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ANALYSIS
OF
ACUTE ABDOMINAL PAIN
PREPARED BY
DR. BOOQ YUOSEF & DR FIAZ FAZILI
SURGICAL DEPTT.OHUD HOSPITAL
PRESENTED BY
DR.MOUSTAFA HEGAZY
• depends in large measure on analysis of
pain , which requires an understanding of
the mechanisms of abdominal pain .
DIAGNOSIS OF ACUTE
ABDOMINAL PAIN
OBJECTIVITY OF THIS
LECTURE
IS
To examine the physiologic background of
abdominal pain
As
An aid to accurate interpretation of
Symptoms & Signs .
Anatomic background
 Parietal peritoneum
clothes the anterior & posterior
abdominal walls the under surface
of the diaphragm & the cavity of
the pelvis.( supplied segmentally
by the spinal nerves ) .
 Visceral peritoneum
is the continuation of the parietal
peritoneum, which leaves the
posterior wall of the abdominal
cavity to invest certain viscera
therein . ( has no nerve supply ).
11 2 3
4 5 6
7
8
9
T6-T9
T6-T9
T8-T12
T8
T10
L2
S4
foregut
midgut
hindgut
DEFINITION OF PAIN
It is an unpleasant sensation of varying
intensity .
Pain fibers are stimulated any time a tissue is being
damaged . However , it is not felt very long after the
damage has been accomplished.
1
2
3
4
5
Mechanical trauma to the tissue .
Excess heat or cold .
STIMULANTS
Chemical damage.
Radiation damage .
Inadequate blood flow.
Intra-thoracic organs
Abdominal Extra-abdominal
Abdominal wall
+
Intra-peritoneal organsRetro-peritoneal organsPelvic organs
Systemic dysfunction
Diabetes ,tabes dorsalis
porphyria
Functional abdominal
pain
Types
of abdominal pain
1
Visceral pain is primitive and therefore related to
embryonic devlepment therefore related to
embryologic development .
Somatic pain is entirely different from visceral pain
2
Visceral pain
1- Receptor
( Visceral peritoneum )
Visceral pain
2 - Stimulus
Pat. Experienced pain by
traction ,distention & spasm
The visceral peritoneum is
insensitive to touch & heat or
any condition that promotes
an inflammatory reaction
Visceral pain
3 - Mediation
Thalamus
Corpuscollosum
Hypothalamus
CerebralCortex
Pons
Medulla
Spinalcord
Cerebellum
Autonomic nervous System Interpreted at
the thalamic level of the brain
Visceral pain
4- Specificity
Vague , often dull , poorly described &
associated with nausea & vomiting
Visceral pain
5- Localization
Is poor & the pat. Placing the entire
hand over the involved region
Somatic pain
1- Receptor
Pain stimuli start in the parietal peritoneum ,
which is innervated by peripheral nerves
P/ peritoneum
Somatic pain
2- Stimulus
Pat. experienced pain by
TouchPressureHeatInflammation
Somatic pain
3- Mediation
Central nervous system
&
Interpreted at a specific cortical
location
Somatic pain
4- Specificity
Precisely described as
SharpKnifelikeCutting
Somatic pain
5- Localization
The pain is localized with
great accuracy by the patient ,
who can often point to the site
with one finger
Analysis of pain
need
DATA COLLECTION
1 2 3
History Physical exam. Lab.inv.
apply
your medical knowledge***
The history of pain betrays the diagnosis
History of pain
SiteMode of onsetNature of painSeverityRadiationDuration
Factors influencing
the clinical
manifestation
Site of pain & radiation
Kidney
Stomach &
duodenum
GB
Pancreas
Kidney
Small bowel
Caecum &
rp.structure
T. Colon
bladder
uterus &
adnexae
Sig.
colon
App. &
Caecum
Radiation of the pain
GB. Pain radiates
through to the back & to
the right , to reach the
tip of the shoulder blade
Radiation indicates source of the pain
& also the extent of the diseaseLower abdominal pain
rarely radiate
The structures in pelvis may
radiate to the lower back or
perineum
Small bowel pain does not
usually radiate
but
may move when somatic
as well as visceral nerves
become irritated
Stomach & duodenal pain
goes
strait through the back
Pancreatic pain
tends to go
through to the back but
to the left
Kidney pain
may radiate
down into the groin
Mode of onset
Sudden onset
[The patient can tell you exactly when the pain started ]
The pain that start suddenly has a mechanical basis
Some thing has been
RupturedTwistedOccluded
Cont’ Mode of onset
Gradual Onset
( The pat. Usually responds vaguely to questions
about time of onset )
Non mechanical or
chronic process
Nature of pain
Two Large Categories
(1)
Conditions associated with obstruction
of a muscular conducting tube
( Small bowel , Ureter , Biliary )
(2)
Conditions associated with
inflammation
( Mild & Localized Response or
Severe , Generalized Response )
Obstruction
Suddenprolonged
Distention of the viscus
( constant stretching pain )
Colic pain = visceral pain
Three Types
( 1 ) Biliary System = ( foregut )
Foregut pain is experienced in the epigastrium
1
(2) Small Intestine = ( midgut )
Pain is experienced in the periumbilical region
(3) Renal system = ( retroperitoneal )
Pain is felt in the flank & radiates to the groin
Important features of colic pain
I. Pat . Is often restless & agitated during exacerbations.
II. Pat. Does not experience a totally pain –free interlude.
III. Colic pain is an intermittent pain .
IV. Colic pain is an visceral pain .
( not influenced by changing relationships between the peritoneal layers )
V. Failing to demonstrate guarding , tenderness ? ????
Inflammation2
Intra-abdominal inflammation is peritonitis
Peritonitis causes somatic pain
Peritonitis
LocalizedGeneralized
Inflammation2
Intra-abdominal inflammation is peritonitis
Peritonitis causes somatic pain
Contamination
BY
ChemicalsBacteriaTraumaForeign body
Important features of somatic pain
I. Pat. Laying quite in bed . ( movement is limited )
II. Examination may demonstrate guarding , tenderness .
III. The pain is localized over the inflamed organ .
IV. Fever , tachycardia & tachypnea are systemic
manifestation for generalized inflammation .
Ischemic pain
Is a somatic pain
Occlusion of blood supply
cause
Tissue Hypoxia
With metabolic
changes
Necrosis
After 6-12 h
Severity of acute abdominal pain
Factors influencing clinical
manifestation
(1)
Extent of the pathologic process
The more severe the process , the
more impressive the manifestation .
Factors influencing clinical
manifestation
(2)
Time of Assessment
Depending on the time of assessment ,
the characteristics will reflect what is
present at that time – not previously &
not subsequently .
Factors influencing clinical
manifestation
(3)
Emotional factors
Objective criteria are more reliable than
subjective factors .When there is a discrepancy
between the severity of pain & objective
findings ,caution should be exercised .
Factors influencing clinical
manifestation
(4)
The Patient’s Intelligence
A clinical history is only as reliable as its source .
If the pat. is
Intellectually impairedSenileVery youngOr PsychoticVery ill
The information obtained must be interpreted
carefully . Objective & subjective findings
should be compatible .
Factors influencing clinical
manifestation
(5)
Level of consciousness
Some neurologic problems make the
interpretation of acute abdominal pain difficult .
UnconsciousnessParaplegiaSympathetic denervation
Bizarre manifestation of abd. Pain .
Factors influencing clinical
manifestation
(6)
Drugs
Many drugs influence both the character ,
perception & the course and effects of disease .
Corticosteroids
Suppress the inflammatory response
Sedatives
Influence pat. Recognition of problems
Analgesics
Decrease pain ( minimized or overlooked )
Medical causes
of abdominal
pain
■ Diabetic
ketoacidosis
■ Porphyria
■ Pain arising
from the heart
or lungs
Embryological division of the
gastrointestinal tract
• Foregut:
• Oesophagus
• Stomach
• Duodenum – first and second parts
• Pancreas
• Liver
• Gall bladder
• Midgut
• Duodenum –third and fourth
parts
• Jejunum
• Ileum
• Right colon
• Transverse colon
• Appendix
•Hindgut
•Left colon
•Sigmoid colon
•Rectum
• Origins and presentation of abdominal
pain
• Parietal Well localised but can radiate
forwards and backwards along the
dermatome
• Visceral Poorly localised; associated
with sweating and nausea
• Retroperitoneal Pain in the back
• The nature of the pain will change as
the cause evolves
Sources of pain that evolve over
hours or days
■ Acute Appendicitis
Cholecystitis
Salpingitis
Mesenteric adenitis
• ■ Infarction
• ■ Free blood in the peritoneum
Sources of pain that evolve over
hours or days
■ Acute Appendicitis
Cholecystitis
Salpingitis
Mesenteric adenitis
• ■ Infarction
• ■ Free blood in the
Causes of sudden onset
abdominal pain
■Perforation
Appendix
Stomach
Duodenum
Colon
Aggravating features of pain
Aggravating feature
Moving or coughing
Interpretation
Peritoneal inflammation
Aggravating feature
Patient rolls around with pain Colic
Interpretation
(suggests obstruction of viscus)
THANK YOU
I. Clinical Exercise
A male pat. ,aged 17, develops mild
periumbilical discomfort not influenced by
activity . Several hours later , his discomfort
increases but is now situated in the right lower
quadrant .Movement becomes painful .
Examination reveals localized pain ,
tenderness , and rebound tenderness localized
to a small area in the right lower quadrant .
Analysis of case ( 1 )
The initial pain is visceral & midgut in origin
but is replaced by somatic pain .
The most common problem in
this region & this age is app .
Why visceral pain ????
The inflammation starts intraluminally , & the app. Becomes
distended. Stretching is a stimulus for visceral pain . As it
becomes transmural , the serosal surface is involved . Contact
between V. peritoneum & P. peritoneum is established .
Somatic pain replaces visceral pain & the localization changes .
II. Clinical exercise
47-year- old woman suddenly develops
severe midepigastric pain . She cannot
find a position that eases the pain , but
the pain is not made worse by
movement . Examination does not
establish the presence of guarding ,
tenderness ,or rebound tenderness .
Analysis of case ( 2 )
This example illustrates all the characteristics of visceral pain.
The most common type of colic in women in this age
group is biliary . This diagnosis can be made with some
confidence . Confirmation by ultrasonography is required .
The epigastric localization is
associated with foregut origin
III. Clinical exercise
A 62 – year- old man is awakened at 2AM by severe
abdominal pain that he describes as generalized over
the entire abdomen . He complains also of pain in his
left shoulder . He does not move , & his breathing
shallow . He feels more comfortable sitting than lying .
He is sweating , tachycardiac , & in acute distress . The
abdomen is rigid . Tenderness , rebound tenderness , &
guarding are evident in all four quadrants . Percussion
over the liver demonstrates absence of liver dullness.
ANALYSIS OF CASE ( 3)
This Pat. has generalized peritonitis
This example illustrates all the characteristics of Somatic pain.
Pain in the left shoulder suggests irritation of the
diaphragm . Resonance over the liver is characteristic of
air in the peritoneal cavity .This features keeping with
bowel perforation .The most common site is the
duodenum secondary to an ulcer .
IV. Clinical exercise
A 76- year-old woman who lives alone in an apartment
is discovered to be ill by the daughter . She is taken to
the hospital, where examination reveals tenderness in
the right upper abdomen . Breathing is shallow and
rapid . Consolidation is suspected in the right lower lob
& confirmed radiographically . Her temperature & wbc .
are elevated .
Does this pat. Have right –sided
bronchopneumonia with pain referred to
the upper abdomen , or acute cholecystitis
with secondary changes in her chest
????????
Analysis of case ( 4 )
The differentiation is crucial
Deep breathing causes discomfort
,when the GB. is inflamed .
The pat. Takes shallow breaths & resists the urge to cough.
Atelectasis develops & the atelectatic segment , becomes
secondarily infected . Pat. Now has right lower lobe
pneumonia & pain in the RUQ. With guarding & rigidity.
If you operate the GB. When the correct DG. is pneumonia ,
the pat. will do poorly .
If you miss the DG. Of necrotic GB. The pat may do very
badly .
V. Clinical exercise
A 47- year-old woman develops a severe sharp , knifelike
pain in her Lt. Lower chest . She also complains of aching
pain at the tip of her Lt . Shoulder . The onset was sudden
& not associated with trauma or any other recognized cause
. It is difficult for her to take a deep breath or cough
effectively . The pain is controlled by analgesics prescribed
by her physician ,who thinks she had pleurisy .within 48
hours the pain subsides,& within a week she has recovered
uneventfully without further incident .
How should the
woman’s physician have
acted differently ??
V. Clinical exercise
This pat. had an infarct of
a peripheral segment of the
spleen
ANALYSIS OF CASE ( 5 )
The sudden onset of chest pain was not typical of
pleuritis secondary to bacterial or viral infection but
of some mechanical event .
The onset was in keeping with the sudden occlusion of a
vessel .The infarcted area produced an inflammatory reaction
that affected the diaphragmatic peritoneum , causing shoulder-
tip pain . The movement of the diaphragm brought the
infarcted segment into contact with the parietal peritoneum ,
mimicking pleuritic pain .
Diagnosis of acute abdominal pain hegazy

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Diagnosis of acute abdominal pain hegazy

  • 1.
  • 2. ANALYSIS OF ACUTE ABDOMINAL PAIN PREPARED BY DR. BOOQ YUOSEF & DR FIAZ FAZILI SURGICAL DEPTT.OHUD HOSPITAL PRESENTED BY DR.MOUSTAFA HEGAZY
  • 3. • depends in large measure on analysis of pain , which requires an understanding of the mechanisms of abdominal pain . DIAGNOSIS OF ACUTE ABDOMINAL PAIN
  • 4. OBJECTIVITY OF THIS LECTURE IS To examine the physiologic background of abdominal pain As An aid to accurate interpretation of Symptoms & Signs .
  • 5. Anatomic background  Parietal peritoneum clothes the anterior & posterior abdominal walls the under surface of the diaphragm & the cavity of the pelvis.( supplied segmentally by the spinal nerves ) .  Visceral peritoneum is the continuation of the parietal peritoneum, which leaves the posterior wall of the abdominal cavity to invest certain viscera therein . ( has no nerve supply ).
  • 6. 11 2 3 4 5 6 7 8 9 T6-T9 T6-T9 T8-T12 T8 T10 L2 S4 foregut midgut hindgut
  • 7. DEFINITION OF PAIN It is an unpleasant sensation of varying intensity . Pain fibers are stimulated any time a tissue is being damaged . However , it is not felt very long after the damage has been accomplished.
  • 8. 1 2 3 4 5 Mechanical trauma to the tissue . Excess heat or cold . STIMULANTS Chemical damage. Radiation damage . Inadequate blood flow.
  • 9. Intra-thoracic organs Abdominal Extra-abdominal Abdominal wall + Intra-peritoneal organsRetro-peritoneal organsPelvic organs Systemic dysfunction Diabetes ,tabes dorsalis porphyria Functional abdominal pain
  • 10. Types of abdominal pain 1 Visceral pain is primitive and therefore related to embryonic devlepment therefore related to embryologic development . Somatic pain is entirely different from visceral pain 2
  • 11. Visceral pain 1- Receptor ( Visceral peritoneum )
  • 12. Visceral pain 2 - Stimulus Pat. Experienced pain by traction ,distention & spasm The visceral peritoneum is insensitive to touch & heat or any condition that promotes an inflammatory reaction
  • 13. Visceral pain 3 - Mediation Thalamus Corpuscollosum Hypothalamus CerebralCortex Pons Medulla Spinalcord Cerebellum Autonomic nervous System Interpreted at the thalamic level of the brain
  • 14. Visceral pain 4- Specificity Vague , often dull , poorly described & associated with nausea & vomiting
  • 15. Visceral pain 5- Localization Is poor & the pat. Placing the entire hand over the involved region
  • 16. Somatic pain 1- Receptor Pain stimuli start in the parietal peritoneum , which is innervated by peripheral nerves P/ peritoneum
  • 17. Somatic pain 2- Stimulus Pat. experienced pain by TouchPressureHeatInflammation
  • 18. Somatic pain 3- Mediation Central nervous system & Interpreted at a specific cortical location
  • 19. Somatic pain 4- Specificity Precisely described as SharpKnifelikeCutting
  • 20. Somatic pain 5- Localization The pain is localized with great accuracy by the patient , who can often point to the site with one finger
  • 21. Analysis of pain need DATA COLLECTION 1 2 3 History Physical exam. Lab.inv. apply your medical knowledge***
  • 22. The history of pain betrays the diagnosis History of pain SiteMode of onsetNature of painSeverityRadiationDuration Factors influencing the clinical manifestation
  • 23. Site of pain & radiation Kidney Stomach & duodenum GB Pancreas Kidney Small bowel Caecum & rp.structure T. Colon bladder uterus & adnexae Sig. colon App. & Caecum Radiation of the pain GB. Pain radiates through to the back & to the right , to reach the tip of the shoulder blade Radiation indicates source of the pain & also the extent of the diseaseLower abdominal pain rarely radiate The structures in pelvis may radiate to the lower back or perineum Small bowel pain does not usually radiate but may move when somatic as well as visceral nerves become irritated Stomach & duodenal pain goes strait through the back Pancreatic pain tends to go through to the back but to the left Kidney pain may radiate down into the groin
  • 24. Mode of onset Sudden onset [The patient can tell you exactly when the pain started ] The pain that start suddenly has a mechanical basis Some thing has been RupturedTwistedOccluded
  • 25. Cont’ Mode of onset Gradual Onset ( The pat. Usually responds vaguely to questions about time of onset ) Non mechanical or chronic process
  • 26. Nature of pain Two Large Categories (1) Conditions associated with obstruction of a muscular conducting tube ( Small bowel , Ureter , Biliary ) (2) Conditions associated with inflammation ( Mild & Localized Response or Severe , Generalized Response )
  • 27. Obstruction Suddenprolonged Distention of the viscus ( constant stretching pain ) Colic pain = visceral pain Three Types ( 1 ) Biliary System = ( foregut ) Foregut pain is experienced in the epigastrium 1 (2) Small Intestine = ( midgut ) Pain is experienced in the periumbilical region (3) Renal system = ( retroperitoneal ) Pain is felt in the flank & radiates to the groin
  • 28. Important features of colic pain I. Pat . Is often restless & agitated during exacerbations. II. Pat. Does not experience a totally pain –free interlude. III. Colic pain is an intermittent pain . IV. Colic pain is an visceral pain . ( not influenced by changing relationships between the peritoneal layers ) V. Failing to demonstrate guarding , tenderness ? ????
  • 29. Inflammation2 Intra-abdominal inflammation is peritonitis Peritonitis causes somatic pain Peritonitis LocalizedGeneralized
  • 30. Inflammation2 Intra-abdominal inflammation is peritonitis Peritonitis causes somatic pain Contamination BY ChemicalsBacteriaTraumaForeign body
  • 31. Important features of somatic pain I. Pat. Laying quite in bed . ( movement is limited ) II. Examination may demonstrate guarding , tenderness . III. The pain is localized over the inflamed organ . IV. Fever , tachycardia & tachypnea are systemic manifestation for generalized inflammation .
  • 32. Ischemic pain Is a somatic pain Occlusion of blood supply cause Tissue Hypoxia With metabolic changes Necrosis After 6-12 h
  • 33. Severity of acute abdominal pain
  • 34. Factors influencing clinical manifestation (1) Extent of the pathologic process The more severe the process , the more impressive the manifestation .
  • 35. Factors influencing clinical manifestation (2) Time of Assessment Depending on the time of assessment , the characteristics will reflect what is present at that time – not previously & not subsequently .
  • 36. Factors influencing clinical manifestation (3) Emotional factors Objective criteria are more reliable than subjective factors .When there is a discrepancy between the severity of pain & objective findings ,caution should be exercised .
  • 37. Factors influencing clinical manifestation (4) The Patient’s Intelligence A clinical history is only as reliable as its source . If the pat. is Intellectually impairedSenileVery youngOr PsychoticVery ill The information obtained must be interpreted carefully . Objective & subjective findings should be compatible .
  • 38. Factors influencing clinical manifestation (5) Level of consciousness Some neurologic problems make the interpretation of acute abdominal pain difficult . UnconsciousnessParaplegiaSympathetic denervation Bizarre manifestation of abd. Pain .
  • 39. Factors influencing clinical manifestation (6) Drugs Many drugs influence both the character , perception & the course and effects of disease . Corticosteroids Suppress the inflammatory response Sedatives Influence pat. Recognition of problems Analgesics Decrease pain ( minimized or overlooked )
  • 40. Medical causes of abdominal pain ■ Diabetic ketoacidosis ■ Porphyria ■ Pain arising from the heart or lungs
  • 41. Embryological division of the gastrointestinal tract • Foregut: • Oesophagus • Stomach • Duodenum – first and second parts • Pancreas • Liver • Gall bladder
  • 42. • Midgut • Duodenum –third and fourth parts • Jejunum • Ileum • Right colon • Transverse colon • Appendix
  • 44. • Origins and presentation of abdominal pain • Parietal Well localised but can radiate forwards and backwards along the dermatome • Visceral Poorly localised; associated with sweating and nausea • Retroperitoneal Pain in the back • The nature of the pain will change as the cause evolves
  • 45. Sources of pain that evolve over hours or days ■ Acute Appendicitis Cholecystitis Salpingitis Mesenteric adenitis • ■ Infarction • ■ Free blood in the peritoneum
  • 46. Sources of pain that evolve over hours or days ■ Acute Appendicitis Cholecystitis Salpingitis Mesenteric adenitis • ■ Infarction • ■ Free blood in the
  • 47. Causes of sudden onset abdominal pain ■Perforation Appendix Stomach Duodenum Colon
  • 48. Aggravating features of pain Aggravating feature Moving or coughing Interpretation Peritoneal inflammation Aggravating feature Patient rolls around with pain Colic Interpretation (suggests obstruction of viscus)
  • 50. I. Clinical Exercise A male pat. ,aged 17, develops mild periumbilical discomfort not influenced by activity . Several hours later , his discomfort increases but is now situated in the right lower quadrant .Movement becomes painful . Examination reveals localized pain , tenderness , and rebound tenderness localized to a small area in the right lower quadrant .
  • 51. Analysis of case ( 1 ) The initial pain is visceral & midgut in origin but is replaced by somatic pain . The most common problem in this region & this age is app . Why visceral pain ???? The inflammation starts intraluminally , & the app. Becomes distended. Stretching is a stimulus for visceral pain . As it becomes transmural , the serosal surface is involved . Contact between V. peritoneum & P. peritoneum is established . Somatic pain replaces visceral pain & the localization changes .
  • 52. II. Clinical exercise 47-year- old woman suddenly develops severe midepigastric pain . She cannot find a position that eases the pain , but the pain is not made worse by movement . Examination does not establish the presence of guarding , tenderness ,or rebound tenderness .
  • 53. Analysis of case ( 2 ) This example illustrates all the characteristics of visceral pain. The most common type of colic in women in this age group is biliary . This diagnosis can be made with some confidence . Confirmation by ultrasonography is required . The epigastric localization is associated with foregut origin
  • 54. III. Clinical exercise A 62 – year- old man is awakened at 2AM by severe abdominal pain that he describes as generalized over the entire abdomen . He complains also of pain in his left shoulder . He does not move , & his breathing shallow . He feels more comfortable sitting than lying . He is sweating , tachycardiac , & in acute distress . The abdomen is rigid . Tenderness , rebound tenderness , & guarding are evident in all four quadrants . Percussion over the liver demonstrates absence of liver dullness.
  • 55. ANALYSIS OF CASE ( 3) This Pat. has generalized peritonitis This example illustrates all the characteristics of Somatic pain. Pain in the left shoulder suggests irritation of the diaphragm . Resonance over the liver is characteristic of air in the peritoneal cavity .This features keeping with bowel perforation .The most common site is the duodenum secondary to an ulcer .
  • 56. IV. Clinical exercise A 76- year-old woman who lives alone in an apartment is discovered to be ill by the daughter . She is taken to the hospital, where examination reveals tenderness in the right upper abdomen . Breathing is shallow and rapid . Consolidation is suspected in the right lower lob & confirmed radiographically . Her temperature & wbc . are elevated . Does this pat. Have right –sided bronchopneumonia with pain referred to the upper abdomen , or acute cholecystitis with secondary changes in her chest ????????
  • 57. Analysis of case ( 4 ) The differentiation is crucial Deep breathing causes discomfort ,when the GB. is inflamed . The pat. Takes shallow breaths & resists the urge to cough. Atelectasis develops & the atelectatic segment , becomes secondarily infected . Pat. Now has right lower lobe pneumonia & pain in the RUQ. With guarding & rigidity. If you operate the GB. When the correct DG. is pneumonia , the pat. will do poorly . If you miss the DG. Of necrotic GB. The pat may do very badly .
  • 58. V. Clinical exercise A 47- year-old woman develops a severe sharp , knifelike pain in her Lt. Lower chest . She also complains of aching pain at the tip of her Lt . Shoulder . The onset was sudden & not associated with trauma or any other recognized cause . It is difficult for her to take a deep breath or cough effectively . The pain is controlled by analgesics prescribed by her physician ,who thinks she had pleurisy .within 48 hours the pain subsides,& within a week she has recovered uneventfully without further incident .
  • 59. How should the woman’s physician have acted differently ?? V. Clinical exercise
  • 60. This pat. had an infarct of a peripheral segment of the spleen ANALYSIS OF CASE ( 5 ) The sudden onset of chest pain was not typical of pleuritis secondary to bacterial or viral infection but of some mechanical event . The onset was in keeping with the sudden occlusion of a vessel .The infarcted area produced an inflammatory reaction that affected the diaphragmatic peritoneum , causing shoulder- tip pain . The movement of the diaphragm brought the infarcted segment into contact with the parietal peritoneum , mimicking pleuritic pain .