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In this podcast…
 Overview of tachycardic arrhythmias
 Look more in detail at the broad QRS
complex arrhythmias
 Causes
 Signs and symptoms
 What they look like on ECG
 Management
Overview
Tachycardiac
Arrhythmias
Broad QRS
Complex
Narrow QRS
Complex
ArrhythmiasArrhythmias
Broad QRS complex arrhythmias
Broad QRS
Complex
Ventricular
Ventricular
Tachycardia
Torsades de
pointes
Ventricular
Fibrillation
Supraventricular
tachycardia PLUS a
bundle branch block
Ventricular Tachycardia
Defined as three or more ventricular extrasystoles in
succession at a rate of >120bpm
 Causes
 Coronary heart disease- including acute MI
 Cardiomyopathies
 Signs and symptoms
 Can be pulseless- indicates minimal cardiac output
 Syncope
 Palpitations, chest pain
 Haemodynamic compromise
Ventricular Tachycardia
Distinguish VT from SVT with BBB--- very broad QRS (> 0.14 s)
Pulseless VT?
Immediate
defibrillation
Haemodynamically
compromised?
Urgent DC
Cardioversion
Stable?
Lignocaine or
amiodarone
Patient is…
2
Torsades de Pointes
Arises when ventricular repolarization (QT interval) is greatly
prolonged. Can degenerate into VF and cause death.
 Causes
 Congenital
 Acquired
 MI
 Drugs
 Bradycardia
 Electrolyte disturbances
 Signs and symptoms
 Palpitations, dizziness and syncope.
 Often terminates spontaneously
Remember
‘all the hypos’
Antiarrhythmic drugs (Sotalol, Amiodarone)
Erthromycin
Amitriptyline
Torsades de Pointes
QRS amplitude varies and the QRS complexes appear to twist around the baseline
 Management
 Correct electrolyte imbalances
 Stop causative drugs
 IV magnesium (even if magnesium levels are normal)
 Be aware that defibrillation may be needed if VF
occurs.
3
Ventricular Fibrillation
Produces rapid, ineffective, uncoordinated movement of the
ventricles, which therefore produce no pulse and no cardiac
output.
 Causes
 MI/ischaemia
 Structural heart disease
 Long QT syndrome (Torsades de Pointes)
 Signs and symptoms
 Patient will likely be unconscious and pulseless
Ventricular Fibrillation
Defibrillation and
resuscitation!
4
Supraventricular tachycardia
with bundle branch block
Causes, symptoms and management are similar to SVT
without BBB
i.e. Treatment with adenosine
Summary
 Broad QRS complex tachycardic arrhythmias.
 Most are ventricular in origin with the exception
of SVT with BBB.
 Ventricular tachycardia
 Assess patient; shockable if pulseless. May
need DC cardioversion or lidocaine or
amiodarone
 Torsades de Pointes
 Remove causes first, then magnesium is
treatment of choice
 Ventricular fibrillation
 shockable!
Thank you for listening!
References
1. Creative Commons Attribution-NonCommercial-ShareAlike 2.0 License. Author; www.soil-net.com (accessed
12th March 2011). Image altered by Charlotte Clifford
2. Creative commons Attribution-Share Alike 3.0 Unported License. Author; Glenlarson (accessed 12th March 2011)
3. Creative commons Attribution-Share Alike 3.0 Unported License. Author; displaced (accessed 12th March 2011)
4. Creative commons Attribution-Share Alike 3.0 Unported License. Author; Andersat (accessed 12th March
2011)Image altered by Charlotte Clifford

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Podcast on Tachycardic Arrhythmias

  • 1. 1
  • 2. In this podcast…  Overview of tachycardic arrhythmias  Look more in detail at the broad QRS complex arrhythmias  Causes  Signs and symptoms  What they look like on ECG  Management
  • 4. Broad QRS complex arrhythmias Broad QRS Complex Ventricular Ventricular Tachycardia Torsades de pointes Ventricular Fibrillation Supraventricular tachycardia PLUS a bundle branch block
  • 5. Ventricular Tachycardia Defined as three or more ventricular extrasystoles in succession at a rate of >120bpm  Causes  Coronary heart disease- including acute MI  Cardiomyopathies  Signs and symptoms  Can be pulseless- indicates minimal cardiac output  Syncope  Palpitations, chest pain  Haemodynamic compromise
  • 6. Ventricular Tachycardia Distinguish VT from SVT with BBB--- very broad QRS (> 0.14 s) Pulseless VT? Immediate defibrillation Haemodynamically compromised? Urgent DC Cardioversion Stable? Lignocaine or amiodarone Patient is… 2
  • 7. Torsades de Pointes Arises when ventricular repolarization (QT interval) is greatly prolonged. Can degenerate into VF and cause death.  Causes  Congenital  Acquired  MI  Drugs  Bradycardia  Electrolyte disturbances  Signs and symptoms  Palpitations, dizziness and syncope.  Often terminates spontaneously Remember ‘all the hypos’ Antiarrhythmic drugs (Sotalol, Amiodarone) Erthromycin Amitriptyline
  • 8. Torsades de Pointes QRS amplitude varies and the QRS complexes appear to twist around the baseline  Management  Correct electrolyte imbalances  Stop causative drugs  IV magnesium (even if magnesium levels are normal)  Be aware that defibrillation may be needed if VF occurs. 3
  • 9. Ventricular Fibrillation Produces rapid, ineffective, uncoordinated movement of the ventricles, which therefore produce no pulse and no cardiac output.  Causes  MI/ischaemia  Structural heart disease  Long QT syndrome (Torsades de Pointes)  Signs and symptoms  Patient will likely be unconscious and pulseless
  • 11. Supraventricular tachycardia with bundle branch block Causes, symptoms and management are similar to SVT without BBB i.e. Treatment with adenosine
  • 12. Summary  Broad QRS complex tachycardic arrhythmias.  Most are ventricular in origin with the exception of SVT with BBB.  Ventricular tachycardia  Assess patient; shockable if pulseless. May need DC cardioversion or lidocaine or amiodarone  Torsades de Pointes  Remove causes first, then magnesium is treatment of choice  Ventricular fibrillation  shockable!
  • 13. Thank you for listening! References 1. Creative Commons Attribution-NonCommercial-ShareAlike 2.0 License. Author; www.soil-net.com (accessed 12th March 2011). Image altered by Charlotte Clifford 2. Creative commons Attribution-Share Alike 3.0 Unported License. Author; Glenlarson (accessed 12th March 2011) 3. Creative commons Attribution-Share Alike 3.0 Unported License. Author; displaced (accessed 12th March 2011) 4. Creative commons Attribution-Share Alike 3.0 Unported License. Author; Andersat (accessed 12th March 2011)Image altered by Charlotte Clifford

Editor's Notes

  1. Haemodynamic compromise- low BP, breathlessness, signs of heart failure
  2. There are a few ways to distinguish VT from SVT with BBB and one of the most important ways is the very broad QRS complex of over 0.14s seen in VT If pulseless then this is a shockable rhythym and so you MUST defib!
  3. Congenital- may be trigger by stress, fear or physical excretion. Bradycardia- sinoatrial disease, atrioventricular (AV) block Electrolyte disturbances- Hypokalaemia, Hypomagnesaemia, Hypocalcaemia Symptoms- Nausea, pallor, cold sweats, shortness of breath and chest pain may occur Will reoccur unless the underlying cause is removed. Can cause sudden death!
  4. ECG shows a bradycardia followed by an episode of tachcardia with variable QRS amplitude- torsades de points Management- ABCD etc
  5. Structural heart disease- Aortic stenosis, Hypertrophic cardiomyopathy, Dilated cardiomyopathy, Congenital heart disease (p. 633)
  6. See the algorithm for ALS for drug administration.
  7. Please see the Part 2 podcast on narrow QRS complex tachycardic arrhythmias
  8. Arises when ventricular repolarization (QT interval) is greatly prolonged. Can degenerate into VF and cause death. Causes Congenital- Jervell-Lange-Nielsen and Romano-Ward Acquired MI Drugs- antiarrhythmic drugs (Sotalol, Amiodarone), Erythromycin, Amitriptyline Bradycardia Electrolyte disturbances- all the ‘hypos’ Signs and symptoms Palpitations, dizziness and syncope. Is not usually sustained and terminates spontaneously