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CONGENITAL LESIONS OF
 LARYNX AND STRIDOR
     DEPT OF OTORHINOLARYNGOLOGY
                JJM M C
              DAVANAGERE
LARYNGOMALACIA
• Most common congenital abnormality of larynx
• Condition manifests at birth or soon after and
  usually disappears by two years of age
• Characterized by excessive flaccidity of
  Supraglottic larynx which is sucked in during
  inspiration producing stridor and cyanosis
• Stridor subsides on placing child in prone position
  and increases with crying
• Direct laryngoscopy elongated and curled up
  epiglottis (omega shaped), floppy AE folds and
  prominent arytenoids
• Treatment is conservative
• In severe stridor tracheostomy
CONGENITAL VOCAL FOLD
     PARALYSIS

• Results from birth trauma when
  RLN is stretched during breach or
  forceps delivery
• Can also results from anomalies of
  CNS
CONGENITAL SUBGLOTTIC
      STENOSIS
• Due to abnormal thickening of cricoid
  cartilage or fibrous tissue seen below
  the vocal cords
• Child may remain asymptomatic till
  URTI causes dyspnoea and stridor
• Diagnosis subglottic diameter less
  than 4mm in full term neonate (normal
  4.5-5.5mm) or 3mm in premature
  neonate (normal 3.5mm)
• Most cases improve as larynx grows but
  some may require surgery
LARYNGEAL WEB
• Due to incomplete recanalization of
  larynx
• Seen between the vocal cords with
  concave posterior margin
• Presents as airway obstruction, weak
  cry or aphonia dating from birth
• Treatment depends on thickness of web
• Thin web cut with knife or CO2 laser
• Thick web excision via laryngofissure
  and placement of silicone keel and
  subsequent dilatation
SUBGLOTTIC HAEMANGIOMA
• Though congenital patient is asymptomatic till 3-6 months
  of age
• About 50% have associated cutaneous haemangioma
• May present with stridor which increases on agitation and
  crying due to venous filling
• Direct laryngoscopy shows reddish blue mass below
  vocal cords
• Biopsy is sometimes, not always associated with
  hemorrhage
• Treatment depends on individual case
 Tracheostomy and observation
 Steroid therapy
 CO2 laser excision
SUBGLOTTIC
HAEMANGIOMA
LARYNGO-OESOPHAGEAL
        CLEFT
• Due to failure of fusion of cricoid
  lamina
• Presents with repeated aspiration
  and pneumonitis
• Coughing, choking and cyanosis
  are present at the time of feeding
• Surgically treated
LARYNGEAL CYST
• Arises in the AE folds and appears as
  bluish fluid filled smooth swelling in
  Supraglottic larynx
• Respiratory obstruction tracheostomy
• Needle aspiration or incision and
  drainage provides emergency airway
• Treatment is deroofing the cyst or
  excision with CO2 laser
STRIDOR
• Defined as noisy respiration
  produced by turbulent airflow
  through narrowed air passage
• Classified as Inspiratory, expiratory
  and biphasic
STRIDOR
• Inspiratory stridor produced in
  obstructive lesion of Supraglottic or
  pharynx
• Expiratory stridor lesions of
  thoracic trachea, primary and
  secondary bronchi
• Biphasic lesions of glottis,
  subglottis and cervical trachea
STRIDOR
• CONGENITAL             •    AQUIRED
 LARYNGOMALACIA
                         1.   FEBRILE
                             EPIGLOTITTIS
 LARYNGEAL WEB
                             LARYNGO-TRACHEITIS
                             DIPHTHERIA
 SUBGLOTTIC STENOSIS
                             INFECTIOUS
                              MONONUCLEOSUS
 HAEMANGIOMA                RETROPHARYNGEAL
                              ABSCESS
 VOCAL CORD PARALYSIS       QUINSY

 TONGUE AND JAW         2.   AFEBRILE
  ABNORMALITIES              PAPILLOMATOSIS
                             FOREIGN BODY
                             LARYNGEAL OEDEMA
                             ADENO TONSILLAR
                              HYPERTROPHY
STRIDOR-CAUSES
• Nose: choanal atresia in newborn
• Tongue: macroglossia, haemangioma,
  lymphangioma, lingual thyroid
• Mandible: micrognathia, Pierre-Robin syndrome
• Pharynx: congenital dermoid, retropharyngeal
  abscess, tumors, adenotonsillar hypertrophy
• Larynx:
 Congenital: web, laryngomalacia, cyst, subglottic
  stenosis
 Inflammatory: epiglottitis, laryngotracheitis, diphtheria
 Neoplastic: haemangioma, juvenile papilloma,
  carcinoma in adults
 Traumatic: injuries to larynx, foreign bodies,
  prolonged intubation
 Neurogenic: laryngeal paralysis
 Miscellaneous: tetanus, tetany, laryngismus stridulus
STRIDOR-CAUSES
• Trachea and bronchi:

   Congenital: atresia, stenosis, malacia
   Inflammatory: tracheobronchitis
   Neoplastic: tumors
   Traumatic: foreign body and stenosis

• Lesions outside respiratory tract

 Congenital: vascular rings, oesophageal atresia, tracheo-
  oesophageal fistula, cystic hygroma
 Inflammatory: retroeosophageal abscess
 Traumatic: foreign body oesophagus
 Tumors: masses in the neck
STRIDOR-MANAGEMENT
• History: onset, progression, duration,
  relation to feeding, cyanotic spell,
  aspiration or ingestion of foreign body,
  laryngeal trauma
• Physical examination: signs of
  respiratory distress, stridor with phase
  of respiration, associated features like
  fever, wheeze, snoring, hoarseness,
  muffled voice
STRIDOR-MANAGEMENT
• Investigations
 X-ray chest and soft tissue neck AP and
  lateral view
 Fluoroscopy
 CT of neck and chest
 Oesophagogram
 Angiography
 Xeroradiography
 Direct laryngoscopy
 Pan endoscopy
STRIDOR-MANAGEMENT


• Treatment: treat the exact cause
  depending on the diagnosis
Congenital lesions of larynx

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Congenital lesions of larynx

  • 1. CONGENITAL LESIONS OF LARYNX AND STRIDOR DEPT OF OTORHINOLARYNGOLOGY JJM M C DAVANAGERE
  • 2. LARYNGOMALACIA • Most common congenital abnormality of larynx • Condition manifests at birth or soon after and usually disappears by two years of age • Characterized by excessive flaccidity of Supraglottic larynx which is sucked in during inspiration producing stridor and cyanosis • Stridor subsides on placing child in prone position and increases with crying • Direct laryngoscopy elongated and curled up epiglottis (omega shaped), floppy AE folds and prominent arytenoids • Treatment is conservative • In severe stridor tracheostomy
  • 3.
  • 4. CONGENITAL VOCAL FOLD PARALYSIS • Results from birth trauma when RLN is stretched during breach or forceps delivery • Can also results from anomalies of CNS
  • 5. CONGENITAL SUBGLOTTIC STENOSIS • Due to abnormal thickening of cricoid cartilage or fibrous tissue seen below the vocal cords • Child may remain asymptomatic till URTI causes dyspnoea and stridor • Diagnosis subglottic diameter less than 4mm in full term neonate (normal 4.5-5.5mm) or 3mm in premature neonate (normal 3.5mm) • Most cases improve as larynx grows but some may require surgery
  • 6.
  • 7. LARYNGEAL WEB • Due to incomplete recanalization of larynx • Seen between the vocal cords with concave posterior margin • Presents as airway obstruction, weak cry or aphonia dating from birth • Treatment depends on thickness of web • Thin web cut with knife or CO2 laser • Thick web excision via laryngofissure and placement of silicone keel and subsequent dilatation
  • 8.
  • 9. SUBGLOTTIC HAEMANGIOMA • Though congenital patient is asymptomatic till 3-6 months of age • About 50% have associated cutaneous haemangioma • May present with stridor which increases on agitation and crying due to venous filling • Direct laryngoscopy shows reddish blue mass below vocal cords • Biopsy is sometimes, not always associated with hemorrhage • Treatment depends on individual case  Tracheostomy and observation  Steroid therapy  CO2 laser excision
  • 11. LARYNGO-OESOPHAGEAL CLEFT • Due to failure of fusion of cricoid lamina • Presents with repeated aspiration and pneumonitis • Coughing, choking and cyanosis are present at the time of feeding • Surgically treated
  • 12. LARYNGEAL CYST • Arises in the AE folds and appears as bluish fluid filled smooth swelling in Supraglottic larynx • Respiratory obstruction tracheostomy • Needle aspiration or incision and drainage provides emergency airway • Treatment is deroofing the cyst or excision with CO2 laser
  • 13.
  • 14. STRIDOR • Defined as noisy respiration produced by turbulent airflow through narrowed air passage • Classified as Inspiratory, expiratory and biphasic
  • 15. STRIDOR • Inspiratory stridor produced in obstructive lesion of Supraglottic or pharynx • Expiratory stridor lesions of thoracic trachea, primary and secondary bronchi • Biphasic lesions of glottis, subglottis and cervical trachea
  • 16. STRIDOR • CONGENITAL • AQUIRED  LARYNGOMALACIA 1. FEBRILE  EPIGLOTITTIS  LARYNGEAL WEB  LARYNGO-TRACHEITIS  DIPHTHERIA  SUBGLOTTIC STENOSIS  INFECTIOUS MONONUCLEOSUS  HAEMANGIOMA  RETROPHARYNGEAL ABSCESS  VOCAL CORD PARALYSIS  QUINSY  TONGUE AND JAW 2. AFEBRILE ABNORMALITIES  PAPILLOMATOSIS  FOREIGN BODY  LARYNGEAL OEDEMA  ADENO TONSILLAR HYPERTROPHY
  • 17. STRIDOR-CAUSES • Nose: choanal atresia in newborn • Tongue: macroglossia, haemangioma, lymphangioma, lingual thyroid • Mandible: micrognathia, Pierre-Robin syndrome • Pharynx: congenital dermoid, retropharyngeal abscess, tumors, adenotonsillar hypertrophy • Larynx:  Congenital: web, laryngomalacia, cyst, subglottic stenosis  Inflammatory: epiglottitis, laryngotracheitis, diphtheria  Neoplastic: haemangioma, juvenile papilloma, carcinoma in adults  Traumatic: injuries to larynx, foreign bodies, prolonged intubation  Neurogenic: laryngeal paralysis  Miscellaneous: tetanus, tetany, laryngismus stridulus
  • 18. STRIDOR-CAUSES • Trachea and bronchi:  Congenital: atresia, stenosis, malacia  Inflammatory: tracheobronchitis  Neoplastic: tumors  Traumatic: foreign body and stenosis • Lesions outside respiratory tract  Congenital: vascular rings, oesophageal atresia, tracheo- oesophageal fistula, cystic hygroma  Inflammatory: retroeosophageal abscess  Traumatic: foreign body oesophagus  Tumors: masses in the neck
  • 19. STRIDOR-MANAGEMENT • History: onset, progression, duration, relation to feeding, cyanotic spell, aspiration or ingestion of foreign body, laryngeal trauma • Physical examination: signs of respiratory distress, stridor with phase of respiration, associated features like fever, wheeze, snoring, hoarseness, muffled voice
  • 20. STRIDOR-MANAGEMENT • Investigations  X-ray chest and soft tissue neck AP and lateral view  Fluoroscopy  CT of neck and chest  Oesophagogram  Angiography  Xeroradiography  Direct laryngoscopy  Pan endoscopy
  • 21. STRIDOR-MANAGEMENT • Treatment: treat the exact cause depending on the diagnosis