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ANATOMY OF EYELID
Dr. Dhwanit Khetwani
MS ophthalmology
Final year.
Gross Anatomy
Mobile Tissue curtain placed in front of eyeball
FUNCTION
Act as shutter- protects
the eye from injuries
and excessive light.
Perform an important
function of spreading
the tear film on the
cornea and conjunctiva.
Help in Drainage of
tears by Lacrimal Pump
Action,
Eyelid has two parts
• Orbital
• Tarsal
• Divided by Horizontal Palpebral
Furrow/sulcus
• Upper lid covers about 1/6th of
the cornea (2mm)
• Lower Lid just touches the
limbus
• Palpebral Fissure -Eliptical space
between upper and lower lid
• Verical extent – 10-11mm
• Horizontal extent – 28-30mm
• Lids meet at Medial and lateral
canthi
• Lateral canthi 2mm higher than
medial canthi.(Mongoloid Slant)
10-11mm
28-30mm
LID MARGIN- 2mm broad, divided
into two parts by punctum
Medial- LACRIMAL PORTION-
Rounded Devoid
of EyelashesLateral- CILIARY PORTION
Anterior Border -
Rounded
Posterior Border –
Sharp
INTERMARGINAL
STRIP- Between 2
borders Grey line – marks
junction of skin and
conjunctiva
Divides intermarginal strip into –
Anterior strip bearing 2-3 layers of
Eyelashes
Posterior strip-Meibomian
glands arranged in a row.
Splitting of Eyelids in
surgeries done at the grey
line.
STRUCTURE OF EYELID
SKIN:
Elastic, fine
texture, thinnest
in the body
Subcutaneous
tissue:
Very Loose,
contains no fat.
Readily
distended by
blood or oedema
Layer of striated muscle:
Orbicularis muscle-Forms an oval sheet
around eyelids
• Orbital, Palpebral and Lacrimal parts.
• Closes the Eyelids, supplied by
Zygomatic branch of the Facial Nerve.
In paralysis of Facial Nerve there occurs
Lagophthalmos which may be
complicated by exposure Keratitis.
Levator palpebrae superioris
muscle (LPS)
It arises from the apex
of the orbit and is
inserted by three parts
on the
skin of lid,
anterior surface of
the tarsal plate
conjunctiva of
superior fornix.
It raises the upper lid. It is supplied
by a branch of oculomotor nerve. Submuscular areolar
tissue.
• It is a layer of loose connective
tissue.
• The nerves and vessels lie in this
layer.
• Therefore, to anaesthetise lids,
injection is given in this plane.
Fibrous layer. It is the framework of the lids
and consists of two parts:
Tarsal plate. dense
connective tissue, one for
each lid,
• shape and firmness to the lids.
• The upper and lower tarsal plates
meet at medial and lateral canthi;
attached to the orbital margins
through medial and lateral
palpebral ligaments.
• In the substance of the tarsal plates
lie Meibomian glands in parallel
rows.
Septum orbitale (palpebral
fascia)
• thin membrane of connective
tissue attached centrally to the
tarsal plates and peripherally to
periosteum of the orbital margin.
• It is perforated by nerves, vessels
and levator palpebrae superioris
(LPS) muscle, which enter the lids
from the orbit.
Layer of non-striated muscle fibres.
consists of the palpebral muscle of Muller
which lies deep to the septum orbitale in
both the lids.
• In the upper lid it arises from the
fibres of LPS muscle and
• lower lid from prolongation of the
inferior rectus muscle; and is
inserted on the peripheral margins
of the tarsal plate.
• It is supplied by sympathetic
fibres.
• Meibomian glands (tarsal glands)
• present in the stroma of tarsal plate arranged
vertically.
• 30–40 in the upper lid ,20–30 in the lower lid.
• modified sebaceous glands. Ducts open at the
lid margin.
• secretion constitutes the oily layer of tear film.
• Glands of Moll.
• modified sweat glands situated near
the hair follicle.
• open into the hair follicles or the
ducts of Zeiss glands.
• Secrete sebum.
• Accessory lacrimal glands of
Wolfring.
• These are present near
the upper border of the
tarsal plate
• Glands of Zeis.
• These are also
sebaceous glands
which open into the
follicles of eyelashes.
Conjunctiva. The part which lines the lids is called palpebral
conjunctiva. It consists of three parts: marginal, tarsal and orbital.
Blood Supply
Arteries of Lids: 2 arcades –
• Marginal arterial arcades- Medial and lateral palpebral
arteries arise from it
• Location- sub muscular plane in front of the Tarsal Plate
• Upper lid- 2mm away from the Lid margin
• Lower lid- 4mm away from the lid margin
• Superior Arterial Arcade
• Location- in the Upper lid, near the upper border of the tarsal
plate.
Veins- Arranged in 2 plexuses
• Post-Tarsal – Draining into Ophthalmic vein,
• Pre-tarsal – Draining into Subcutaneous Veins.
Lymphatics- Arranged into Pretarsal, post tarsal
• Median half of lids : drain into Pre-auricular Lymph Nodes
• lateral Half : Drain into Submandibular Lymph nodes.
Nerve Supply
Motor Nerves-
• Facial- supplies Orbicularis Oculi
• Oculomotor – Levator palpebrae
supirioris
• Sympathetic Fibres – Muller’s Muscle
Sensory Supply-
• Branches of Trigeminal Nerve
• Upper lid- Lacrimal, Supraorbital,
Supratrochlear
• Lower Lid- Infraorbital, Infra trochlear.
PTOSIS
• Abnormal drooping of eyelids.
• The Upper eyelid covers more than 2 mm of cornea
Congenital Ptosis
Blepharophymosis
Syndrome
Marcus Gunn jaw-
winking syndrome
Acquired
Neurogenic
Third nerve palsy
Horner syndrome
Myogenic
Myasthenia gravis
Myotonic
dystrophy
Ocular myopathies
Aponeurotic Mechanical
Clinical Evaluation
• History -
• age of onset,
• family history,
• history of trauma,
• eye surgery
• variability in degree of
the ptosis.
Pseudoptosis- Rule out
• Ipsilateral conditions
• microphthalmos,
• phthisis bulbi,
• enophthalmos,
• prosthesis,
• brow ptosis,
• Dermatochalasis
• hypotropia.
• Contralateral conditions :
• eyelid retraction,
• high myopia
• proptosis
Examination
• Unilateral/Bilateral
• Function of orbicularis oculi muscle.
• Eyelid crease .
• Jaw-winking phenomenon is
present or not.
• Associated weakness of any
extraocular muscle.
• Bell’s phenomenon (up and
outrolling of the eyeball during
forceful closure) is present or
absent.
PTOSIS VITAL SIGNS
• PALPEBRAL FISSURE HEIGHT
• MARGIN-REFLEX DISTANCE-1
• MARGIN REFLEX DISTANCE-2
• LEVATOR FUNCTION
• SKIN CREASE HEIGHT
Margin Reflex Distance
• Distance between upper
lid margin and light
reflex (MRD1, 4-
4.5mm)
• Mild ptosis MRD
1(2-2.5mm)
• Moderate ptosis
(MRD1 1-1.5mm)
• Severe ptosis (MRD1
<1mm) MRD 2-Normal value is 5- 5.5mm
Levator Function- Upper Lid Excursion
• Reflects levator function
• Normal (15 mm or more)
• Good (12 mm or more)
• Fair (5-11 mm)
• Poor (4 mm or less)
Important to apply
pressure on the
Eyebrow to block the
action of frontalis
muscle
Special Investigations.
• Phenylephrine test and cocaine test is carried out in patients
suspected of Horner’s syndrome.
• Neurological investigations may be required to find out the cause in
patient with neurogenic ptosis.
TYPES OF PTOSIS
CONGENITAL PTOSIS
ETIOLOGY : Associated with congenital
weakness of Levator Palpabrae Superioris.
Features:
• Drooping of one/both eyelids since birth.(mild/moderate or
severe)
• Lid Crease- Diminished/Absent.
• Lid Lag on Downgaze( i.e. Ptotic Lid is higher than the
Normal),due to tethering effect of LPS, in contrast to
acquired where ptotic lid is lower in downgaze also.
• LPS function may be poor, fair or good depending upon
degree of weakness.
Associations
Blepharophymosis Syndrome
• Congenital Ptosis with Poor levator function
• Blepharophymisis
• Telecanthus(lateral displacement of medial
canthus)
• Epicanthus Inversus (lower lid fold larger than
upper)
Marcus Gunn Jaw Winking Ptosis
• Retraction of Ptotic lid with Jaw Movements.
• With stimulation of ipsilateral Pterygoid muscle
• Caused by abberent innervation
ACQUIRED PTOSIS
PTOSIS
MIOSIS
ANHYDROSIS
Rare entity, occurs due to interruption in
the sympathetic innervation to the eye.
Pre-Ganglionic
Post-Ganglionic
Central
•Cocaine Test
•Hydroamphetamine test
Conjunctivomullerectomy
OCULOMOTOR NERVE PALSY
MYSTHENIA GRAVIS
• Ptosis and diplopia
• Levator function-decreased
• Eyelid twitches-falls on prolonged gaze
• CAUSE-AUTOIMMUNE,ANTIBODIES TO
ach RECEPTORS
• Treatment-Medical,
Thymectomy(selected Patients)
Before injection Positive result
Edrophonium/
anticholinesterases prevents
breakdown of neurotransmitter
Acetylcholine
Chronic Progressive
External
Ophthalmoplegia
• Progressive Bilateral
mitochondrial
myopathy affecting
Extra ocular muscles.
• Gradually increasing
ptosis with lost
ocular motility
• Associated with
pigmentary
retinopathy and
heart block
• Treatment- Frontalis
Sling
Oculopharyngeal
Dystrophy
• Autosomal dominant
• Bilateral progressive
ptosis with facial
weakness.
Myotonic Dystrophy
• Characterised by
failure to relax
muscles after
sustained
contraction
• Associated with
Christmas Tree
cataract, frontal
balding, testicular
atrophy
MYOGENIC PTOSIS
APONEUROTIC PTOSIS
• Develops due to defects of Levator
aponeurosis.
• Involution Ptosis(senile)
• Aponeurotic Weakness(Blepharochalasis)
• Traumatic Dehiscence/Disinsertion.
• Levator Function- Good.
• Rx Levator Resection
Mechanical Ptosis:
• It results from impaired mobility
of upper lid.
• Causes include dermatochalasis,
large tumours such as
neurofibromas, heavy scar tissue,
severe oedema and anterior orbital
leisions.
TREATMENT
MILD PTOSIS + GOOD LF
+ Phenylephrine Test - Phenylephrine Test
Fasanella-Servat operation. Levator Advancement
with Resection
POOR LF -<5mm – FRONTALIS SLING
LF> 5MM – LEVATOR RESECTION
LF> 6mm – LEVATOR Advancement/
Fasanella Servat
Congenital Ptosis
Tarso-conjunctivo-Mullerectomy (Fasanella-
Servat operation).
• mild ptosis (1.5–2 mm) and good levator function.
• upper everted,and the upper tarsal border along
with its attached Muller’s muscle and conjunctiva
are resected.
• Almost always needs
surgical correction.
• SEVERE PTOSIS surgery
performed - earliest to
prevent stimulus
deprivation amblyopia
• MILD AND MODERATE
PTOSIS, surgery delayed
until the age of 3-4
years, when accurate
measurements are
possible.
Levator Resection
■Moderate ptosis.
•Depending on the level of LPS function the amount of LPS to be resected is as below:
•Good function : 16–17 mm (minimal)
•Fair function : 18–22 mm (moderate)
•Poor function : 23–24 mm (maximum)
■Severe ptosis.
•Fair levator function: 23–24
mm (maximum LPS
resection).
Conjunctival approach (Blaskowics’ operation):Skin approach (Everbusch’s operation):
Frontalis sling operation (Brow suspension).
• severe ptosis, no levator function.
• In this operation, lid is anchored to the frontalis muscle via a
sling. Fascia lata (best material) or some non-absorbable
material (e.g., supramide suture, silicon rod) may be used as
sling.
Treatment of acquired ptosis
• Treat the underlying cause wherever possible.
• Conservative treatment should be carried out and surgery deferred
at least for 6 months in neurogenic ptosis.
• Surgical procedures (when required) for acquired ptosis are
essentially the same as described for congenital ptosis. However, the
amount of levator resection required is always less than the
congenital ptosis of the same degree. Further, in most cases the
simple Fasanella-Servat procedure is adequate.
BLEPHARITIS
Bacterial Blepharitis Seborrhoeic/squamous
Posterior Blepharitis/Meibomitis
STYE
HORDEOLUM INTERNUM
Chalazion

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Anatomy Of Eyelid And Ptosis

  • 1. ANATOMY OF EYELID Dr. Dhwanit Khetwani MS ophthalmology Final year.
  • 2. Gross Anatomy Mobile Tissue curtain placed in front of eyeball FUNCTION Act as shutter- protects the eye from injuries and excessive light. Perform an important function of spreading the tear film on the cornea and conjunctiva. Help in Drainage of tears by Lacrimal Pump Action,
  • 3. Eyelid has two parts • Orbital • Tarsal • Divided by Horizontal Palpebral Furrow/sulcus • Upper lid covers about 1/6th of the cornea (2mm) • Lower Lid just touches the limbus • Palpebral Fissure -Eliptical space between upper and lower lid • Verical extent – 10-11mm • Horizontal extent – 28-30mm • Lids meet at Medial and lateral canthi • Lateral canthi 2mm higher than medial canthi.(Mongoloid Slant) 10-11mm 28-30mm
  • 4. LID MARGIN- 2mm broad, divided into two parts by punctum Medial- LACRIMAL PORTION- Rounded Devoid of EyelashesLateral- CILIARY PORTION Anterior Border - Rounded Posterior Border – Sharp INTERMARGINAL STRIP- Between 2 borders Grey line – marks junction of skin and conjunctiva Divides intermarginal strip into – Anterior strip bearing 2-3 layers of Eyelashes Posterior strip-Meibomian glands arranged in a row. Splitting of Eyelids in surgeries done at the grey line.
  • 5. STRUCTURE OF EYELID SKIN: Elastic, fine texture, thinnest in the body Subcutaneous tissue: Very Loose, contains no fat. Readily distended by blood or oedema Layer of striated muscle: Orbicularis muscle-Forms an oval sheet around eyelids • Orbital, Palpebral and Lacrimal parts. • Closes the Eyelids, supplied by Zygomatic branch of the Facial Nerve. In paralysis of Facial Nerve there occurs Lagophthalmos which may be complicated by exposure Keratitis.
  • 6. Levator palpebrae superioris muscle (LPS) It arises from the apex of the orbit and is inserted by three parts on the skin of lid, anterior surface of the tarsal plate conjunctiva of superior fornix. It raises the upper lid. It is supplied by a branch of oculomotor nerve. Submuscular areolar tissue. • It is a layer of loose connective tissue. • The nerves and vessels lie in this layer. • Therefore, to anaesthetise lids, injection is given in this plane.
  • 7. Fibrous layer. It is the framework of the lids and consists of two parts: Tarsal plate. dense connective tissue, one for each lid, • shape and firmness to the lids. • The upper and lower tarsal plates meet at medial and lateral canthi; attached to the orbital margins through medial and lateral palpebral ligaments. • In the substance of the tarsal plates lie Meibomian glands in parallel rows. Septum orbitale (palpebral fascia) • thin membrane of connective tissue attached centrally to the tarsal plates and peripherally to periosteum of the orbital margin. • It is perforated by nerves, vessels and levator palpebrae superioris (LPS) muscle, which enter the lids from the orbit.
  • 8. Layer of non-striated muscle fibres. consists of the palpebral muscle of Muller which lies deep to the septum orbitale in both the lids. • In the upper lid it arises from the fibres of LPS muscle and • lower lid from prolongation of the inferior rectus muscle; and is inserted on the peripheral margins of the tarsal plate. • It is supplied by sympathetic fibres.
  • 9. • Meibomian glands (tarsal glands) • present in the stroma of tarsal plate arranged vertically. • 30–40 in the upper lid ,20–30 in the lower lid. • modified sebaceous glands. Ducts open at the lid margin. • secretion constitutes the oily layer of tear film. • Glands of Moll. • modified sweat glands situated near the hair follicle. • open into the hair follicles or the ducts of Zeiss glands. • Secrete sebum. • Accessory lacrimal glands of Wolfring. • These are present near the upper border of the tarsal plate • Glands of Zeis. • These are also sebaceous glands which open into the follicles of eyelashes. Conjunctiva. The part which lines the lids is called palpebral conjunctiva. It consists of three parts: marginal, tarsal and orbital.
  • 10. Blood Supply Arteries of Lids: 2 arcades – • Marginal arterial arcades- Medial and lateral palpebral arteries arise from it • Location- sub muscular plane in front of the Tarsal Plate • Upper lid- 2mm away from the Lid margin • Lower lid- 4mm away from the lid margin • Superior Arterial Arcade • Location- in the Upper lid, near the upper border of the tarsal plate. Veins- Arranged in 2 plexuses • Post-Tarsal – Draining into Ophthalmic vein, • Pre-tarsal – Draining into Subcutaneous Veins. Lymphatics- Arranged into Pretarsal, post tarsal • Median half of lids : drain into Pre-auricular Lymph Nodes • lateral Half : Drain into Submandibular Lymph nodes.
  • 11. Nerve Supply Motor Nerves- • Facial- supplies Orbicularis Oculi • Oculomotor – Levator palpebrae supirioris • Sympathetic Fibres – Muller’s Muscle Sensory Supply- • Branches of Trigeminal Nerve • Upper lid- Lacrimal, Supraorbital, Supratrochlear • Lower Lid- Infraorbital, Infra trochlear.
  • 12. PTOSIS • Abnormal drooping of eyelids. • The Upper eyelid covers more than 2 mm of cornea Congenital Ptosis Blepharophymosis Syndrome Marcus Gunn jaw- winking syndrome Acquired Neurogenic Third nerve palsy Horner syndrome Myogenic Myasthenia gravis Myotonic dystrophy Ocular myopathies Aponeurotic Mechanical
  • 13. Clinical Evaluation • History - • age of onset, • family history, • history of trauma, • eye surgery • variability in degree of the ptosis. Pseudoptosis- Rule out • Ipsilateral conditions • microphthalmos, • phthisis bulbi, • enophthalmos, • prosthesis, • brow ptosis, • Dermatochalasis • hypotropia. • Contralateral conditions : • eyelid retraction, • high myopia • proptosis Examination
  • 14. • Unilateral/Bilateral • Function of orbicularis oculi muscle. • Eyelid crease . • Jaw-winking phenomenon is present or not. • Associated weakness of any extraocular muscle. • Bell’s phenomenon (up and outrolling of the eyeball during forceful closure) is present or absent.
  • 15. PTOSIS VITAL SIGNS • PALPEBRAL FISSURE HEIGHT • MARGIN-REFLEX DISTANCE-1 • MARGIN REFLEX DISTANCE-2 • LEVATOR FUNCTION • SKIN CREASE HEIGHT
  • 16. Margin Reflex Distance • Distance between upper lid margin and light reflex (MRD1, 4- 4.5mm) • Mild ptosis MRD 1(2-2.5mm) • Moderate ptosis (MRD1 1-1.5mm) • Severe ptosis (MRD1 <1mm) MRD 2-Normal value is 5- 5.5mm
  • 17. Levator Function- Upper Lid Excursion • Reflects levator function • Normal (15 mm or more) • Good (12 mm or more) • Fair (5-11 mm) • Poor (4 mm or less) Important to apply pressure on the Eyebrow to block the action of frontalis muscle
  • 18. Special Investigations. • Phenylephrine test and cocaine test is carried out in patients suspected of Horner’s syndrome. • Neurological investigations may be required to find out the cause in patient with neurogenic ptosis.
  • 19. TYPES OF PTOSIS CONGENITAL PTOSIS ETIOLOGY : Associated with congenital weakness of Levator Palpabrae Superioris. Features: • Drooping of one/both eyelids since birth.(mild/moderate or severe) • Lid Crease- Diminished/Absent. • Lid Lag on Downgaze( i.e. Ptotic Lid is higher than the Normal),due to tethering effect of LPS, in contrast to acquired where ptotic lid is lower in downgaze also. • LPS function may be poor, fair or good depending upon degree of weakness.
  • 20. Associations Blepharophymosis Syndrome • Congenital Ptosis with Poor levator function • Blepharophymisis • Telecanthus(lateral displacement of medial canthus) • Epicanthus Inversus (lower lid fold larger than upper) Marcus Gunn Jaw Winking Ptosis • Retraction of Ptotic lid with Jaw Movements. • With stimulation of ipsilateral Pterygoid muscle • Caused by abberent innervation
  • 21. ACQUIRED PTOSIS PTOSIS MIOSIS ANHYDROSIS Rare entity, occurs due to interruption in the sympathetic innervation to the eye. Pre-Ganglionic Post-Ganglionic Central •Cocaine Test •Hydroamphetamine test Conjunctivomullerectomy
  • 23. MYSTHENIA GRAVIS • Ptosis and diplopia • Levator function-decreased • Eyelid twitches-falls on prolonged gaze • CAUSE-AUTOIMMUNE,ANTIBODIES TO ach RECEPTORS • Treatment-Medical, Thymectomy(selected Patients)
  • 24. Before injection Positive result Edrophonium/ anticholinesterases prevents breakdown of neurotransmitter Acetylcholine
  • 25. Chronic Progressive External Ophthalmoplegia • Progressive Bilateral mitochondrial myopathy affecting Extra ocular muscles. • Gradually increasing ptosis with lost ocular motility • Associated with pigmentary retinopathy and heart block • Treatment- Frontalis Sling Oculopharyngeal Dystrophy • Autosomal dominant • Bilateral progressive ptosis with facial weakness. Myotonic Dystrophy • Characterised by failure to relax muscles after sustained contraction • Associated with Christmas Tree cataract, frontal balding, testicular atrophy MYOGENIC PTOSIS
  • 26.
  • 27. APONEUROTIC PTOSIS • Develops due to defects of Levator aponeurosis. • Involution Ptosis(senile) • Aponeurotic Weakness(Blepharochalasis) • Traumatic Dehiscence/Disinsertion. • Levator Function- Good. • Rx Levator Resection
  • 28. Mechanical Ptosis: • It results from impaired mobility of upper lid. • Causes include dermatochalasis, large tumours such as neurofibromas, heavy scar tissue, severe oedema and anterior orbital leisions.
  • 30. MILD PTOSIS + GOOD LF + Phenylephrine Test - Phenylephrine Test Fasanella-Servat operation. Levator Advancement with Resection POOR LF -<5mm – FRONTALIS SLING LF> 5MM – LEVATOR RESECTION LF> 6mm – LEVATOR Advancement/ Fasanella Servat
  • 31. Congenital Ptosis Tarso-conjunctivo-Mullerectomy (Fasanella- Servat operation). • mild ptosis (1.5–2 mm) and good levator function. • upper everted,and the upper tarsal border along with its attached Muller’s muscle and conjunctiva are resected. • Almost always needs surgical correction. • SEVERE PTOSIS surgery performed - earliest to prevent stimulus deprivation amblyopia • MILD AND MODERATE PTOSIS, surgery delayed until the age of 3-4 years, when accurate measurements are possible.
  • 32. Levator Resection ■Moderate ptosis. •Depending on the level of LPS function the amount of LPS to be resected is as below: •Good function : 16–17 mm (minimal) •Fair function : 18–22 mm (moderate) •Poor function : 23–24 mm (maximum) ■Severe ptosis. •Fair levator function: 23–24 mm (maximum LPS resection). Conjunctival approach (Blaskowics’ operation):Skin approach (Everbusch’s operation):
  • 33. Frontalis sling operation (Brow suspension). • severe ptosis, no levator function. • In this operation, lid is anchored to the frontalis muscle via a sling. Fascia lata (best material) or some non-absorbable material (e.g., supramide suture, silicon rod) may be used as sling.
  • 34. Treatment of acquired ptosis • Treat the underlying cause wherever possible. • Conservative treatment should be carried out and surgery deferred at least for 6 months in neurogenic ptosis. • Surgical procedures (when required) for acquired ptosis are essentially the same as described for congenital ptosis. However, the amount of levator resection required is always less than the congenital ptosis of the same degree. Further, in most cases the simple Fasanella-Servat procedure is adequate.

Editor's Notes

  1. The upper lid crease or sup palpabral sulcus is due to the lecator palpabrae muuscle which entes the eye lids, making it tarsal part distinct. Inf palpabral sulcus is due to fibrous bands ariisng from inferior rectus muscle which insert into the lower lid. Naso jugal and malar sulcus marks juntion of denser tissue of cheeks.
  2. 2-3 layers of eye lashes , 100-150 rows upper eye lid,50-70 rows lower eyelid. lash direction- forward upward bakward upper, forward downwards and backwards lower. Anterior strip also consistd of glands of ziess asnd moll opens into the piliary canal. Lash Follicle surrounded by veins and nerves hence have exquisite tactile sensitivity.
  3. Skin thin, variable no.of melanocytes, which may increase pigment production in response to chronic oedema/ inflammation.
  4. Flat ribbon like muscle, (fleshy)passes through the orbital septum , and at the upper border of tarsus, devides into 3 parts,(tendinous part) SUBMUSCULAR AREOLAR TISSUE- UPPER lid the levator p s divides spaces into preseptal and pre tarsal space.
  5. These glands open into hair follicle and not into skin
  6. MRD 1 + MRD 2 IS PALPEBRAL FISSURE HEIGHT. NO SOMETIMES THE PF CAN BE NORMAL AROUND 9, WITH MRD 1 AROUND 1-2 AND MRD 2 8 MM, SO THIS IS A CASE OF PTOSIS AND LOWER LID RETRACTION.
  7. Blepharophymosis – narrow horizontal palpebral fissure. Normal 25-30. usually 20-22 in blepharophymosis. Medial and lateral canthoplasty for blepharophymosis followed by frontalis sling Obliteration of levator function and frontalis sling is procedure of choice for marcus gun jaw winking.
  8. Sympathetic fibres supply Mullers muscle, dilator pupilae and inferior tarsus muscle