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Hemorrhage & Shock
Its Management In
Oral Surgery
Instructor – Dr.Jesus George 1
Hemorrhage
ī° Bleeding
ī° Escape of blood from a blood vessel due to
damage or injury in vasculature
2
Types
ī° A-depending upon the nature of the
vessel
ī° Arterial hemorrhage
īŽ Bright red color
īŽ Pulsation of the artery can be seen
ī° Venous hemorrhage
īŽ Dark red color,
īŽ Non-pulsating with steady flow(constant
flow)
īŽ Difficult to control because vein gets
retracted
3
Cont.
ī° Capillary hemorrhage
īŽ Red colour
īŽ Slowly oozes out
īŽ It becomes significant if there is
bleeding tendencies
ī° Bony bleeding
īŽ From nutrient vessels in bone
īŽ Usually follows removal of lower 3rd
molar
4
Cont.
ī° B. Depending upon timing of
hemorrhage
ī° Primary hemorrhage -occurs at the time
of surgery
ī° Reactionary or intermediate
hemorrhage -occurs after 24 hrs of
surgery(causes-hypertension in
postoperative period, sneezing,coughing)
ī° Secondary hemorrhage -occurs after
48-72 hrs after surgery (infection)
5
Cont.
ī° C-depending upon the duration of
hemorrhage
ī° Acute hemorrhage -occurs suddenly
(esophageal variceal bleeding due to
portal hypertension)
ī° Chronic hemorrhage-(hemorrhoids or
piles or chronic duodenal ulcer
6
Cont.
ī° D-depending upon the nature of
bleeding
ī° External hemorrhage - epistaxis
ī° Internal hemorrhage - splenic rupture
7
Laboratory tests for screening
ī° Bleeding time (aspirin therapy)
ī° Platelet count (liver disease, c/c leukemia)
ī° Prothrombin time (heparin therapy, liver
disease)
8
Hemostasis
ī° Control or arrest of bleeding
9
Local hemostatic measures
ī° Mechanical methods
ī° Thermal methods
ī° Chemical methods
10
Mechanical methods
ī° Pressure
ī° Use of hemostats
ī° Sutures & ligation
11
Thermal methods
ī° Cautery
ī° Electrosurgery
ī° Cryosurgery
12
Chemical methods
ī° Astringents (ferric sulfate in ethyl cellulose)
ī° Styptics (epsilon amino caproic acid local &
systemic)
ī° Bone wax (bees wax(7)+olive
oil(2)+phenol(1),helps in platelet aggregation)
ī° Gelfoam (compressed sponge made up of porcine
skin & have physical action)
ī° Adrenaline
ī° Surgicel (knitted fabric, interaction with platelets)
ī° Fibrin glue (thrombin+fibrinogen+factor8)
13
Prevention of hemorrhage during &
after extraction
ī° Primary hemorrhage – tender handling
of soft tissues & bone.
ī° Reactionary hemorrhage – semi supine
or sitting posture, diazepam 5 mg at
bedtime or antihypertensive drugs after
consultation with the physician
ī° Secondary hemorrhage - antibiotics
14
Control of hemorrhage from major
arteries
ī° Greater palatine artery
ī° Pressure pack
ī° A round bolus of gauze is made of
adequate size , so that it does not cause
gagging
ī° It is kept in place by tie over sutures for
24 to 48 hours
ī° Pressure pack can be safely removed
after 48 hours
15
Cont.
ī° Lingual artery
ī° Local clamping of the artery & application
of electrocautery usually controls bleeding
ī° Superficial temporal artery
ī° Electrocautery
16
Hereditary coagulopathies
ī° Hemophilia -a
ī° Hemophilia -b
17
Hemophilia -a
ī° Caused by deficiency of factor 8
ī° Sex-linked recessive inherited disorder
ī° Seen in males
ī° Hemophilia-a is graded as mild ,
moderate ,& severe
ī° Mild - factor 8 level ranges from 6 to 30
percent
ī° Moderate - factor 8 level ranges from 1
to 5 percent
18
Cont.
ī° Severe - factor 8 level ranges from nil to
1 percent
ī° Clinical features - prolonged bleeding
tendencies even after a mild trauma
ī° Patients are higher risk for bleeding after
oral surgical procedures
19
Hemophilia b
ī° It is caused by deficiency of factor 9
ī° It is congenital disorder& transmitted as
sex--linked recessive trait
ī° Clinical manifestations of this disease
are similar to hemophilia a
20
Shock
ī° Shock is a pathophysiologic condition
clinically recognized as a state of
inadequate perfusion due to
inadequate blood flow,there is
inadequate delivery of nutrients to the
tissues & inadequate removal of
cellular waste products from tissue cells,
results in disruption of vital organ
functions.
21
Clinical features
ī° Mild - <20% blood loss
īŽ Cold, calmy,moist skin
īŽ ↓ in BP
īŽ Rapid pulse
īŽ Collapsed neck veins
īŽ Concentrated urine
22
Cont.
ī° Moderate - >20-40%
īŽ Feeling of thirsty
īŽ Hypotension
īŽ Rapid pulse
īŽ Oliguria or anuria
ī° Severe - >40 %
īŽ Agitated & confused patient
īŽ Hypotension
īŽ Rapid pulse
īŽ Rapid respiration
23
Classification
ī° Hypovolemic shock
ī° Cardiogenic shock
ī° Septic shock
ī° Neurogenic shock
ī° Anaphylactic shock
24
Hypovolemic shock
ī° Decrease in the circulating or effective
intravascular volume
ī° Most common type of shock in
maxillofacial trauma
ī° Hypovolemic shock is classified into
ī° 1.Haemorrhagic shock
ī° 2.Non- hemorrhagic shock
25
Cont.
ī° Hemorrhagic shock is due to loss of
blood from the body causing decreased
venous return & thus decreasing the
cardiac output
26
Cont.
ī° Non-hemorrhagic shock-there is
massive fluid shift from intravascular
compartment to extra vascular
compartment causing decreased blood
volume &thus decreasing tissue perfusion.
īŽ can result from burns, crush injuries,
pancreatitis, peritonitis & pleural effusion,
water loss due to severe diarrhea, vomiting,
hyperglycemia & nephritis
27
Cont.
ī° Treatment
ī° Control bleeding
ī° Legs raised & body supine is preferred
posture as this increases venous return &
cardiac index
ī° Fluid replacement; normal saline or ringer
lactate
28
Cardiogenic shock
ī° Inadequate cardiac output , impaired
oxygen delivery & reduced tissue
perfusion, caused by loss of effective
contractile function of myocardium
ī° Treatment
ī° Dopamine is the vasopressor of choice. It
is diluted in normal saline or 5%dextrose
drip
29
Septic shock
ī° Produced by microorganisms or toxins
ī° May be produced by bacteria ,virus &
fungi
ī° Inflammatory response causing
vasodilatation & hypovolemia
ī° Clinically: fever, tachycardia&
respiratory alkalosis
30
Cont.
ī° Treatment
ī° Early & effective volume replacement
ī° Restoration of tissue perfusion
ī° Control of infection with antibiotic therapy
ī° I.V betamethasone or dexamethasone
31
Neurogenic shock
ī° Vasovagal syncope or emotional fainting
ī° Blockage of sympathetic nervous system
→dialatation of blood vessels →hypovolemia
→shock
ī° Predisposing factors – fear, anxiety, painPredisposing factors – fear, anxiety, pain
- fatigue- fatigue
- fasting- fasting
- hot & humid weather- hot & humid weather
32
Cont.
ī° Signs & symptoms(Clinical feature):Signs & symptoms(Clinical feature):
īŽ Pale, cold & moist skinPale, cold & moist skin
īŽ Slow pulse initially followed by rapid pulseSlow pulse initially followed by rapid pulse
īŽ DizzinessDizziness
īŽ WeaknessWeakness
īŽ Nausea & sweatingNausea & sweating
īŽ Loss of consciousnessLoss of consciousness
īŽ Dilated pupilDilated pupil
33
Cont.
ī° Prevention
īŽ Reassurance
īŽ Achieve confidence of the patient
īŽ Anxiolytic medication prior to surgery
(diazepam 5 mg 1 tablet night before the
surgery & one 1/2hr before surgery)
īŽ patient should not come with empty stomach
34
Cont.
ī° ManagementManagement
īŽ Monitoring pulseMonitoring pulse
īŽ Lowering head to improve cerebellar circulationLowering head to improve cerebellar circulation
īŽ Loosening of tight cloths at neckLoosening of tight cloths at neck
īŽ Smelling of salt of aromatic ammoniaSmelling of salt of aromatic ammonia
īŽ Flashing the face with cold waterFlashing the face with cold water
īŽ Glucose drinksGlucose drinks
īŽ Inj.dexamethasoneInj.dexamethasone
īŽ Reassurance of patientReassurance of patient
35
Anaphylactic shock
ī° Caused by anaphylaxis of penicillin&
streptomycin
ī° Treatment
ī° Oxygen
ī° Airway
ī° Injection adrenaline
ī° Cardiopulmonary resuscitation
36

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14 hemorrhage & shock

  • 1. Hemorrhage & Shock Its Management In Oral Surgery Instructor – Dr.Jesus George 1
  • 2. Hemorrhage ī° Bleeding ī° Escape of blood from a blood vessel due to damage or injury in vasculature 2
  • 3. Types ī° A-depending upon the nature of the vessel ī° Arterial hemorrhage īŽ Bright red color īŽ Pulsation of the artery can be seen ī° Venous hemorrhage īŽ Dark red color, īŽ Non-pulsating with steady flow(constant flow) īŽ Difficult to control because vein gets retracted 3
  • 4. Cont. ī° Capillary hemorrhage īŽ Red colour īŽ Slowly oozes out īŽ It becomes significant if there is bleeding tendencies ī° Bony bleeding īŽ From nutrient vessels in bone īŽ Usually follows removal of lower 3rd molar 4
  • 5. Cont. ī° B. Depending upon timing of hemorrhage ī° Primary hemorrhage -occurs at the time of surgery ī° Reactionary or intermediate hemorrhage -occurs after 24 hrs of surgery(causes-hypertension in postoperative period, sneezing,coughing) ī° Secondary hemorrhage -occurs after 48-72 hrs after surgery (infection) 5
  • 6. Cont. ī° C-depending upon the duration of hemorrhage ī° Acute hemorrhage -occurs suddenly (esophageal variceal bleeding due to portal hypertension) ī° Chronic hemorrhage-(hemorrhoids or piles or chronic duodenal ulcer 6
  • 7. Cont. ī° D-depending upon the nature of bleeding ī° External hemorrhage - epistaxis ī° Internal hemorrhage - splenic rupture 7
  • 8. Laboratory tests for screening ī° Bleeding time (aspirin therapy) ī° Platelet count (liver disease, c/c leukemia) ī° Prothrombin time (heparin therapy, liver disease) 8
  • 9. Hemostasis ī° Control or arrest of bleeding 9
  • 10. Local hemostatic measures ī° Mechanical methods ī° Thermal methods ī° Chemical methods 10
  • 11. Mechanical methods ī° Pressure ī° Use of hemostats ī° Sutures & ligation 11
  • 12. Thermal methods ī° Cautery ī° Electrosurgery ī° Cryosurgery 12
  • 13. Chemical methods ī° Astringents (ferric sulfate in ethyl cellulose) ī° Styptics (epsilon amino caproic acid local & systemic) ī° Bone wax (bees wax(7)+olive oil(2)+phenol(1),helps in platelet aggregation) ī° Gelfoam (compressed sponge made up of porcine skin & have physical action) ī° Adrenaline ī° Surgicel (knitted fabric, interaction with platelets) ī° Fibrin glue (thrombin+fibrinogen+factor8) 13
  • 14. Prevention of hemorrhage during & after extraction ī° Primary hemorrhage – tender handling of soft tissues & bone. ī° Reactionary hemorrhage – semi supine or sitting posture, diazepam 5 mg at bedtime or antihypertensive drugs after consultation with the physician ī° Secondary hemorrhage - antibiotics 14
  • 15. Control of hemorrhage from major arteries ī° Greater palatine artery ī° Pressure pack ī° A round bolus of gauze is made of adequate size , so that it does not cause gagging ī° It is kept in place by tie over sutures for 24 to 48 hours ī° Pressure pack can be safely removed after 48 hours 15
  • 16. Cont. ī° Lingual artery ī° Local clamping of the artery & application of electrocautery usually controls bleeding ī° Superficial temporal artery ī° Electrocautery 16
  • 17. Hereditary coagulopathies ī° Hemophilia -a ī° Hemophilia -b 17
  • 18. Hemophilia -a ī° Caused by deficiency of factor 8 ī° Sex-linked recessive inherited disorder ī° Seen in males ī° Hemophilia-a is graded as mild , moderate ,& severe ī° Mild - factor 8 level ranges from 6 to 30 percent ī° Moderate - factor 8 level ranges from 1 to 5 percent 18
  • 19. Cont. ī° Severe - factor 8 level ranges from nil to 1 percent ī° Clinical features - prolonged bleeding tendencies even after a mild trauma ī° Patients are higher risk for bleeding after oral surgical procedures 19
  • 20. Hemophilia b ī° It is caused by deficiency of factor 9 ī° It is congenital disorder& transmitted as sex--linked recessive trait ī° Clinical manifestations of this disease are similar to hemophilia a 20
  • 21. Shock ī° Shock is a pathophysiologic condition clinically recognized as a state of inadequate perfusion due to inadequate blood flow,there is inadequate delivery of nutrients to the tissues & inadequate removal of cellular waste products from tissue cells, results in disruption of vital organ functions. 21
  • 22. Clinical features ī° Mild - <20% blood loss īŽ Cold, calmy,moist skin īŽ ↓ in BP īŽ Rapid pulse īŽ Collapsed neck veins īŽ Concentrated urine 22
  • 23. Cont. ī° Moderate - >20-40% īŽ Feeling of thirsty īŽ Hypotension īŽ Rapid pulse īŽ Oliguria or anuria ī° Severe - >40 % īŽ Agitated & confused patient īŽ Hypotension īŽ Rapid pulse īŽ Rapid respiration 23
  • 24. Classification ī° Hypovolemic shock ī° Cardiogenic shock ī° Septic shock ī° Neurogenic shock ī° Anaphylactic shock 24
  • 25. Hypovolemic shock ī° Decrease in the circulating or effective intravascular volume ī° Most common type of shock in maxillofacial trauma ī° Hypovolemic shock is classified into ī° 1.Haemorrhagic shock ī° 2.Non- hemorrhagic shock 25
  • 26. Cont. ī° Hemorrhagic shock is due to loss of blood from the body causing decreased venous return & thus decreasing the cardiac output 26
  • 27. Cont. ī° Non-hemorrhagic shock-there is massive fluid shift from intravascular compartment to extra vascular compartment causing decreased blood volume &thus decreasing tissue perfusion. īŽ can result from burns, crush injuries, pancreatitis, peritonitis & pleural effusion, water loss due to severe diarrhea, vomiting, hyperglycemia & nephritis 27
  • 28. Cont. ī° Treatment ī° Control bleeding ī° Legs raised & body supine is preferred posture as this increases venous return & cardiac index ī° Fluid replacement; normal saline or ringer lactate 28
  • 29. Cardiogenic shock ī° Inadequate cardiac output , impaired oxygen delivery & reduced tissue perfusion, caused by loss of effective contractile function of myocardium ī° Treatment ī° Dopamine is the vasopressor of choice. It is diluted in normal saline or 5%dextrose drip 29
  • 30. Septic shock ī° Produced by microorganisms or toxins ī° May be produced by bacteria ,virus & fungi ī° Inflammatory response causing vasodilatation & hypovolemia ī° Clinically: fever, tachycardia& respiratory alkalosis 30
  • 31. Cont. ī° Treatment ī° Early & effective volume replacement ī° Restoration of tissue perfusion ī° Control of infection with antibiotic therapy ī° I.V betamethasone or dexamethasone 31
  • 32. Neurogenic shock ī° Vasovagal syncope or emotional fainting ī° Blockage of sympathetic nervous system →dialatation of blood vessels →hypovolemia →shock ī° Predisposing factors – fear, anxiety, painPredisposing factors – fear, anxiety, pain - fatigue- fatigue - fasting- fasting - hot & humid weather- hot & humid weather 32
  • 33. Cont. ī° Signs & symptoms(Clinical feature):Signs & symptoms(Clinical feature): īŽ Pale, cold & moist skinPale, cold & moist skin īŽ Slow pulse initially followed by rapid pulseSlow pulse initially followed by rapid pulse īŽ DizzinessDizziness īŽ WeaknessWeakness īŽ Nausea & sweatingNausea & sweating īŽ Loss of consciousnessLoss of consciousness īŽ Dilated pupilDilated pupil 33
  • 34. Cont. ī° Prevention īŽ Reassurance īŽ Achieve confidence of the patient īŽ Anxiolytic medication prior to surgery (diazepam 5 mg 1 tablet night before the surgery & one 1/2hr before surgery) īŽ patient should not come with empty stomach 34
  • 35. Cont. ī° ManagementManagement īŽ Monitoring pulseMonitoring pulse īŽ Lowering head to improve cerebellar circulationLowering head to improve cerebellar circulation īŽ Loosening of tight cloths at neckLoosening of tight cloths at neck īŽ Smelling of salt of aromatic ammoniaSmelling of salt of aromatic ammonia īŽ Flashing the face with cold waterFlashing the face with cold water īŽ Glucose drinksGlucose drinks īŽ Inj.dexamethasoneInj.dexamethasone īŽ Reassurance of patientReassurance of patient 35
  • 36. Anaphylactic shock ī° Caused by anaphylaxis of penicillin& streptomycin ī° Treatment ī° Oxygen ī° Airway ī° Injection adrenaline ī° Cardiopulmonary resuscitation 36