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Lymphedema Physical Therapy Treatment & Management
“I wouldn’t want to be a physical therapist – you have to touch people’s feet!”
Overview	 Theory  Anatomy & Physiology Pathology Principles of Management Treatment Manual Lymphatic Drainage Massage Compression Bandaging/Garments Exercise Patient Education
The Lymphatic System Anatomy & Physiology
The big picture Works with venous system for fluid return Removes plasma proteins  Filters antigens, bacteria, waste products Produces white blood cells
Initial Lymphatics – “Pickles” ,[object Object]
Lymph capillaries
Superficial
Overlapping endothelial cells
Anchoring fibers
Valves prevent fluid backflow,[object Object]
Afferent Collectors
Muscular cell walls with valves
Lymphangions – section between the valves
Lymph Nodes  ,[object Object]
Collector Sequence – cont.	 Efferent collectors Lymphatic Trunks Lymphatic Ducts (2)
Anatomical landmarks Lymphotomes  Distinct regions of the body for fluid drainage Watersheds Division areas between lymphotomes where direction of flow changes Anastomoses Collateral vessels offering alternate routes of drainage
Lymph Fluid Transparent, yellowish fluid 96% water – more dilute than plasma Also consists of proteins, lipids, minerals, hormones, cells, bacteria, cell waste, etc. Body produces 2.4L of lymph fluid daily, 25L of lymph fluid cycles through the heart
Pathology	 Lymphedema An excessive accumulation of protein-rich fluid in the tissues caused by a transport failure of the lymphatic system.
Pathophysiology Mechanical Insufficiency - lymphostatic Low-flow edema, low-volume insufficiency A breakdown in the transport capacity of the lymphatic system Dynamic Insufficiency - lymphodynamic High-flow edema, high-volume insufficiency A high load placed on the lymph system exceeds its capacity Combination – Safety Valve Insufficiency
Causes of Lymphedema Primary Secondary Congenital deficit in number or size of lymph nodes and/or pathways Milroy’s Disease – present at birth Meige’s Disease or Lymphedema praecox – appears at puberty Lymphedema tarda – adult onset Caused by removal or or damage to lymph nodes and/or pathways Cancer/cancer treatments Trauma Chronic Venous Insufficiency Paralysis Filariasis
Sequellae Chronic swelling Excessive tissue proteins Fibrotic changes Chronic inflammation Infections – cellulitis Skin changes – “peau d’ orange”
Signs of Lymphedema	 Puffiness, heaviness, fullness of limb Stiffness, decreased ROM Weakness, fatigue Skin tension – feeling of “bursting” Pain Numbness, paresthesias
Consequences Functional deficits Loss of mobility Difficulty wearing normal clothing Psychological issues
Stages of Lymphedema Stage 0  Latent, sub-clinical condition Swelling not evident despite impaired lymph transport Stage 1  Completely & spontaneously reversible Soft, pitting edema Little to no fibrosis Skin easily pinched & moved
Stages – cont. Stage 2 Spontaneously Irreversible Tissues usually fibrotic Pitting requires strong pressure, or no pitting Can usually be reversed with treatment Stage 3 - Elephantiasis Irreversible No pitting, significant fibrosis Huge size, papillomas, hardening of the skin
Other Types of Edema Acute Venous Lipedema
Principles of Management	 “If you can help me, I’ll add you to my Christmas card list!”
Goals While the immediate goal of lymphedema therapy is to relieve swelling, the ultimate goal is to enable the patient to self-manage and control this chronic condition.
Complete Decongestive Therapy Manual Lymphatic Drainage (MLD) Compression Bandaging Exercise Skin care – patient education Compression Garments
Adjunct Treatments Myofascial Release Kinesiotaping Aquatic therapy Wound Care Pneumatic pumps
Team Approach ,[object Object]
Other therapists
Certified garment fitter
Nutritionist
Psychologist,[object Object]
Treatment Techniques “You’re just making that up to make me look stupid!”
Patient Examination History Functional mobility Musculoskeletal Neurological Skin Integrity Edema measurement Cardiovascular
Diagnostic Testing Lymphoscinitigraphy Radiological isotope evaluation Lymphography Direct oil contrast – “blue-dye” Ultrasound Assess presence of filarial worm MRI CT
Red Flags! Contraindications Precautions Any bacterial infection Acute congestive heart failure Acute deep vein thrombosis Acute renal disease Acute pulmonary edema Malignancy Arterial disease Sensation deficits Low blood counts Areas of inflammation Wounds
Skin and Wound Care Prior to treatment, skin should be clean and moisturized Wounds must be addressed
Manual Lymphatic Drainage Goals of technique: Increase peristalsis of lymphangion Break down fibrotic tissues Increase lymph volume in lymph vessels Decrease congestion in interstitium
Basic Principles Treatment begins proximal, to “clear” proximal lymphotomes before moving to affected lymphotomes Massage is directed towards the cleared lymphotome Pressure is very light Strokes are rhythmic
Strokes	 “Scoops” – massaging lymph nodes Firmer pressure, circular motion “Clearing”  Begin furthest from the affected area Clears the way for fluid drainage Moves proximal to distal “Flowing” Begin closest to the affected area Always done after clearing Moves distal to proximal

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Lymphedema

  • 1. Lymphedema Physical Therapy Treatment & Management
  • 2. “I wouldn’t want to be a physical therapist – you have to touch people’s feet!”
  • 3. Overview Theory Anatomy & Physiology Pathology Principles of Management Treatment Manual Lymphatic Drainage Massage Compression Bandaging/Garments Exercise Patient Education
  • 4. The Lymphatic System Anatomy & Physiology
  • 5. The big picture Works with venous system for fluid return Removes plasma proteins Filters antigens, bacteria, waste products Produces white blood cells
  • 6.
  • 11.
  • 13. Muscular cell walls with valves
  • 14. Lymphangions – section between the valves
  • 15.
  • 16. Collector Sequence – cont. Efferent collectors Lymphatic Trunks Lymphatic Ducts (2)
  • 17.
  • 18. Anatomical landmarks Lymphotomes Distinct regions of the body for fluid drainage Watersheds Division areas between lymphotomes where direction of flow changes Anastomoses Collateral vessels offering alternate routes of drainage
  • 19. Lymph Fluid Transparent, yellowish fluid 96% water – more dilute than plasma Also consists of proteins, lipids, minerals, hormones, cells, bacteria, cell waste, etc. Body produces 2.4L of lymph fluid daily, 25L of lymph fluid cycles through the heart
  • 20. Pathology Lymphedema An excessive accumulation of protein-rich fluid in the tissues caused by a transport failure of the lymphatic system.
  • 21.
  • 22. Pathophysiology Mechanical Insufficiency - lymphostatic Low-flow edema, low-volume insufficiency A breakdown in the transport capacity of the lymphatic system Dynamic Insufficiency - lymphodynamic High-flow edema, high-volume insufficiency A high load placed on the lymph system exceeds its capacity Combination – Safety Valve Insufficiency
  • 23. Causes of Lymphedema Primary Secondary Congenital deficit in number or size of lymph nodes and/or pathways Milroy’s Disease – present at birth Meige’s Disease or Lymphedema praecox – appears at puberty Lymphedema tarda – adult onset Caused by removal or or damage to lymph nodes and/or pathways Cancer/cancer treatments Trauma Chronic Venous Insufficiency Paralysis Filariasis
  • 24. Sequellae Chronic swelling Excessive tissue proteins Fibrotic changes Chronic inflammation Infections – cellulitis Skin changes – “peau d’ orange”
  • 25. Signs of Lymphedema Puffiness, heaviness, fullness of limb Stiffness, decreased ROM Weakness, fatigue Skin tension – feeling of “bursting” Pain Numbness, paresthesias
  • 26. Consequences Functional deficits Loss of mobility Difficulty wearing normal clothing Psychological issues
  • 27. Stages of Lymphedema Stage 0 Latent, sub-clinical condition Swelling not evident despite impaired lymph transport Stage 1 Completely & spontaneously reversible Soft, pitting edema Little to no fibrosis Skin easily pinched & moved
  • 28. Stages – cont. Stage 2 Spontaneously Irreversible Tissues usually fibrotic Pitting requires strong pressure, or no pitting Can usually be reversed with treatment Stage 3 - Elephantiasis Irreversible No pitting, significant fibrosis Huge size, papillomas, hardening of the skin
  • 29. Other Types of Edema Acute Venous Lipedema
  • 30. Principles of Management “If you can help me, I’ll add you to my Christmas card list!”
  • 31. Goals While the immediate goal of lymphedema therapy is to relieve swelling, the ultimate goal is to enable the patient to self-manage and control this chronic condition.
  • 32. Complete Decongestive Therapy Manual Lymphatic Drainage (MLD) Compression Bandaging Exercise Skin care – patient education Compression Garments
  • 33. Adjunct Treatments Myofascial Release Kinesiotaping Aquatic therapy Wound Care Pneumatic pumps
  • 34.
  • 38.
  • 39.
  • 40. Treatment Techniques “You’re just making that up to make me look stupid!”
  • 41. Patient Examination History Functional mobility Musculoskeletal Neurological Skin Integrity Edema measurement Cardiovascular
  • 42. Diagnostic Testing Lymphoscinitigraphy Radiological isotope evaluation Lymphography Direct oil contrast – “blue-dye” Ultrasound Assess presence of filarial worm MRI CT
  • 43. Red Flags! Contraindications Precautions Any bacterial infection Acute congestive heart failure Acute deep vein thrombosis Acute renal disease Acute pulmonary edema Malignancy Arterial disease Sensation deficits Low blood counts Areas of inflammation Wounds
  • 44. Skin and Wound Care Prior to treatment, skin should be clean and moisturized Wounds must be addressed
  • 45. Manual Lymphatic Drainage Goals of technique: Increase peristalsis of lymphangion Break down fibrotic tissues Increase lymph volume in lymph vessels Decrease congestion in interstitium
  • 46. Basic Principles Treatment begins proximal, to “clear” proximal lymphotomes before moving to affected lymphotomes Massage is directed towards the cleared lymphotome Pressure is very light Strokes are rhythmic
  • 47. Strokes “Scoops” – massaging lymph nodes Firmer pressure, circular motion “Clearing” Begin furthest from the affected area Clears the way for fluid drainage Moves proximal to distal “Flowing” Begin closest to the affected area Always done after clearing Moves distal to proximal
  • 48.
  • 50. Medical Compression Bandaging Low stretch bandages Provides low stretch when no contraction Higher compression when muscles contract Prevent re-fill of lymphatics Work with muscle pumping Follow “Law of Laplace” – the smaller the radius the greater the pressure Help break up fibrotic areas
  • 51. Bandaging Guidelines Worn 23 hrs/ day (off only for bathing) Should re-wrap daily to prevent loosening Should have more compression (more layers) distally Bandages should be washed frequently
  • 53. Compression Garments Maintains reduction gained during therapy Transition to garments when reduction plateaus Garments for daytime wear, options for nighttime Insurance coverage varies greatly
  • 54. Therapeutic Exercise Exercises facilitate muscle pumping Should be done with compression Progress proximally to distally Very low resistance, few repetitions, rests as needed
  • 55. Patient Education Home exercise Self-massage Skin care Precautions Compression garments
  • 56. Conclusion Do all the good you can,By all the means you can,In all the ways you can,In all the places you can,At all the times you can,To all the people you can,As long as ever you can -John Wesley
  • 57. Thank You! Presented by Jennifer K. Root, PT, DPT, CLT Clinical Specialist MidMichigan Medical Center Rehabilitation Services

Editor's Notes

  1. 90% of fluid that is in the interstitial spaces is resorbed by venous capillaries, 10% absorbed by lymphaticsLymph system can take up to 10% overload
  2. Cell junctions open/close in response to changes in interstitial pressure.Pressure changes occur from movements of anchoring fibers, light compression, muscle contraction, respiration, pulsation of nearby arteries
  3. Collectors – 3 cells thick – inner layer muscularValves separating lymphangion are 6 to 20 mm apart.Lymph enters the lympangion, causing a stretch reflex of the smooth muscle layer, which then contracts to move the lymph into the next lymphagionRate is appx. 10x per minute, increases 10 to 30 x per minute w/exercise
  4. Efferent pathways can connect w/ venous system, other lymph vessels, or can continue along same lymph vessel
  5. Right Lymphatic DuctTerminates at junction of R. internal jugular & subclavian veinDrains right side of thorax, right arm, and right side of face & neckThoracic DuctBegins at cysternachyliTerminates at junction of L. internal jugular & subclavianDrains left side of thorax, left arm, left side of face & neck, entire abdomen and lower extremities
  6. Lymphotomes – 4 of trunk, multiple lymphotomes in each extremityWatersheds – no valves, can act as detours from congested areas
  7. Affects 140-150 million people worldwide.In our clinic lymphedema quickly became 25% of our total caseload.
  8. Primary – estimated 1 in 6000 individuals (your text, 1985), typically females, unilateral LE involvementOf primary – 75% are praecoxSecondary – Cancer/Rx is most common cause in developed countriesWorldwide – filariasis is most common. Caused by a parasitic infection carried by mosquitoes. Affects 120 million people in over 80 countries.
  9. Mention Stemmer’s Sign!Measurement technique – volume calculationsDiagnostic tests – lymphoscintigraphy (radiological isotope evaluation), lymphography (blue dye test),Ultrasound (for filriasis worm) MRI (lymph in