This document outlines a psychoeducational group for patients with medically unexplained physical symptoms (MUPS). It includes an initial assessment, follow up letters, and an 8-week program outline. The program aims to educate patients on MUPS and teach coping strategies. Sessions cover topics like the impact of stress, illness beliefs, pain management, and putting learning into practice. The goal is for patients to better understand and manage their symptoms.
2. During this presentation we will
examine:
The outline of a psychoeducuative group
Different classifications of MUPS
An overview of theories of aetiology
What do Neuro-imaging studies have to offer?
An overall theory of MUPs
So what ? How might this be relevant to 10 minute
consultation?
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3. Initial letter to patient from GP
I am writing to let you know about a new course that we are
about to start that will be held here at the surgery. It is
called ‘Coping with Health Problems’. I know that you
have had a series of health problems and I thought that
this programme might be helpful to you
It will be run by … who has a special interest in working
with people with health problems in order to help them
cope with their illness.
The course consists of an initial assessment when a
comprehensive history will be taken along with few simple
and harmless tests. You will be told more about the
course and, if you both agree that it might be useful, you
will be invited to eight one-and-a-half-hour weekly group
meetings.
If you would like to attend simply contact….
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4. Assessment
Client’s attitude to the referral and the problem
Details of the illness
Present symptoms: Type of pain, Occurrence
What makes it worse and what makes it better
Thoughts accompanying pain
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5. Assessment
Coping Strategies
Consequences on life
Others response
History including previous treatments
General beliefs about the nature and meaning
of the symptoms
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6. Assessment
Biofeedback:EMG,GSR,Peripheral Temp
Medication Other treatments
Goals of treatment What would you like to
change?
Rationale for psychological treatment
(contribution of stress as a cause or effect.
Test out hypothesis of psychological element)
Description of course:
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7. Follow-up letter from group leader to
patient
Following our meeting on ….. concerning the effects of your
(symptom description), enclosed is the course outline that I
promised. The course will start on (date) at the … (directions) .
It will start at ... in … room. Each meeting will last for one and
a half hours.
As I explained when we met the course is a mixture of education
and self help. This means that in between each meeting an
exercise will be set which will help you to monitor your progress.
Before we meet I'd be grateful if you would have a think about what
you would like to gain from the course - try imagining yourself in
eight weeks time and think of what you would like to have
changed by then. You'll be able to discuss this in more detail at
our first meeting.
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8. Programme Outline
Week 1: General introduction , goal setting
and models of health
Introduction to the notion of coping
Hot cross bun
Goals on a flip chart
Introduction to self monitoring
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9. Programme Outline
Week 2: What happens when we are stressed
by health problems
Self monitoring review
Stress what is it?
Individual stress reactions to symptoms
Early experiences of illness
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10. Programme Outline
Week 3: Physical reactions to stress
Self monitoring review
Cave person exercise
Active progressive relaxation
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11. Programme Outline
Week 4: How do individual views of illness
effect health?
Review of self monitoring
How thinking and beliefs effect feelings
NATS and BATS
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12. Programme Outline
Week 5 : How does behaviour affect health: in
introduction to TA
Self monitoring review
How behaviour can maintain behaviour
How we interact with other people
TA overview – Ego states, transactions and
games
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13. Programme Outline
Week 6: Pain and Pain management
Review of self monitoring exercises
The relative nature of pain – the gateway
theory
Autogenic training
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14. Programme Outline
Week 7: Putting together the pieces
Open ended group for participants to bring own
agenda
So what?
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16. Exercise
Thinking of your childhood how was illness
managed in your family?
How did your parents respond to you/your
siblings when you were ill?
How did you respond to your parents/siblings?
How has this effected your current relationship
with illness?
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17. The Organic Genesis of Patient
Presentations (Kroenke and Manglesdorf 1993)
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20. Somatisation
A transient or persistent tendency to
experience and communicate psychological
distress in the form of somatic symptoms and
to seek medical help for them. It occupies a
continuum from a simple misinterpretation of a
subjective bodily sensation to an unwavering
belief that a physical disease is present
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21. Health Anxiety
Health anxiety OCD spectrum of ‘disorders’.
Obsessional preoccupation with the idea or the
thought of currently (or will be) experiencing a
physical illness.
Common health anxieties tend to centre on
conditions such as cancer, HIV, AIDs etc,
May fixate on any type of illness.
Also called illness phobia/ illness anxiety or
hypochondriasis.
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22. The Spectrum of MUPS
Duration: transient persistent
No. of Sx: one multiple
Insight: good none
Disability: none severe
23. Theories of Aetiology
Psychobiological
High levels of physiological arousal
Alexithymia
Cognitive/Behavioural
Somatisation is rewarded
Secondary gains reinforce symptoms
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24. Theories of Aetiology
Psychoanalytical
Real conflicts denied, suppressed or repressed
Anxiety displaced into physical symptoms
Sociocultural
Emotions expressed through physical
symptoms
Specific “culture bound” syndromes (Koro,
taijin kyofusho)
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25. • EP posits a theoretical framework to understand false
illness signaling
• An EP approach to somatisation asks whether false
illness signaling represents an innate psychological
mechanism triggered by situational exigencies
• Somatisation may represent a behavioural strategy
that bestows survival value
Theories of Aetiology
Evolutionary psychology (EP)
26. Theories of Aetiology
Early Trauma
High correlation with early trauma and MUPs
(Roelofs and Spinhoven 2006) (Salmon et al 2003)
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27. Theories of Aetiology
Attachment styles
Secure – reliable care giving as children, positive view
of self and comfortable depending on others
Dismissing – unresponsive caregiving, self reliant
others not to be relied on
Preoccupied – inconsistent caregiving, negative view of
self seen as unlovable and expecting others to view
negatively preoccupied vigilant
Fearful – needs not met when young negative view of
self and others approach and avoidant
High correlation with MUPS and preoccupied and
fearful AS hemfipsych.com
29. Theories of Aetiology
Dissociation
Higher levels of dissociative amnesia in
somatising patients (Brown et al 2005)
Linked to pseudo-seizures (Prueter et al 2002)
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30. Theories of Aetiology
Immune system
Activation of the immune system seems to
induce behaviour patterns that are similar to
the illness behaviour seen in depression and
somatisation (Rief and Barsky 2005).
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31. Neuro-imaging studies: Irritable
Bowel Syndrome
Anterior Cingulate Cortex (ACC) as having a
role in the regulation of pain in IBS (Ringel et al
1999)
Uprated in chronic and downrated in acute
(Peyron et al 2000)
Association with CSA and dissociation (Salmon
et al 2003)
32. Neuro-imaging studies: Chronic
Fatigue Syndrome
Significant positive relationships were found for
cerebellar, temporal, cingulate and frontal regions and a
significant negative relationship was found for the left
posterior parietal cortex in CFS patients v controls (Cook
et al. 2007).
Increased activation in the occipito-parietal cortex,
posterior cingulate gyrus and parahippocampal gyrus,
and decreased activation in dorsolateral and
dorsomedial prefrontal cortices (Caseras et al. 2008).
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33. Neuro-imaging studies:
Fybromyalgia
Low stimulus pressure associated with 13
regions of brain activated compared with only
one in controls. (Gracely et al. 2002).
Greater activation in contralateral insular cortex
in both non painful warm and pain stimulus
(Cook et al. 2004).
34. Neuro-imaging studies: Expectation
(Nocebo effect)
(Rief & Broadbent 2007)
Expectation of symptoms leads to the
activation of brain areas corresponding to
symptom perception
Distraction from symptoms reduces brain
activity in perception areas
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35. The perception-filter model of
somatisation
(modified from Rief and Barsky 2005 in Rief and Broadbent 2007)
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37. The role of emotion
Emotion associated with profound physiological
changes
Often unconscious as bypasses frontal cortex
Often clients have limited language for emotions
Affect avoidance (having feelings, expressing
feelings and confusion about feelings)
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38. Common Safety Behaviours
Checking pulse
Hypervigilence of ‘symptoms’
Reducing activity
Symptom browsing on internet
Seeking reassurance from GP
Palpating parts of the body
Reducing food intake
Slowing down/speeding up
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39. Potential interventions: Behavioural
Reducing “boom and bust” mode
Reducing symptom-focusing behaviours
Anxiety management skills
Re-education re somatising precipitators and
perpetuators and treatment programme
Graded exposure (using exposure hierachy)
Identifying and reducing safety behaviours
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40. Potential interventions:
physiological
Relaxation exercises (diaphragmatic breathing,
APR autogenics)
Graded exercise
Moving specific symptom focused parts of
body
Diet
Substance use
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41. Potential interventions: Emotional
Identifying feelings
Developing language for feeling
Reducing feeling avoidance
Having conversation with symptom
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42. Development of ‘illness behaviour’
Often related to attachment styles
When GP and patient are together – two
attachment styles and illness behaviours are
interacting
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43. Attunement enables
affect-regulation
Like the securely attached mother, the
empathic psychobiologically attuned clinician’s
regulation of the patient’s affective-arousal
states is critical to transforming the patient’s
insecure nonconscious internal working model
that encodes strategies of affect regulation
Schore 2007 12
44. • B.. Background -What is happening in your
life at the moment?
• A.. Affect- How do you feel about that?
• T.. Trouble -What is the most troubling part
of..
• H..Handling - How are you managing
to deal with that?
• E..Empathy - That must be difficult for you.
The BATHE technique
(Stuart and Leiberman 2002)
45. Dos and Don’ts:
DO
Talk about coping
Use one designated GP
Schedule frequent, brief, regular visits not
contingent on new complaints.
Allow "sick role;" focus on function rather than
symptoms.
Explore psychosocial issues.
Prescribe benign treatments and enjoyment
time. hemfipsych.com
46. Dos and Don’ts:
DON’T
Suggest "It's all in your head.“
Pursue invasive diagnostic tests, medications
or surgical interventions without good
indications.
Refer excessively to specialists.
Focus on the symptoms themselves
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47. MUPS Further Reading
Bass, C Ed (1990). Somatisation: physical symptoms and psychological illness.
Blackwell Oxford.
Donohugue, P. & Seigel ,M. (1997). Sick and Tired of Feeling Sick and Tired,
Living with invisible chronic illness and Sage
Mayou, R., Bass,C. & Sharpe, M. (1995). Treatment of functional somatic
symptoms. Oxford University Press. Oxford.
Gill, D. (2007). Hughes’ Outline of Modern Psychiatry. Wiley & Sons (see
Chapter 9 on Physical Symptoms and Psychiatric Disorders)
Sanders, D., (1996) ofor Psychosomatic Problems. London: Sage Publicationst
Woolfolk, R. & Allen, L. (2007).Treating Somatization. A Cognitive Behavioral
Approach Guildford Press
Johnson,S. (2008). Medically Unexplained Illness. Gender and Biopsychosocial
Implications. APA
Journals: Journal of Psychosomatic Research, Psychosomatics
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Editor's Notes
The anterior cingulate cortex can be divided anatomically based on cognitive (dorsal), and emotional (ventral) components.[4] The dorsal part of the ACC is connected with the prefrontal cortex and parietal cortex as well as the motor system and the frontal eye fields[5] making it a central station for processing top-down and bottom-up stimuli and assigning appropriate control to other areas in the brain. By contrast, the ventral part of the ACC is connected with amygdala, nucleus accumbens, hypothalamus, and anterior insula, and is involved in assessing the salience of emotion and motivational information. The ACC seems to be especially involved when effort is needed to carry out a task such as in early learning and problem-solving.[6] Many studies attribute functions such as error detection, anticipation of tasks, motivation, and modulation of emotional responses to the ACC.[4][5][7]
The parietal lobe integrates sensory information from different modalities, particularly determining spatial sense and navigation. For example, it comprises somatosensory cortex and the dorsal stream of the visual system. This enables regions of the parietal cortex to map objects perceived visually into body coordinate positions.
It is an integral part of the limbic system, which is involved with emotion formation and processing, learning, and memory, and is also important for executive function and respiratory control.
The parahippocampal gyrus (Syn. hippocampal gyrus)[1] is a grey matter cortical region of the brain that surrounds the hippocampus. This region plays an important role in memory encoding and retrievaL
DL-PFC serves as the highest cortical area responsible for motor planning, organization, and regulation. It plays an important role in the integration of sensory and mnemonic information and the regulation of intellectual function and action. It is also involved in working memory. However, DL-PFC is not exclusively responsible for the executive functions. All complex mental activity requires the additional cortical and subcortical circuits that DL-PFC is connected with
HANDLING>>>
The locus of control returns to them.