powdery mildew – Levilulla taurica • Chilli mosaic – CMV( cucumber mosaic virus )
  • 7. • The diseases prevently occurs and greatly damages many plants such as chilli ,tomato , brinjal belonging to solanaceae family. SYMPTOM: • Symptom of this diseases generally manifest into two stages pre emergence and post emergence stages. • Pre emergence stage symptom occur when the seeds are still to grow in soil or the developing seedlings are still to come over the soil surface. • Seed generally fail to germinate ,become soft and mushy then turn to brown , shrink and finally degenerate.
  • 8. • Post emergence symptom occur when the seedling are out on the surface of the soil. • The pathogen attack the young tissues at or below the ground level. The infected tissues become discoloured ,water soaked and soon collapse. • The infected part of the seedlings looks much thinner softer than healthy part. • The pathogen continues to invade the seedling tissues after it has fallen on the ground and seedling quickly withers and dies.
  • 11. ETIOLOGY • Diseases caused by pythium spp (Pythium ultimum ,Pythium debaryanum ,Pythium aphanidermatum) • The mycelium is colourless ,slender , coenocytic ,profusely branching and rapid growing. • Mycelium produces terminal or intercalary sporangia, which are globose to oval. • The zoospores are produced within a vesicle. • Zoospores are reniform and biflagellate. • The size of the resting zoospores is upto 8µm in diameter.
  • 12. • Sexual spores are oospores ,the oospore develops into a smooth and thick walled oospore. It measures about 12-20µm. • Primary source :oospores in soil or plant debries. • Secondary source: zoospores through irrigation water. .
  • 13. Epidemology: • High soil moisture • higher soil temperature. • The disease is further aggravated in ill-aerated soils with poor drainage having thick stand of the seedlings
  • 14. CULTURAL PRACTICES: • Field sanitation: 1. Collect and burn the infected plant debries of chilli. 2. Destroy self-grown solanaceous plant, other volunteer plants from the field and surrounding areas.
  • 15. Deep ploughing: 1. Post harvest ,plough the left over inoculum deep into the soil 2. Practice summer ploughing 3. solarization Cropping system: • Follow crop rotation. • Avoid growing of two solanaceous crop in a single year in and around the same field. Seed: • Use certified seed of varieties for different areas.
  • 16. 2.Use Seeds obtained from a diseases free crop. 3. Seed treatment with Bavistin or Agrosan GN @2g/kg of seeds Sowing: • Thin sowing to avoid crowding , • light sandy soils for nurseries or use of pure time sand dust mixture for raising the seedlings . • Under well drained conditions seedling can be transplanted into loamy soil also.
  • 17. NUTRITION Apply fully decomposed FYM or compost to balance the nutrient supply . Add NPK fertilizer where necessary to make up for a 90:60:60 kg/ha. Thinning and weeding: • Remove the weeds . • Rouge out the mixture and stay diseased plant as their serve as foci of infection.
  • 18. IRRIGATION • Provide only need based light irrigations VARIETAL: • Resistant/ tolerant genotypes against disease G-5,Pusa Jwala ,PantC-1 ,NP-46
  • 19. CHEMICAL: • seed treatment such as Thiram or captan @ 3g/kg of seeds for the better control of damping off . • Soil drenching with Captan or Thiram @0.2-0.5%
  • 20. • Also called dieback or ripe fruit rot of chillies. • The disease is one of the most serious disease of chilli . It occurs throughout India SYMPTOMS: • The pathogen cause two different types of symptoms on two different parts of the host. • Called die back because the disease cause necrosis of the tender twigs from the tip back wards. • The entire branch of the plant may wither away.
  • 21. • The twigs are water soaked to brown , become grayish white or straw coloured in advance stage of the disease. • Large number of black dots called acervuli are formed on the affected twigs. • The disease usually occurs on mature fruits as circular to elliptical sunken black margins and marketed with concentric rings. • Badly diseasd fruits turn straw coloured from normal red. • On this discoloured area ,numerous black dots are present. The diseased fruit may drop off prematurely.
  • 22.
  • 24. ETIOLOGY • Disease is caused by Colletotrichum capsici . • Mycelium septate ,colourless and inter and intracellular. • Hyphae develop the fruiting body called acervulli. • Acervulli consists of septate ,conidiophore and conidia. • The setae are septate and dark brown with light brown tip. • Conidiophores are mostly clubshaped ,under- branched, small and unicellular. • The conidia are hyaline ,unicellular ,sickle shaped and are produced singhly at the tip of the conidiophores.
  • 25. Primary source: Infected seeds and diseased crop debries . Secondary source :Conidia dispersed by rain splash and wind . It also infect tomato ,potato ,brinjal ,wild bringal are the other hosts. Epidemology: • Ideal temperature is 28-30°.the pathogen infects the host at a minimum temperature of 22-25°c. • Relative humidity : An RH of above 80% is ideal. Heavy prolonged dew deposition is important for dieback development.
  • 26. • Integrated of following practices manage the diseases effictely economically and in a most environment friendly manner. CULTURAL PRACTICES: • Field sanitation: 1. Collect and burn the infected plant debries of chilli 2. Destroy self-grown solanaceous plant, other volunteer plants from the field and surrounding areas.
  • 27. • Seeds obtained from a diseases free crop. • Treat the seeds with 2g of Thiram per kg of seeds before sowing for anthracnose of chillies. Thinning and weeding: • Weeds serve carriers of plant pathogens and high plant density favour development . • Remove the weeds once before and then after the irrigation. • Rouge out the mixture and stay diseased plant as their serve as foci of infection.
  • 28. IRRIGATION • Provide only need based light irrigations, high humidity, and frequent irrigation favour disease development. VARIETAL: • Resistant/ tolerant genotypes against major disease G-4 ,Pusa Jwala ,PantC-1 ,JCA 196.
  • 29. Chemical method: • For the management of Anthracnose chilli – • spray of Captafol @ 0.2% followed by copper oxy chloride @ 0.25% and Caebendizim @0.1% , two sprays at 10-15 days interval ,35-45 days after transplanting .
  • 30. Bacterial spots • Major disease of chillis in tropical and subtropical climates. This appears on leaves and fruits. • In Rajasthan the disease cause 8-16% loss in yield of fruits. SYMPTOM: • On leaves ,the first indication of the disease is appear of small , circular to irregular, water soaked area which appear as definite spots on the lower surface of the leaf. • As the colour changes from dark green to purplish grey with a black centre. • A narrow yellow halo may surround the spot.
  • 31. • In the wet weather , the leaves are too many ,the intervening tissues become dry and brown and the whole leaf dies.
  • 33. Etiology • Diseases caused by Xanthomonas campestris pv. Vesicatoria. • The bacterial cells are single ,straight rods 0.2-0.8*0.6-2.0 µm in size. • They are gram negative and motile by a single polar flagellum. • These bacterial are strict aerobes and optimum temperature for growth is 25-27°C.
  • 34. Disease cycle • It is a seed borne bacteria .10-15% seeds carry the bacteria which is enough to initiate the disease through seedlings. • The bacterium also subsists on infected plant debries, weeds and volunteer plants. • Spattering rains are the chief means of local dissemination of bacterial cells from the ooze developed on the affected parts. • The bacteria are also rapidly spread throughout the chilli. • movement of equipments and workers having come in contact with the diseased plants.
  • 35. • Field sanitation • Use diseases free seedlings and seeds. • Use of resistant varieties. • Avoid sprinkler irrigation. • Seed treatment with Agronson GN @2.5g/kg of seeds and Cersan @2.5g/kg of seeds were used for bacterial leaf spot of chilli . • Application of streptomycin sulphate 500ppm+COC 0.3%
  • 36. Leaf Curl of Chilli: • The leaf curl is very common wherever the crop is grown.it is a major initiation in successful cultivation of both hot and bell pepper. SYMPTOM: The most characteristics symptom are • the curling of leaves , small size , shortened internodes and general dwarfing of the plant which assumes a bushy appearance. • Leaves are pale color and roll downwards . Fruiting is stopped, if fruit are formed , they are small and deformed.
  • 37. • Causal agent: Tobacco leaf curl virus • The genome consists of a single circular ssDNA. • Whereas the genome of the other known whitefly transmitted Gemini virus consists of two ssDNA. • Gemini particles are paired and polyhedral .The member of this group are found predominantly in tropical area.
  • 38.
  • 40. Transmission: •The disease is transmitted by whitefly , Bemisia tobasci. • This vector spread the maximum diseases during the month of august to October. • The vector acquires the virus after feeding on an infected plant for at least 15-30 minutes. •There latent period of several hours after which the virus can be inoculated into a healthy plant.
  • 41. Management Cultural control • Nursery beds should be covered with nylon net or straw to protect the seedlings from viral infection. • Raise 2 -3 rows of maize or sorghum as a border crop to restrict the spread of vector. • Rogue out the infected plants as soon as they are noticed in the field.
  • 42. Bio-control agents • Different botonicals , animal products and other materials such as ash ,cow urine ,and dung ,fish meal,and milk etc. • Rhizobacteria(PGPR),leaf proteins,proline,and Trichoderma spp.reduce diseases in many plant species manipulating host plant’s physical and biochemical properties.
  • 43. • Use of milk in plant diseases management – it is the indigenous ancient tool against plant diseases. • Amino acids ie, proline, isoleucine and phenyl alanine in milk induce resistance against plant diseases. • potassium phosphate content strengthens host immune system against diseases. Chemical method: • Protect the crop in nursery bed from insect vectors(whitefly)by spraying Metasystox 1ml/lt of water at 10days interval.
  • 44. OTHER MINOR DISEASES • BACTERIAL SOFT ROT Erwinia carotovora pv. carotovora Symptom: • The internal tissue softens before eventually turning into a watery mass with a foul smell. • This problem is worst in wet weather beacouse the bacteria are rain splashed from the ground and on to the fruit. • Also started by insect injury.
  • 45. Management : • Keep plants off the ground • Controlling insects can help reduce the threat
  • 46. • BACTERIAL WILT: Pseudomonas solanacearum Symptom: • Start with wilting of the leaves. • After a few days a permanent wilt of the entire plant results,with no leaf yellowing. • Cutting roots or stems show milky streams of bacteria when they suspended in water. Management Plant clean and transplants and to remove diseased plants.
  • 47. • CERCOSPORA LEAF SPOT Crecospora capsici Symptom: • Worst under extended warm, wet conditions. • Characterized by small brown circular leaf lesions that have a watery appearance. • Excessive leaf drop may occur in common infestations.
  • 48. Management: • Use clean seed and crop rotation • Good air flow round plantsin sheltered areas will also help minimize this problem. • Fungicides like Hexaconazole 0.1% etc.
  • 49. • PHYTOPTHORA BLIGHT PHYTOPTHORA CAPSICI SYMPTOM: • Plants suffering from this condition often wilt and die, leaving brown stalks and leaves ,small • Poor quality fruits • If the fungus enters the roots . • Symptoms of the less serve leaf blight include brown or black spots that may kill a localized portion of the plant. • Affected areas are often borded with a white mold.
  • 50. • Wind borne fungus ,observed in wet water logged condition. • It is promoted by warm , wet weather Management: • Avoid excess watering and poorly drained soil. • Fungicides : Mancozeb@0.2%
  • 51. • The cultivated chilli suffers from number of fungal ,bacterial ,and viral diseases. • Among the fungal diseases damping off and anthracnose are the major devastring diseases causing considerable yield loss . • Environment play an effective role for development of these diseases. • Diseases perpetuate through plant debries in soil ,special fruiting body ,volunteer host and weeds year to year . • The diseases can be managed effectively and eco friendly by cultural practices and applying economic chemicals.