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Temporomandibular Dysfunctions – Part 2 History, Clinical Examination and Diagnosis
1. Temporomandibular
Dysfunctions – Part 2
History, Clinical Examination and
Diagnosis
Presented by:
Bishow Prakash Thakur
PG Resident
Department of orthodontics and Dentofacial orthopaedics
Peoples Dental College and Teaching Hospital
Kathmandu, Nepal
2. Contents
Part I
• Etiology of Functional Disturbances in the Masticatory
System
• Signs and Symptoms of Temporomandibular Disorders
Part II
• History of and Examination for Temporomandibular
Disorders
• Diagnosis of Temporomandibular Disorders
2
3. History and Examination
for TMDs
NOTHING IS MORE CRITICAL TO SUCCESS THAN BEGINNING
WITH ALL THE NECESSARY DATA
—Jeffrey P Okeson
4. Topics
• Screening History and Examinations
• History taking for TMDs
• Clinical examination
• Cranial nerve examinations
• Eye, ear and Neck examination
• Masticatory structures examinations
• Muscle examinations
• TMJ examinations
• Diagnostic tests
4
5. Screening History and Examinations
• Prevalence of TMDs is high
• Purpose is to identify patients with subclinical signs
• Should consist of several short questions that will help alert
clinician to any TMDs
• Asked personally by clinician or included in dental
questionnaire to be filled by patient completes prior to
treatment
5
6. Screening History
Questions should include:
1. Do you have difficulty and/or pain opening your mouth, for
instance, when yawning?
2. Does your jaw get “stuck,’’ “locked,’’ or “go out’’?
3. Do you have difficulty and/or pain when you are chewing,
talking, or using your jaws?
4. Are you aware of noises in jaw joints?
5. Do your jaws regularly feel stiff, tight, or tired?
6. Do you have pain in or about ears, temples, or cheeks?
7. Do you have frequent headaches, neck aches, or toothaches?
8. Have you had a recent injury to your head, neck, or jaw?
9. Have you been aware of any recent changes in your bite?
10. Have you previously been treated for any unexplained facial
pain or a jaw joint problem? 6
7. Screening Examinations
• Short screening examinations to identify any variation from
normal anatomy and functions
• Begins with:
• Inspection of facial symmetry
• Observation of jaw movements (restriction, irregular
movements)
• Palpation of structures of masticatory system (temporal ,
masseter muscle, and lateral aspect of TMJ)
• If positive findings if observed during screening history and
Examination, more thorough and detailed history and
examination for TMDs should be done
7
8. Detailed History Taking for TMDs
• History is key in making accurate diagnosis
• 70-80% of information for diagnosis of TMDs with pain comes
from detailed history
• Most often patient will provide essential information that
cannot be acquired from examination procedures
• Should include complete medical history, for eg: arthritic
condition
• Can be obtained in 2 ways:
1. Direct conversation
2. Written questionnaire
8
23. Clinical Examination
• Due complexity of head and neck pain disorders, certain
nonmasticatory structures should also be grossly examined
to rule out other possible disorders, if any abnormal
findings found, immediate referral needed
• Categorised into:
1. Cranial nerve examination
2. Eye examination
3. Ear examination
4. Cervical examination
5. Muscle examination
6. TMJ examination
23
24. 1. Cranial Nerve examination
24
Olfactory Nerve:
• Ask patient to detect differences between odour of
pipperment, vanilla, chocolate
• Before examination, any obstruction in nose should be
assessed by asking to exhale nasally into mirror and look
for fogging
25. 1. Cranial Nerve examination
25
Optic Nerve:
• Sensory fiber in retina
• Cover one eye and read sentence
• Visual field should be assessed by standing behind patient
and slowly bringing fingers from behind into view
• Normal : No variation in right and left eyes
26. 1. Cranial Nerve examination
26
Oculomotor, Trochlear and Abducens nerves:
• 3rd, 4th and 6th CN, Motor fiber to extraocular muscles
• Patient should follow examiner’s finger as it make “X”
(both eye should move smoothly and similarly)
• Pupilary reflex , Accomodation reflex, Consensual light
reflex
27. 1. Cranial Nerve examination
27
Trigeminal nerve:
• 5th CN, Motor (Muscle of
mastication), Sensory ( face, scalp,
nose and mouth)
• Sensory: cotton tipped applicator
B/L in forehead, cheek and lower
jaw
• Motor: Clench teeth and examiner
should feel masseter an temporal
muscles
28. 1. Cranial Nerve examination
28
Facial nerve:
• 7th CN, Motor (Muscle of facial expression), Sensory
(anterior 2/3rd of tongue)
• Sensory: Distinguish between sugar and salt using just tip
of tongue
• Motor: raise both eyebrows, smile, and show the lower
teeth
29. 1. Cranial Nerve examination
29
Vestibulocochlear nerve:
• 8th CN, senses balance and hearing
• Balance: walk heel to toe along straight line
• Hearing: rubbing strand of hair between fingers near
patient ear and noting any difference
30. 1. Cranial Nerve examination
30
Glossopharyngeal and Vagus nerve:
• 9th and 10th CN, supply fibers to back of throat
• Patient is asked to say “ah” and soft palate is observed
for symmetrical elevations
• Gag reflex is tested by touching each side of pharynx
31. 1. Cranial Nerve examination
31
Spinal accessory nerve:
• 11th CN, supply fibers to
trapezius and
Sternocleidomastoid
• Trapezius: asking patient to
shrug shoulders against
resistance
• Sternocleidomastoid: move
head first to right and then to
left against resistance
32. 1. Cranial Nerve examination
32
Hypoglossal nerve:
• 12th CN, supply fibers to tongue
• Patient is asked to protrude tongue; any uncontrolled or
consistent lateral deviation is noted
33. 2. Eye examination
• Patient is questioned about vision and any recent changes,
requiring treatment
• Simple techniques as used in cranial nerve examination will
be suffcient in testing gross vision
• Any diplopia or blurriness of vision is noted
• Pain felt in or around eyes is noted
33
34. 3. Ear examination
• 70% of patients reporting with
TMJ pain also complain of ear
discomfort
• Infection of otitis externa should
be assessed which is painful
• Dentist’s role should be merely to
attempt to rule out gross ear
disease if found proper referral
should be done
34
35. 4. Cervical examination
• Cervicospinal pain and dysfunction can refer pain to
masticatory apparatus
• Mobility of neck is examined for range and symptoms
• 70 degrees of rotation in right and left direction
• Extension 60 degrees and flexion downward 45 degrees
• 40 degrees during bending right and left
35
36. 4. Cervical examination
• Any pain and limitation of movement carefully investigated
to determine whether its source is muscular or vertebral
• When patients with limited range of movement can be
passively stretched to a greater range, source is usually
muscular
• Patients with vertebral problems cannot normally be
stretched to a greater range
• If craniocervical disorder is suspected, proper referral for
more complete (cervicospinal) evaluation is needed as
craniocervical disorders are closely associated with TMDs
36
37. 5. Muscle Examination
• Frequent clinical sign of compromised muscle tissue is pain
• Most often muscles of mastication become compromised
through increased activity
• In early stages, myalgia is noticed only during function of
muscle
• If hyperactivity continues, it can result in a dull, aching pain
that often radiates over entire muscle
• Later, pain can eventually become severe limiting
mandibular function
• Muscle examination can be done by:
A. Direct Palpation: Temporalis, Masseter, SCM, Posterior
cervical
B. Functional Manipulation: medial and lateral pterygoid 37
38. A. Direct Palpation
• Deformation of compromised muscle tissue by palpation
can elicit pain
• Firm thrust of 1 or 2 seconds in small circular motion is
given and asked for discomfort of pain
When a muscle is palpated, the patient’s response is placed
in one of 4 categories:
i. 0 : No pain or tenderness
ii. 1 : Uncomfortable ( Tenderness or soreness)
iii. 2 : Definite discomfort or pain
iv. 3 : Evasive action or eye tearing or verbalizes no to
palpate again
38
39. A. Direct Palpation
• Not only generalized muscle tenderness and pain but also
presence of any trigger points should also be assessed
• Generalized muscle pain may not exist in muscle with
trigger points
• Thus, during palpation generalized muscle pain and trigger-
point pain should be differentiated
• Done for Temporalis, Masseter, Sternocleidomastoid, and
posterior cervical (Splenius capitis and Trapezius)
39
40. Temporalis
• Divided into 3 functional areas:
i. Anterior : Above zygomatic arch and
anterior to TMJ, Vertical fibers
ii. Middle : Above TMJ and superior to
zygomatic arch, Oblique fibers in
lateral aspect of skull
iii. Posterior: Above and behind ear,
Horizontal fibers
40
41. Temporalis
• If a trigger point is located, it should be identifed along with
any pattern of referred pain
• Palpation of its tendon should also be done , because
Temporalis tendinitis may cause referred pain behind eye
(retro orbital Pain)
41
42. Tendon of Temporalis
• Temporalis tendon is palpated by
• Placing finger of one hand intraorally on anterior border of
ramus and finger of other hand extra orally on same area
• Intraoral finger is moved up anterior border of ramus until
coronoid process and tendon are palpated
42
43. Masseter
• Palpation at origin and insertion
• Deep masseter: just anterior to
TMJ on zygomatic arch
• Superficial masseter: inferior
attachment on inferior border of
ramus
43
44. Sternocleidomastoid
• No direct effect on mandibular
function
• Palpated entire length from mastoid
fossa behind ear (insertion) to
clavicle (origin)
• Any trigger point present or not
44
46. Posterior cervical muscle
Trapezius muscle:
• Major purpose is to search for “active trigger points”
• Commonly has trigger points that refer pain to face
• Palpated from behind SCM inferolaterally to shoulder
46
47. B. Functional Manipulation
• For muscles that are impossible or nearly impossible to
palpate manually
• For years an intraoral technique was suggested for
palpating the lateral pterygoid muscle, but this has not
proven effective, So functional manipulation is needed
• Principle: “as a muscle becomes fatigued and symptomatic,
further function will elicit more pain”
47
Johnstone DR, Templeton M: The feasibility of palpating the lateral pterygoid muscle, J
Prosthet Dent 44(3):318 323, 1980
Stratmann U, Mokrys K, Meyer U, et al: Clinical anatomy and palpability of the inferior
lateral pterygoid muscle, JProsthet Dent 83(5):548–554, 2000
48. Inferior Lateral pterygoid
Contraction:
• Causes protrusion and opening of
mandible
• Ask patient to protrude against
resistance and assess whether it will
increase pain or not
48
Stretching:
• ILP stretches during maximum intercuspation, so pain
should be during clenching
• When tongue blade is placed between posterior teeth,
maximum intercuspation cannot be reached; so ILP donot
fully stretch,so biting on separator willnot increase pain
but may even decrease or eliminate it
49. Superior Lateral Pterygoid
Contraction:
• Elevator muscle so during
clenching will increase pain
• If tongue blade is placed, on
clenching pain will increase
49
Stretching:
• Clenching will increase pain (seen in other elevators also)
• SLP pain can be differentiated from elevator pain by asking
patient to open wide:
• If pain, then SLP and other elevators are involved
• If no pain, then pain of clenching is from SLP
50. Medial Pterygoid
Contraction:
• Elevator muscle so clenching will increase pain
• Biting on tongue blade: still increase pain
Stretching:
• Opening of mouth increases pain
Thus , all information needed is obtained by having
i. Wide mouth opening
ii. Protrude mandible against resistance
iii. Clench teeth, and then
iv. Bite on a separator
50
52. Intracapsular disorders
• If source of pain is Intracapsular, it can confuse previously
mentioned functional manipulation
• Functional manipulation not only affects muscle, but it also
increases interarticular pressure and moves condyle
creating confusion
52
Fig: When a patient bites unilaterally on a hard
substance, joint on biting side has a sudden reduction in
interarticular pressure while opposite joint has a sudden
increase in pressure. Therefore a patient with
intracapsular pain will often report increased pain when
biting on the teeth; but when a tongue blade is placed
between the teeth, the pain is reduced. Biting on
opposite side will often increase pain in involved joint
Intracapsular Vs ILP disorders is evaluated
by “unilateral biting on tongue blade
followed by Protrusion”
54. Maximal interincisal distance
• Normal interincisal opening 53-58 mm
• Even 6 year old child has 40mm or more mouth opening
• If no pain, Maximal comfortable opening = Maximum
opening
54
55. Restricted mouth opening
• Less than 40 mm
• If restricted opening, it is helpful to test “end feel”
55
• Soft end feel : Associated
with muscle-induced
restriction
• Hard end feel :
Associated with
intracapsular sources
(e.g., a disc dislocation)
56. Lateral movement of mandible
• Any lateral movement less than 8 mm is recorded as a
restricted movement
• Protrusive movement is also evaluated in a similar manner
56
57. Deviation Vs Deflection
• Deviation: Any shift of jaw
midline during opening
that disappears with
continued opening
• Due to disc derangement
57
• Deflection: Any shift of midline
to one side that becomes
greater with opening and does
not disappear at maximal
opening
• Due to restricted movement of
joint
58. Examination of TMJ
• Examined for Pain and Dysfunction
• Radiographs and Other imaging techniques can be used
TMJ PAIN:
• Palpation during static and dynamic movement
58
Fig: Palpation of the TMJ.
A, Lateral aspect of the joint with the
mouth closed
59. Examination of TMJ
TMJ PAIN:
59
Fig: B Lateral aspect of the joint during
opening and closing
Fig: C, With the patient’s mouth fully
open, the clinician moves a finger behind
the condyle to palpate the posterior
aspect of the joint
60. Examination of TMJ
TMJ DYSFUNCTION:
• Joint sounds:
• Click (Single sound of short duration)
• Pop (Single sound relatively loud)
• Crepitations (Gravel like grating, associated with
osteoarthritic changes in articular surface)
• Sound occurs during opening (single click) or closing
(reciprocal click)
Absence of sounds does not always mean normal disc
position. 15% of silent, asymptomatic joints were found to
have disc displacements on arthrogram
(Westesson PL, Eriksson L, Kurita K: Reliability of a negative clinical temporomandibular
joint examination: prevalence of disk displacement in asymptomatic temporomandibular
joints, Oral Surg Oral Med Oral Pathol 68:551–554, 1989.) 60
61. Additional Diagnostic Tests
• Used only to gain additional information and never to
establish diagnosis
• Includes:
A. Radiographic techniques
B. MRI
C. Bone Scanning
D. Mounted Cast
E. Mandibular Tracking devices
F. Sonography
G. Vibration analysis
H. Thermography
61
62. • Complicated by several anatomic and technical
circumstances that superimposes joint
• Pure lateral view of condyle is impossible to achieve with
conventional radiographs
• Only CT can view lateral view of condyle
• 4 basic Radiographic technique:
1. Panoramic
2. Lateral Transcranial
3. Transpharyngeal
4. Transmaxillary (Antero-Posterior)
A. Radiographic techniques
62
63. Panoramic View
• Good screening of condyle as it results in minimum
superimposition over condyle
• Limitations: to view condyle mouth should be opened
maximally, so in patient with restricted mouth opening
superimposition occurs
63
64. Panoramic View
• Since panoramic radiograph is infracranial view, lateral pole
of condyle becomes superimposed over condylar head
• Area that appears to represent superior subarticular
surface of condyle is actually only subarticular surface of
medial pole
64
Fig: Area that appears to be superior subarticular surface of condyle is actually
medial pole. Lateral pole is superimposed inferiorly over body of condyle. Fossa is
also superimposed over condyle, which complicates interpretation of radiograph
65. Lateral Transcranial View
• Good visualization of both condyle and fossa
• X-rays are directed inferiorly across skull (above midface) to
contralateral TMJ and recorded during maximum
intercuspation and maximum opening
65
Fig: Transcranial projection.
A, Teeth together.
B, Maximal open position.
C, The condyle can be visualized
in the fossa with the articular
eminence directly anterior. The
relatively round (dark) area
posterior to the condyle is the
external auditory meatus.
D, The condyle has translated out
of the fossa during an opening
movement.
66. Lateral Transcranial View
• Since x-rays pass downward across skull, this angulation
superimposes medial pole of condyle below central
subarticular surface and lateral pole
• It is more acceptable than infracranial view for visualizing
articular fossae
66
Fig: Transcranial projection superimposition. The area that appears to be superior
subarticular surface of condyle is actually lateral pole. Medial pole is superimposed inferiorly
over body of condyle. In this projection fossa is not superimposed over condyle; thus a
clearer view of condyle is usually obtained
67. Transpharyngeal
• Similar to Panoramic view
• X ray notch directed below angle of mandible or through
sigmoid notch
• Mandibular fossa is not usually visualized as well as in this
view (same as OPG)
67
68. Transmaxillary (AnteroPosterior) view
• Obtained from anterior to posterior with mouth wide open
and condyles translated out of fossae
• If correctly taken, good view of superior subarticular bone
of condyle, medial and lateral poles is obtained
• Excellent view for evaluating a fracture in neck of condyle
68
69. Tomography
• It utilizes controlled movement of head of x-ray tube and
film to obtain a radiograph of desired structures that
deliberately blurs out other structures
• It is neither infracranial or transcranial projections but true
lateral projections
69
• Helpful in evaluating articular
surface of condyle
• Advantages:
• More accurate than OPG and
transcranial view
• True sagittal view
• Condylar position in fossa is
evaluated accurately
70. Tomography
70
Fig: A lateral tomography view of the TMJ. A, Closed position. B, Open position. C, An AP
tomogram. Note the fine clarity provided by tomography
Disdvantages:
• High cost
• Hight exposure to radiation
71. Computed Tomography
• Developed in past decade
• CT scanners produce digital data after measuring extent of
x-ray transmitted through various tissues, data is
transformed into density scale and used to generate or
reconstruct visual image
• Can reconstruct 3D image
71
72. Cone beam tomography
• Allows visualization of condyle in multiple plane
• Can image hard and Soft tissue both, disc condyle
relationship can be obtained
• Disadvantages: Expensive , more radiation exposure (In
CBCT, radiation exposure is less comparect to CT)
72
Fig: 3D image reconstructed from a cone beam image. These 3D
images can be rotated on computer screen so that clinician can
visualize precise area of interest. (Courtesy of Dr. Allan Farmer
and Dr. William Scarf, Louisville, KY.)
73. B. Magnetic Resonance Imaging
• Gold standard for evaluation of soft tissue of TMJ
• Uses strong magnetic field to create changes in energy
level of soft tissue molecules (principally hydrogen ions),
These changes in energy level create image similar to that
of a CT scan
• Better visualization of soft tissue than in CT scans
73
74. B. Magnetic Resonance Imaging
• Advantage: No harmful effects
• Disadvantage:
• Expensive
• Technology varies from site to site changing quality of
images
• Static image
• Cine MRI is beginning to provide information on disc and
joint movements
74
75. C. Bone scanning
• Helps to know if there is active inflammatory process in
TMJs
• Obtained by injecting a radiolabeled material into
bloodstream that concentrates in areas of rapid bone
turnover, and emission image is taken
75
Fig: Bone scan of head and neck reveals a high concentration of radiolabeled material
present in TMJ and maxillary regions. This finding suggests increased cellular activity
in these regions
• SPECT (Single photon emission
computed tomography) is similar
technique to identifiy increased bone
activity
76. D. Mounted cast
• To assess occlusal condition
• Semi adjustable or fully adjustable articulator needed
• Indicated only when future dental treatment is planned
(i.e., prosthodontics, orthodontics, etc)
• Provide better visualization of occlusal contacts (especially
in lingual view) and remove influence of neuromuscular
control from eccentric movements
• Should be mounted using accurate Facebow transfer and
CR record
76
77. E. Mandibular Tracking devices
• Exact movement of mandible can be recorded
• Can be used to diagnose and monitor treatment of TMDs
• Should be used only in conjunction with history and
examination findings
77
• No evidence to suggest that
sensitivity and specifcity of
these devices are reliable
enough to be used for
diagnosis and management of
TMD
78. F. Sonography
• Technique to record and graphically demonstrate joint
sounds
• Doppler Ultrasonography uses ultrasound echo recordings
• Although these devices may accurately record joint sounds,
significance of these sounds has not been well established
• It does not provide any additional diagnostic information
over manual palpation or stethoscopic evaluation
78
79. G: Vibration analysis
• Was used in diagnosing intracapsular TMD—internal
derangements in particular
• Measures minute vibrations made by condyle as it
translates and has been shown to be reliable
• It diagnoses 25% of normal joints as derangements, and
misclassifies many deranged joints as normal, especially if
joint sounds are not audible or if derangement has
advanced to nonreducing stage
• Thus due to high cost and the results, it is not test of
choice for suspected internal derangement 79
80. H. Thermography
• Technique that records and graphically illustrates surface
skin temperatures
• It has been suggested that normal subjects have bilaterally
symmetrical thermograms. From this concept some have
suggested that thermograms that are not symmetrical
reveal a problem, such as a TMD
• Sensitivity and specifcity of identifying myofascial trigger
points with thermography has not been demonstrated to
be reliable
80
83. Diagnosing pain disorders
• Primary pain: Source and site of pain is same
• Referred pain: Heterotopic, patient will have attention to
site of pain
• On Local provocation,
• If pain doesn’t increase: Referred pain
• If pain increases : Source and site is same, Primary pain
83
Fig: Local provocation of site
of pain does not increase pain
Fig: Local provocation of source of pain
increases the pain not only at source
but may also increase pain at site
84. Diagnosing pain disorders
• Sometime it is difficult to differentiate “site” from
“source” of pain : Anesthetic blockade recommended
84
Fig: Local anesthesia at site of pain
fails to reduce pain
Fig: Local anesthesia at source of
pain reduces pain at source as well as
site
85. Diagnosing pain disorders
Thus 4 rules to summarize examination techniques used to
differentiate primary pain from referred pain:
1. Local provocation of site of pain does not increase pain
2. Local provocation at source of pain increases pain not
only at source but also at site
3. Local anesthetic blocking of site of pain does not
decrease pain
4. Local anesthetic blocking of source of pain decreases
pain at source as well as site
85
86. Diagnostic Analgesic Blocking
INDICATIONS:
• To diagnose primary from secondary (referred) pain
• Therapeutic value in some pain disorders
• Educate patients about site and source of pain
GENERAL RULES:
1. Clinicians should have sound knowledge of anatomy in that
region
2. Sound knowledge of pharmacology of drugs used
3. Injecting into inflamed or diseased tissue should be avoided
4. Avoided in patient with bleeding disorders and anti
coagulatant therapy
5. Strict asepsis should be followed
6. Always aspiration before injecting the solution should be
done
86
87. Analgesic blocks
Diagnostic and therapeutic anesthetic blocks are divided
into three types according to structures targeted:
1. Muscle injections
2. Nerve block injections
• Dental Block
• Auriculotemporal nerve block
• Infraorbital nerve block
3. Intracapsular injections
87
88. Muscle injections
• Valuable in determining source of a pain disorder
• Trigger point can be injected with local anesthetic, which
results in shut down of pattern of referred pain
88
Procedure:
A. Trigger point (tight band) is located,
finger is moved up and down, until
most painful area is located
B. Tissue over trigger point is cleaned
with alcohol, trapped between two
fingers so that when needle is placed,
tight band will not move away
89. Muscle injections
89
Procedure:
C. Needle tip is inserted into tissue
superficial to trigger point and
advanced to depth of tight band
D. Initial anesthetic solution should be
deposited and “fanning” of needle
tip is done and solution deposited
E. Once injection has been completed,
needle is withdrawn and sterile
gauze is held over injection site
with slight pressure for 10 to 20 s to
ensure hemostasis
91. Nerve Blocks
• Short-acting local anesthetic should be used without
vasoconstrictor
• Therapeutically, in chronic pain, Long acting LA can be
used
DENTAL BLOCKS:
These blocks are useful in separating dental pain
from muscle or joint pain since they block only dental
structure
• Inferior alveolar nerve
• Mental nerve
• Posterior superior alveolar
• Infiltrations
91
92. Nerve Blocks
AURICULOTEMPORAL NERVE:
• Very important nerve block with which all orofacial pain
clinicians should become very familiar
• TMJ innervation :
• Primary innervation Auriculotemporal nerve
• Secondary innervation Masseteric and Deep temporal
nerves
92
93. Nerve Blocks
AURICULOTEMPORAL NERVE:
Procedure:
93
Fig A: Tissue at site of
injection is thoroughly
cleansed
Fig B: Drawing showing
position of nerve as it
transverses around posterior
aspect of condyle. It also
demonstrates proper needle
placement
Fig C: Needle is placed
slightly anterior to junction
of tragus and earlobe and is
advanced until posterior
neck of condyle is felt
94. Nerve Blocks
AURICULOTEMPORAL NERVE:
Procedure:
94
Fig D: Needle is then repositioned in
more posterior direction until its tip
is able to pass posterior to neck of
condyle
Fig E: Once needle tip has passed beyond neck of
condyle, syringe is again positioned in a more
anterior direction and tip is inserted behind neck
of condyle, total depth of needle is approx 1 cm
The syringe is aspirated; if no blood , anesthetic
solution is deposited
* Placement of needle in this manner will minimize the risk of anesthetizing facial nerve
96. Intracapsular Injection
• Indicated for therapeutic purpose to introduce some
medication to joint structures
• Normally, superior joint space is target because it is
largest joint space and is simplest to locate consistently
96
Fig A: Joint can be entered by locating lateral pole of condyle
(asking patient to open and close mouth), Once pole is
located, patient is asked to open slightly and examiner
palpates directly above to locate zygomatic arch. Tissue is
cleaned and tip of needle is placed just below zygomatic arch
and slightly behind posterosuperior aspect of condyle
Fig B: Needle is angulated slightly
anterosuperiorly to avoid retrodiscal
tissues. Once capsule has been
penetrated, tip of needle will be in
superior joint space
97. Keys to making differential diagnosis
• Besides Pain, most common masticatory problems
include:
• Masticatory muscle disorder
• Intra capsular joint disorder
• It is extremely important that they be differentiated, since
their treatments are quite different
• The clinician who cannot routinely separate them is likely
to have relatively poor success in managing TMDs
97
98. Keys to making differential diagnosis
There are 7 areas of information acquired during history
and examination that will assist in separating them:
1. History
2. Mandibular Restriction
3. Mandibular interference
4. Acute malocclusion
5. Loading of joint
6. Functional manipulation, and
7. Diagnostic anesthetic blockade
98
99. 1. History
• History is always helpful in distinguishing joint from
muscle disorders
• Any Trauma present in past or not
• Muscle disorders appear to fluctuate and cycle from
severe to mild with no apparent initiating event
• Muscle problems are more closely related to changes in
levels of emotional stress; therefore periods of total
remission are common when stress is low
99
100. 2. Mandibular Restriction
During mouth opening:
• Intracapsular problem (dislocated disc without reduction):
• 25 to 30 mm, presence of “Hard End Feel”
• Muscle Problem:
• 8 to 10 mm, presence of “Soft end feel”
During eccentric movement:
• Intracapsular:
• Limited contralateral eccentric movement
• Normal ipsilateral movement
• Muscle:
• Normal range of eccentric movement exists
100
101. 3. Mandibular Interference
• Any deviation or deflection
Deviation
• If speed of opening alters location of deviation:
Disc displacement with reduction
• If speed of opening does not alter location of deviation:
Structural incompatibility
• Subluxation (Intra capsular, not necessarily pathologic)
101
102. Movement Capsular Muscular
Protrusive - Ipsilateral side No deflection
Lateral
Eccentric
- Restricted to contralateral movement
- Normal Ipsilateral movement
No deflection
3. Mandibular Interference
Deflection:
Can be seen in
• Intracapsular disorders
eg: Disc dislocation without reduction,
Adhesion problem
• Myospasm of unilateral elevator
muscles
eg masseter
102Table to differentiate between deflection due to intracaspsular or muscular defect
103. 3. Mandibular Interference
Deflection:
• If deflection is due to shortened muscle, direction in
which mandible moves will depend on position of
involved muscle with respect to joint
• If muscle (i.e., masseter or temporalis) is lateral to joint,
deflection will be toward involved muscle
• If muscle (i.e., medial pterygoid) is medial to joint,
deflection will be away from involved muscle
103
104. 4. Acute malocclusion
• ILP spasm: Condyle will be brought slightly forward in
fossa on involved side resulting in disocclusion of
ipsilateral posterior teeth & heavy contact on contralateral
canine
104
• Elevator muscles spasm: Patient report
feeling teeth “suddenly don’t fit right,”
clinically may be difficult to visualize
any change
• Intracapsular disorder : If disc is
suddenly displaced anteriorly, thicker
posterior border may be superimposed
between condyle and fossa and cause
sudden increase in discal space,
resulting clinically as a loss of ipsilateral
posterior tooth contact
105. 5. Loading of Joint
• When there is orthopaedic stability, Loading of structures
with manipulative force does not produce pain in healthy
joint
• If Pain is produced, one should be suspicious of
intracapsular source of pain
105
106. 6. Functional Manipulation
• No pain during Functional manipulation: No muscle
disorder
106
• Biting on Tongue blade unilaterally:
• Interarticular pressure and pain is
reduced ipsilateral TMJ
For eg: If Right side TMJ pain,
clenching normally will increase pain,
• If tongue blade kept right side and
bite, it will decrease pain
• But if tongue blade kept on left side,
increased loading in right side will
result in pain in preauricular region
in right side
107. 6. Functional Manipulation
In primary muscle pain (eg: right-side masseter pain)
107
• When asked to clench teeth , right
masseter will produce pain
• If tongue blade is placed on right
molar and bite on it, pain will also
be increased due to increase
activity of painful masseter muscle
• If tongue blade is moved to left side
and bite on it, there will be
reduction in pain on right
108. 7. Diagnostic Anesthetic Blockade
• Indicated for patients in whom preceding 6 procedures
didn’t help in making a differential diagnosis
• Anesthetic blocking of auriculotemporal nerve can quickly
rule in or out an intracapsular disorder
• Dentist working with pain disorders should be very
familiar with this injection technique and use it without
hesitation to assist in diagnosis
108
109. Classification of Temporomandibular disorders
109
Adapted from Okeson JP: Orofacial Pain: Guidelines
for Assessment, Diagnosis, and, Management, 3rd
ed. Chicago: Quintessence;1996:45-52.
* The code number after each disorder has been
established by American Academy of Orofacial Pain
in cooperation with International Headache Society
110. Conclusion
• Methods to aid in identification and diagnosis of TMDs
has been presented which does not include all disorders
that cause pain and dysfunction of head and neck
• Diseases of Dental origin, vascular origin (e.g., arteritis or
migraine headache), neural origin (e.g., neuralgias,
neuropathies), craniocervical disorders as well as ear and
eye diseases have not been addressed
• This classification is useful in identifying common
functional disturbances of masticatory system
• When a patient’s problems do not fit into one of these
categories, more extensive examination procedures are
indicated
110
111. References
• Jeffrey P. Okeson, Management of Temporomandibular
disorders and occlusion, 2013, 7th edition
• Costen JB: Syndrome of ear and sinus symptoms
dependentupon functions of the temporomandibular
joint, Ann Otol RhinolLaryngol 3:1–4, 1934
• Pullinger AG, Seligman DA, Gornbein JA: A multiple logistic
regression analysis of the risk and relative odds of
temporomandibular disorders as a function of common
occlusal features, J Dent Res 72(6):968–979, 1993
• Ware JC, Rugh JD: Destructive bruxism: sleep stage
relationship, Sleep 11(2):172–181, 1988
111
112. References
• Rugh JD, Solberg WK: Electromyographic studies of bruxist
behavior before and during treatment, J Calif Dent Assoc
3(9): 56–59, 1975
• Kieser JA, Groeneveld HT: Relationship between juvenile
bruxing and craniomandibular dysfunction, J Oral Rehabil
25(9):662–665, 1998
• Johnstone DR, Templeton M: The feasibility of palpating
the lateral pterygoid muscle, J Prosthet Dent 44(3):318
323, 1980
• Stratmann U, Mokrys K, Meyer U, et al: Clinical anatomy
and palpability of the inferior lateral pterygoid muscle,
JProsthet Dent 83(5):548–554, 2000
112
113. References
• Westesson PL, Eriksson L, Kurita K: Reliability of a negative
clinical temporomandibular joint examination: prevalence
of disk displacement in asymptomatic temporomandibular
joints, Oral Surg Oral Med Oral Pathol 68:551–554, 1989
113
When pain has a stimulating or exciting effect on the patient, it is classifed as bright.
When the pain has a depressing effect that causes the patient to withdraw to some extent, it is classifed as dull
Bright, tingling pain is classifed as a pricking sensation, especially when it is mild and stimulating.
Superfcial discomfort that does not reach pain threshold intensity may be described as itching.
As intensity increases, it may take on a pricking, stinging, aching, or burning quality.
Deep discomfort that does not reach pain threshold intensity may be described as a vague, diffuse sensation of pressure, warmth,or tenderness.
As intensity increases, the pain may take on a sore, aching, throbbing, or burning quality.
When the discomfort has an irritating, hot, raw, caustic quality, it is usually described as burning.
Most pains have an aching quality. Some noticeably increase with each heartbeat and are described as pulsating or throbbing
Temporal behavior reflects the frequency of the pain as well as the periods between episodes of pain.
If the suffering distinctly comes and goes, leaving pain-free intervals of noticeable duration, it is classifed as intermittent. If such pain-free intervals do not occur, it is classifed as continuous.
Intermittency should not be confused with variability, in which there may be alternating periods of high- and low-level discomfort.
Intermittent pain implies the occurrence of true intermissions or pain-free periods during which comfort is complete.
This temporal behavior should not be confused with the effect of medications that induce periods of comfort by analgesic action. When episodes of pain,whether continuous or intermittent, are separated by an extended period of freedom from discomfort only to be followed by another similar episode of pain, the syndrome is said to be recurrent
If the patient is able to defne the pain to an exact anatomic location, it is classifed as localized pain.
If such description is less well defned and somewhat vague and variable anatomically, it is termed diffuse pain.
Rapidly changing pain is classifed as radiating.
A momentary cutting exacerbation is usually described as lancinating.
More gradually changing pain is described as spreading; if it progressively involves adjacent anatomic areas, the pain is called enlarging.
If it changes from one location to another, the complaint is described as migrating.
Referred pain and secondary hyperalgesia are clinical expressions of secondary or heterotopic pain
Pain that the patient describes as mild is generally not associated with any display of visible physical reactions.
Severe pain is associated with signifcant reactions of the patient to provocation of the painful area.
Visual analogue scale, however, is not appropriate for comparing different patients, since pain is a very personal experience and varies greatly from patient to patient, but it can be helpful in comparing initial pain with pain at follow-up appointments to evaluate the success or failure of therapies
Sensations such as hyperesthesia, hypoesthesia, anesthesia, paresthesia, or dysesthesia should be mentioned
Motor changes expressed as muscular weakness, muscular contractions, or actual spasm should be recognize
Autonomic symptoms: Ocular symptoms may include lacrimation, injection of the conjunctivae, pupillary changes, and edema of the lids. Nasal symptoms include nasal secretion and congestion. Cutaneous symptoms have to do with skin temperature, color, sweating, and piloerection. Gastric symptoms include nausea and indigestion
A flowing type of pain, even though variable in intensity or distinctly intermittent, is described as steady.
Such pain is to be distinguished from paroxysmal pain, which characteristically consists of sudden volleys or jabs.
The volleys may vary considerably in both intensity and duration. When they occur frequently, the pain may become nearly continuous
Sleep: There are relationships between some pain conditions and the quality of the patient’s sleep. Ask about pattern of sleep, Patients should be asked how long it takes for them to fall asleep; how often they wake during the night; whether, if they wake up during the night, they can then get back to sleep easily; and whether they feel rested in the morning
Litigation: During the interview it is important to inquire if the patient is involved in any form of litigation related to the pain complaint. This information may help the clinician better appreciate all the conditions surrounding the pain complaint. The presence of litigation does not directly imply a wish to achieve secondary gains, but this may be present
Disability: A similar condition may exist with disability. If the patient is either receiving or applying for disability that will allow him orher not to work yet receive compensation, it may have a powerful effect on the individual’s desire to get well and return to work. Secondary gains may have a direct effect on the success or failureof treatment
Often the general practitioner may not have immediate access to psychological evaluation support. In this instance the practitioner may elect to use the TMJ scale
The pupils should be of equal size and rounded and should reactto light by constricting. The accommodation reflex is tested byhaving the patient change focus from a distant to a near object.The pupils should constrict as the object (the fnger) approachesthe patient’s face. Not only should they both constrict to directlight, but each should also constrict to light directed in the othereye (consensual light reflex)
Sensory input is tested by lightly stroking the face with a cotton-tipped applicator bilaterally in threeregions: forehead, cheek, and lower jaw (Figure 9-5). This willgive a rough idea of the function of the ophthalmic, maxillary,and mandibular branches of the trigeminal nerve. The patientshould describe similar sensations on each side.
Gross motor input is tested by having the patient clench the teeth while the examiner feels both themasseter and temporal muscles (Figure 9-6). The muscles shouldcontract equally bilaterally
The proximity of the ear to the TMJ and muscles of mastication as well as their common trigeminal innervation creates a frequent condition for referral of pain. Although few of these patients have actual ear disease, when they do it is important to identify it and refer them for proper treatment
As the number and duration of contractions increase, so also do the physiologic needs of the muscle tissues. Increased muscle tonicity or hyperactivity, however, can lead to a decrease in blood flow to the muscle tissues, lowering the inflow of nutrient substances needed for normal cell function while also accumulating metabolic waste products. This accumulation of metabolic waste products and of other algogenic substances is thought to cause the muscle pain. It is now appreciated that the central nervous system can contribute to the myalgia associated with neurogenic inflammation
Palpation of muscle is accomplished mainly by the palmar surface of the middle fnger, with the index fnger and forefinger testing the adjacent areas. Soft but frm pressure is applied to designated muscles, with the fngers compressing the adjacent tissues in a small circular motion. A single frm thrust of 1 or 2 s duration is usually better than several light thrusts. During palpation the patient is asked whether it hurts or is just uncomfortable
When trigger points are located, an attempt should be made to determine if there is any pattern of pain referral. Pressure should be applied to the trigger point for 4 to 5 s, and the patient is then asked if the pain is felt to radiate in any direction. If a pattern of referred pain is reported, it should be noted on a drawing of the face for future reference. Patterns of referred pain are often helpful in identifying and diagnosing certain pain conditions
If uncertainty arises regarding the proper finger placement, the patient is asked to clench the teeth. The temporalis will then contract and the fibers should be felt beneath the examiner’s fingertips
It is helpful to be positioned behind the patient and to use the right and left hands to palpate respective muscle areas simultaneously
Classified as 0,1,2,3
The fbers of the temporalis muscle extend inferiorly to converge into a distinct tendon that attaches to the coronoid process of the mandible. It is common for some TMDs to produce a temporalis tendinitis, which can create pain in the body of the muscle as well as referred pain behind the adjacent eye (retro-orbital pain).
Palpation is important because it often becomes symptomatic with TMDs
The posterior cervical muscles (trapezius, longissimus [capitis and cervicis], splenius [capitis and cervicis], and levator scapulae) do not directly affect mandibular movement; however, they do become symptomatic during certain TMDs and therefore are routinely palpated
They originate at the posterior occipital area and extend inferiorly along the cervicospinal region. Because they are layered over each other, they are sometimes diffcult to identify individually
During functional manipulation, each muscle is contracted and then stretched. If the muscle is a true source of pain, both activities will increase the pain
Stretching. As with the inferior lateral pterygoid, stretchingof the superior lateral pterygoid occurs at maximal intercuspation. Therefore stretching and contraction of this muscle occurduring the same activity, clenching. If the superior lateral pterygoid is the source of pain, clenching will increase it. Superiorlateral pterygoid pain can be differentiated from elevator painby having the patient open wide. This will stretch the elevatormuscles but not the superior lateral pterygoid. If opening elicitsno pain, then the pain of clenching is from the superior lateralpterygoid. If the pain increases during opening, then both thesuperior lateral pterygoid and the elevators may be involved. Itis often diffcult to differentiate pain in the former from pain inthe latter unless the patient can isolate the location of the painful muscle.
For example, if an inflammatory disorder exists and the patient opens wide, pain is increased as a result of movement and function of the inflamed structures. If the mandible is protruded against resistance, pain is also increased, since movement and interarticular pressure arecausing force to be applied to the inflamed structures. If the teeth are clenched, pain is again increased with the increased interarticular pressure and force to the inflamed structures. If, however, the patient clenches unilaterally on a separator, the interarticularpressure is decreased on the ipsilateral side and pain in that joint is decreased
Maximal comfortable opening: Open mouth slowly until pain is first felt
Maximum opening: open mouth maximally even pain is present
END FEEL: characteristics of the restriction that limits the full range of joint movement.The end feel can be evaluated by placing the fngers between the patient’s upper and lower teeth and applying gentle but steady force in an attempt to passively increase the interincisal distance
If the end feel is “soft,” increased opening can be achieved, but it must be done slowly. A soft end feel suggests muscle-induced restriction.
If no increase in opening can be achieved, the end feel is said to be “hard.” Hard end feels are more likely associated with intracapsularsources (e.g., a disc dislocation
Deviation: usually due to a disc derangement in one or both joints and is a result of the condylar movement necessary to get past the disc during translation. Once the condyle has overcome this interference, the straight midline path is resumed
Deflection: It is due to restricted movement in one joint. The source of the restriction varies and must be investigated
If uncertainty exists regarding the proper position of the fngers, the patient is asked to open and close a few times. The fngertips should feel the lateral poles of the condyles passing downward and forward across the articular eminences. Once the position of the fngers over the joints has been verifed, the patient is asked to relax and medial force is applied to the joint areas
To evaluate the TMJ effectively, one must have a sound understanding of the anatomy in the region. When the fngers are placed properly over the lateral poles of the condyles and the patient is asked to clench, very little to no movement is felt. However, if the fngers are misplaced only 1 cm anterior to the lateral pole and the patient is asked to clench, the deep portion of the masseter can be felt contracting. This very slight difference in positioning of the fngers may influence the examiner’s interpretation of the origin of the pain.
It is not wise to examine the joint for sounds by placing thefngers in the patient’s ears. It has been demonstrated that thistechnique can actually produce joint sounds that are not presentduring normal function of the joint.45 It is thought that this technique forces the ear canal cartilage against the posterior aspectof the joint, so that this tissue may produce sounds or this forcedisplaces the disc, which produces additional sounds
To view the condyle best, it is often necessary for the patientto open maximally so that the structures of the articular fossaewill not be superimposed on the condyle. If the patient has onlylimited mandibular opening, superimposition is likely. With thistechnique the condyles are the only structures that are visualized well. The articular fossae are often partially if not totallyobscured
The patient is placed in a head positioner and the x-rays are directed inferiorly across the skull (above the midface) to the contralateral TMJ and recorded. Usually several projections of each joint are taken so that the function can be evaluated. For example, one projection is obtained with the teeth together in maximum intercuspation and another with the mouth maximally opened. Interpretation of the transcranial view begins with an understanding of the angle from which the projection was made
Standard radiographs may reveal that the morphology of a condyle has changed, but they are not helpful in determining ifWhen this information is important for treatmethe process is active (osteoarthritis) or dormant (osteoarthrosis).nt, a bone scancan be helpful.
“fan” the needle tip slightly. This is done by withdrawing the needle halfway, changing the needle’s direction slightly, and reentering the frm band to the same depth (Figure 10-2, D). The needle tip should not be completely removed from the tissue. This manipulation of the needle tip should be repeated several times, especially if the patient has not confrmed that the needle tip has reached the area of exquisite tenderness. At each site the syringe is aspirated and a small amount of anesthetic is deposited.
Fig: Joint can be entered by locating lateral pole of condyle. This can be assisted by asking thepatient to open and close the mouth. Once the pole is located, the patient is asked to open slightly and the examiner palpates directly above to locate thezygomatic arch. The tissue is cleaned and the tip of the needle is placed just below the zygomatic arch and slightly behind the posterosuperior aspect ofthe condyle
In patients with an intracapsular restriction (i.e., disc dislocation without reduction), a contralateral eccentric movement will be limited but an ipsilateral movement will be normal. However, with muscle disorders, the elevators (temporalis, masseter, medial pterygoid) are responsible for the limited mouth opening and, because eccentric movements do not generally lengthen these muscles, a normal range of eccentric movement exists
Myospasm can be separated from intracapsular disorders by observing protrusive and lateral eccentric movements.
If the problem is intracapsular, the mandible will often deflect to the side of the involved joint during protrusion and be restricted during a contralateral movement (normal movement to the ipsilateral side)
If the problem is extracapsular (i.e., stemming from muscle), there will be no deflection during the protrusive movement and no restrictions in lateral movement