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CARDIOVASCULAR DISEASES Nelia B. Perez RN, MSN PCU – MJCN BSN 2013
THE CARDIOVASCULAR SYSTEM
GENERAL CARDIAC ASSESSMENT Health history Demographic information Family/genetic history Cultural/social factors Risk factors Modifiable: High blood cholesterol, obesity, smoking, stress,      hypertension, diabetes mellitus. Nonmodifiable: Family history, increasing age, gender, race
Pathophysiology
ASSESSING CHEST PAIN
COMPARISON OF PHYSICAL CAUSES OF CHEST PAIN
COMPARISON OF PHYSICAL CAUSES OF CHEST PAIN
Angina Pectoris / Myocardial Ischemia ,[object Object]
Angina – to choke
Occurs when blood supply is inadequate to meet the heart’s metabolic demands
Symptomatic paroxysmal chest pain or pressure sensation associated with transient ischemia,[object Object]
Types Stable angina – the common initial manifestation of  a heart disease ,[object Object]
Pain is precipitated by increased work demands of the heart (i.e.. physical exertion, exposure to cold, & emotional stress)
Pain location: precordial or substernal chest area,[object Object]
constricting, squeezing, or suffocating sensation
Usually steady, increasing in intensity only at the onset & end of attack
May radiate to left shoulder, arm, jaw, or other chest areas
Duration: < 15mins
Relieved by rest (preferably sitting or standing with support) or by use of  NTG,[object Object]
Cause: spasm of coronary arteries (vasospasm) due to coronary artery stenosis
Mechanism is uncertain (may be from hyperactive sympathetic responses, mishandling defects of calcium in smooth vascular muscles, reduced prostaglandin I2 production),[object Object],[object Object]
Cont… ,[object Object]
Tx: directed towards MI prevention/li>,[object Object]
Stress reduction
Diet changes
Avoidance of cold
PTCA (percutaneoustransluminal coronary angioplasty) may be indicated if with severe artery  occlusion,[object Object]
patch (Deponit, Transderm-NTG)
sublingual (Nitrostat)
oral (Nitroglyn)
IV (Nitro-Bid)
Β-adrenergic blockers:
Propanolol (Inderal)
Atenolol (Tenormin)
Metoprolol (Lopressor)
Calcium channel blockers:
Nifedipine (Calcibloc, Adalat)
Diltiazem (Cardizem)
Lipid lowering agents –statins:
Simvastatin
Anti-coagulants:
ASA (Aspirin)
Heparin sodium
Warfarin (Coumadin),[object Object]
Class II – angina occurs on walking or going up the stairs rapidly or after meals, walking uphill, walking more than 2 blocks on the level or going more than 1 flight of ordinary stairs at normal pace, under emotional stress, or in cold
Class III – angina occurs on walking 1-2 blocks on the level or going 1 flight of ordinary stairs at normal pace
Class IV – angina occurs even at rest,[object Object]
E.g.. White meat – chicken w/o skin, fish
Stop smoking & avoid alcohol
Activity restrictions are placed within client’s limitations
NTGs – max of 3doses at 5-min intervals
Stinging sensation under the tongue for SL is normal
Advise clients to always carry 3 tablets
Store meds in cool, dry place, air-tight amber bottles & change stocks every 6months
Inform clients that headache, dizziness, flushed face are common side effects. ,[object Object]
For patches, rotate skin sites usually on chest wall
Instrct on evaluation of effectiveness based on pain relief
Propanolols causes bronchospasm & hypoglycemia, do not administer to asthmatic & diabetic clients
Heparin – monitor bleeding tendencies (avoid punctures, use of soft-bristled toothbrush); monitor PTT levels; usedfor 2wks max; do not massage if via SC; have protaminesulfate available
Coumadin – monitor for bleeding & PT; always have vit K readily available (avoid green leafyveggies),[object Object]
Causes: atherosclerotic plaque disruption or significant CHD, cocaine use (risk factor)
 Defining guidelines: (3 presentations)Symptoms at rest (usually prolonged, i.e.. >20mins) New onset exertional angina (increased in severity of at least 1 class – to at least class III) in <2months Recent acceleration of angina to at least class III in <2months
[object Object]
When chest pain has been unremitting for >20mins, possibility of ST-Segment Elevation MI is usually considered,[object Object]
Characterized by ischemic death of myocardial tissue associated with atherosclerotic disease of coronary arteries
Area of infarction is determined by the affected coronary artery & its distribution of blood flow (right coronary artery, left anterior descending artery, left circumflex artery),[object Object]
Typical ECG changes: ST-segment elevation, Q wave prolongation, T wave inversion,[object Object]
Feeling of impending doom,[object Object]
Heart failure & cardiogenic shock – profound LV failure from massive MI resulting to low cardiac output
Thromboemboli – leads to immobility & impaired cardiac function contributing to blood stasis in veins
Rupture of myocardium
Ventricular aneurysms – decreases pumping efficiency of heart & increases work of LV,[object Object]
Tissue Changes After MI
Management of MI ,[object Object],	    - O2 therapy via nasal prongs  		- adequate analgesia (Morphine via IV – also has vasodilator property) 		- ECG monitoring 		-sublingual NTG (unless contraindicated; IV may be given to limit infarction size & most effective if given within 4hrs of onset) ,[object Object]
Anti-arrhythmics: lidocaine, atropine, propanolol
Anticoagulants & antiplatelets: ASA, heparin
Stool softeners,[object Object]
ASSESSMENT Subjective data: PAIN!!! Nausea SOB Apprehension Objective data: VS Diaphoresis Emotional restlessness
ANALYSIS / NURSING DIAGNOSES Decreased cardiac output related to myocardial damage Impaired gas exchange related to poor perfusion, shock Pain related to myocardial ischemia Activity intolerance related to pain or inadequate oxygenation Fear related to possibility of death
NURSING CARE PLAN Goal # 1: reduce pain / discomfort Narcotics – morphine; note response; Avoid IM Humidified oxygen 2-4 L/min; mouth care – O2 is drying Position: semi-Fowler’s to improve ventilation
NURSING CARE PLAN Goal # 2: maintain adequate circulation; stabilize heart rhythm Monitor VS/UO; observe for cardiogenic shock Monitor ECG for arrhythmias Medications: antiarrhythmics; anticoagulants; thrombolytics Diagnostics: cardiac catheterizations, CAB surgery Recognize heart failure: edema, cyanosis, dyspnea, crackles Check labs: troponin, blood gases, electrolytes, clotting time CVP: (5-15 cm H2O) increases with heart failure ROM of lower extremities; antiembolic stockings
NURSING CARE PLAN Goal # 3: decrease oxygen demand/promote oxygenation, reduce cardiac workload O2 as ordered Activity: bedrest (24-48 H) with bedside commode; planned rest periods; control visitors Position: semi-Fowler’s to facilitate lung expansion and decrease venous return Anticipate needs of client: call light, water / Reassurance Assist with feeding, turning Environment: quiet and comfortable Medications: CCBs, vasodilators, cardiotonics
NURSING CARE PLAN Goal # 4: maintain fluid electrolyte, nutritional status IV (KVO); CVP; vital signs UO: 30 cc/hr Labs: electrolytes (Na, K, Mg) Monitor ECG Diet: progressive low calorie, low sodium, low cholesterol, low fat, without caffeine
NURSING CARE PLAN Goal # 5: facilitate fecal elimination Medications: stool softeners to prevent Valsalva maneuver; mouth breathing during bowel movement Bedside commode
NURSING CARE PLAN Goal # 6: provide emotional support Recognize fear of dying: denial, anger, withdrawal Encourage expression of feelings, fears, concerns Discuss rehabilitation, lifestyle changes: prevent cardiac-invalid syndrome by promoting self-care activities, independence
NURSING CARE PLAN Goal # 7: promote sexual functioning Encourage verbalization of concerns regarding activity, inadequacy, limitations, expectations – include partner (usually resume activity 5-8 wks after uncomplicated MI or when client can climb 2 flights of stairs Identify need for referral for sexual counselling
NURSING CARE PLAN Goal # 8: health teaching Diagnosis and treatment regimen Caution when to avoid sexual activity: after heavy meal, alcohol ingestion; when fatigued, stressed; with unfamiliar partners; in extreme temperatures Information about sexual activity: less fatiguing positions Support groups / Follow-up care Medications: administration, importance, untoward effects; pulse taking Control risk factors: rest, diet, exercise, no smoking, weight control, stress reduction
EVALUATION No complications: stable vital signs; relief of pain Adheres to medication regimen Activity tolerance is increased Reduction or modification of risk factors
CONGESTIVE HEART FAILURE inability of the heart to pump sufficient blood to meet the needs of the tissue for oxygen and nutrient.
PATHOPHYSIOLOGY
ASSESSMENT Subjective data: Shortness of breath Orthopnea (sleeps on two or more pillows) Paroxysmal nocturnal dyspnea (sudden breathlessness during sleep) Dyspnea on exertion (climbing stairs) Apprehension; anxiety; irritability Fatigue; weakness Reported weight gain; feeling of puffiness
ASSESSMENT Objective data: VS: BP: decreasing systolic; narrowing pulse pressure Pulse: pulsusalternans (alternating strong-weak-strong cardiac contraction); increased. Respirations: crackles; Cheyne-Stokes Edema: dependent, pitting (1+ to 4+ mm) Liver: enlarged, tender Distended neck veins Chest X-ray: enlarged heart; dilated pulmonary vessels; lung edema
Left Ventricular Compared with Right Ventricular Heart Failure
ANALYSIS / NURSING DIAGNOSES Decreased cardiac output related to decreased myocardial contractility Activity intolerance related to generalized body weakness and inadequate oxygenation Fatigue related to edema and poor oxygenation
Fluid volume excess related to compensatory mechanisms Impaired gas exchange related to pulmonary congestion Anxiety related to shortness of breath Sleep pattern disturbance related to paroxysmal nocturnal disturbance
NURSING CARE PLAN Goal # 1: provide physical rest/ reduce emotional stimuli Position: sitting or semi-Fowler’s until tachycardia, dyspnea, edema resolved; change position frequently; pillows for support Rest: planned periods; limit visitors, activity, noise. Chair and commode privileges Support: stay with client who is anxious; have family member who is supportive present; administer sedatives/tranquilizers as ordered Warm fluids if appropriate
NURSING CARE PLAN Goal # 2: provide for relief of respiratory distress; reduce cardiac workload Oxygen: low flow rate; encourage deep breathing (5-10 min q 2H); auscultate breath sounds for congestion, pulmonary edema. Position: elevating head of bed 20-25 cm (8-10 in) alleviates pulmonary congestion Medications – digitalis, ACE inhibitors, inotropic agents, diuretics, tranquilizers, vasodilators
NURSING CARE PLAN Goal # 3: provide for special safety needs Skin care: Inspect, massage, lubricate bony prominences Use foot cradle, heel protectors; sheepskin Side rails up if hypoxic (disoriented) Vital signs: monitor for signs of fatigue, pulmonary emboli ROM: active, passive; elastic stockings
NURSING CARE PLAN Goal # 4: maintain fluid and electrolyte balance, nutritional status Urine output: 30 cc/hr minimum; estimate insensible loss in client who s diaphoretic. Monitor BUN, serum creatinine, and electrolytes. Daily weight; same time, clothes, scale IV: IV infusion pump to avoid circulatory overload; strict I/O Diet Low sodium Small, frequent feedings Discuss food preferences with client.
NURSING CARE PLAN Goal # 5: health teaching Diet restrictions; meal preparation Activity restrictions; planned rest periods Medications: schedule (e.g. diuretics in early morning); purpose; dosage; side effects (pulse taking, daily weights, intake of potassium-containing foods) Refer to available communityresources for dietary assistance, weight reduction, exercise program.
EVALUATION	 Increase in activity level tolerance – fatigue decreased No complications – pulmonary edema, respiratory distress Reduction in dependent edema
DAY 3  of  Cardiovascular diseases
hyperlipidemia means high lipid levels.  High lipid levels can speed up a process called atherosclerosis, or hardening of the arteries.
Most hyperlipidemia is caused by lifestyle habits or treatable medical conditions. Lifestyle contributors include obesity, not exercising, and smoking. Conditions that cause hyperlipidemia include diabetes, kidney disease, pregnancy, and an underactive thyroid gland.
You can also inherit hyperlipidemia. The cause may be genetic if you have a normal body weight and other members of your family have hyperlipidemia.
You have a greater chance of developing hyperlipidemia if you are a man older than age 45 or a woman older than age 55. If a close relative had early heart disease (father or brother affected before age 55, mother or sister affected before age 65), you also have an increased risk.
Treatment of Hyperlipidemia It is necessary to first identify and treat any potential underlying medical problems, such as diabetes or hypothyroidism, that may contribute to hyperlipidemia. Treatment of hyperlipidemia itself includes dietary changes, weight reduction and exercise. If lifestyle modifications cannot bring about optimal lipid levels, then medications may be necessary.
Medications most commonly used to treat high LDL cholesterol levels are statins, such as atorvastatin (Lipitor) or simvastatin (Mevacor). These medications work by reducing the production of cholesterol within the body. 
CARDIOMYOPATHIESHeart muscle disease associated with cardiac dysfunction CARDIOMYOPATHIES 1. Dilated Cardiomyopathy 2. Hypertrophic Cardiomyopathy 3. Restrictive cardiomyopathy
DILATED CARDIOMYOPATHY ASSOCIATED FACTORS 1. Heavy alcohol intake 2. Pregnancy 3. Viral infection 4. Idiopathic
DILATED CARDIOMYOPATHYPATHOPHYSIOLOGY Diminished contractile proteins - poor contraction -decreased blood ejection -increased blood remaining in the ventricle -ventricular stretching and dilatation.- SYSTOLIC DYSFUNCTION
HYPERTROPHIC CARDIOMYOPATHY Associated factors: 1. Genetic 2. Idiopathic
HYPERTROPHIC CARDIOMYOPATHY Pathophysiology Increased size of myocardium - reduced ventricular volume - increased resistance to ventricular filling - diastolic dysfunction
RESTRICTIVE CARDIOMYOPATHY Associated factors 1. Infiltrative diseases like AMYLOIDOSIS 2. Idiopathic
RESTRICTIVE CARDIOMYOPATHYPathophysiology Rigid ventricular wall -impaired stretch and diastolic filling -decreased output  -  Diastolic dysfunction
CARDIOMYOPATHIES Assessment findings 1. PND 2. Orthopnea 3. Edema 4. Chest pain 5. Palpitations 6. dizziness 7. Syncope with exertion
CARDIOMYOPATHIES Laboratory Findings 1. CXR- may reveal cardiomegaly 2. ECHOCARDIOGRAM 3. ECG 4. Myocardial Biopsy
CARDIOMYOPATHIES Medical Management 1. Surgery 2. pacemaker insertion 3. Pharmacological drugs for symptom relief
CARDIOMYOPATHIES Nursing Management 1.Improve cardiac output Adequate rest Oxygen therapy Low sodium diet

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Cardiovascular diseases modified

  • 1. CARDIOVASCULAR DISEASES Nelia B. Perez RN, MSN PCU – MJCN BSN 2013
  • 3. GENERAL CARDIAC ASSESSMENT Health history Demographic information Family/genetic history Cultural/social factors Risk factors Modifiable: High blood cholesterol, obesity, smoking, stress, hypertension, diabetes mellitus. Nonmodifiable: Family history, increasing age, gender, race
  • 5.
  • 7. COMPARISON OF PHYSICAL CAUSES OF CHEST PAIN
  • 8. COMPARISON OF PHYSICAL CAUSES OF CHEST PAIN
  • 9.
  • 11. Occurs when blood supply is inadequate to meet the heart’s metabolic demands
  • 12.
  • 13.
  • 14. Pain is precipitated by increased work demands of the heart (i.e.. physical exertion, exposure to cold, & emotional stress)
  • 15.
  • 16. constricting, squeezing, or suffocating sensation
  • 17. Usually steady, increasing in intensity only at the onset & end of attack
  • 18. May radiate to left shoulder, arm, jaw, or other chest areas
  • 20.
  • 21. Cause: spasm of coronary arteries (vasospasm) due to coronary artery stenosis
  • 22.
  • 23.
  • 24.
  • 28.
  • 40. Lipid lowering agents –statins:
  • 45.
  • 46. Class II – angina occurs on walking or going up the stairs rapidly or after meals, walking uphill, walking more than 2 blocks on the level or going more than 1 flight of ordinary stairs at normal pace, under emotional stress, or in cold
  • 47. Class III – angina occurs on walking 1-2 blocks on the level or going 1 flight of ordinary stairs at normal pace
  • 48.
  • 49. E.g.. White meat – chicken w/o skin, fish
  • 50. Stop smoking & avoid alcohol
  • 51. Activity restrictions are placed within client’s limitations
  • 52. NTGs – max of 3doses at 5-min intervals
  • 53. Stinging sensation under the tongue for SL is normal
  • 54. Advise clients to always carry 3 tablets
  • 55. Store meds in cool, dry place, air-tight amber bottles & change stocks every 6months
  • 56.
  • 57. For patches, rotate skin sites usually on chest wall
  • 58. Instrct on evaluation of effectiveness based on pain relief
  • 59. Propanolols causes bronchospasm & hypoglycemia, do not administer to asthmatic & diabetic clients
  • 60. Heparin – monitor bleeding tendencies (avoid punctures, use of soft-bristled toothbrush); monitor PTT levels; usedfor 2wks max; do not massage if via SC; have protaminesulfate available
  • 61.
  • 62. Causes: atherosclerotic plaque disruption or significant CHD, cocaine use (risk factor)
  • 63. Defining guidelines: (3 presentations)Symptoms at rest (usually prolonged, i.e.. >20mins) New onset exertional angina (increased in severity of at least 1 class – to at least class III) in <2months Recent acceleration of angina to at least class III in <2months
  • 64.
  • 65.
  • 66. Characterized by ischemic death of myocardial tissue associated with atherosclerotic disease of coronary arteries
  • 67.
  • 68.
  • 69.
  • 70. Heart failure & cardiogenic shock – profound LV failure from massive MI resulting to low cardiac output
  • 71. Thromboemboli – leads to immobility & impaired cardiac function contributing to blood stasis in veins
  • 73.
  • 75.
  • 78.
  • 79.
  • 80. ASSESSMENT Subjective data: PAIN!!! Nausea SOB Apprehension Objective data: VS Diaphoresis Emotional restlessness
  • 81. ANALYSIS / NURSING DIAGNOSES Decreased cardiac output related to myocardial damage Impaired gas exchange related to poor perfusion, shock Pain related to myocardial ischemia Activity intolerance related to pain or inadequate oxygenation Fear related to possibility of death
  • 82. NURSING CARE PLAN Goal # 1: reduce pain / discomfort Narcotics – morphine; note response; Avoid IM Humidified oxygen 2-4 L/min; mouth care – O2 is drying Position: semi-Fowler’s to improve ventilation
  • 83. NURSING CARE PLAN Goal # 2: maintain adequate circulation; stabilize heart rhythm Monitor VS/UO; observe for cardiogenic shock Monitor ECG for arrhythmias Medications: antiarrhythmics; anticoagulants; thrombolytics Diagnostics: cardiac catheterizations, CAB surgery Recognize heart failure: edema, cyanosis, dyspnea, crackles Check labs: troponin, blood gases, electrolytes, clotting time CVP: (5-15 cm H2O) increases with heart failure ROM of lower extremities; antiembolic stockings
  • 84. NURSING CARE PLAN Goal # 3: decrease oxygen demand/promote oxygenation, reduce cardiac workload O2 as ordered Activity: bedrest (24-48 H) with bedside commode; planned rest periods; control visitors Position: semi-Fowler’s to facilitate lung expansion and decrease venous return Anticipate needs of client: call light, water / Reassurance Assist with feeding, turning Environment: quiet and comfortable Medications: CCBs, vasodilators, cardiotonics
  • 85. NURSING CARE PLAN Goal # 4: maintain fluid electrolyte, nutritional status IV (KVO); CVP; vital signs UO: 30 cc/hr Labs: electrolytes (Na, K, Mg) Monitor ECG Diet: progressive low calorie, low sodium, low cholesterol, low fat, without caffeine
  • 86. NURSING CARE PLAN Goal # 5: facilitate fecal elimination Medications: stool softeners to prevent Valsalva maneuver; mouth breathing during bowel movement Bedside commode
  • 87. NURSING CARE PLAN Goal # 6: provide emotional support Recognize fear of dying: denial, anger, withdrawal Encourage expression of feelings, fears, concerns Discuss rehabilitation, lifestyle changes: prevent cardiac-invalid syndrome by promoting self-care activities, independence
  • 88. NURSING CARE PLAN Goal # 7: promote sexual functioning Encourage verbalization of concerns regarding activity, inadequacy, limitations, expectations – include partner (usually resume activity 5-8 wks after uncomplicated MI or when client can climb 2 flights of stairs Identify need for referral for sexual counselling
  • 89. NURSING CARE PLAN Goal # 8: health teaching Diagnosis and treatment regimen Caution when to avoid sexual activity: after heavy meal, alcohol ingestion; when fatigued, stressed; with unfamiliar partners; in extreme temperatures Information about sexual activity: less fatiguing positions Support groups / Follow-up care Medications: administration, importance, untoward effects; pulse taking Control risk factors: rest, diet, exercise, no smoking, weight control, stress reduction
  • 90. EVALUATION No complications: stable vital signs; relief of pain Adheres to medication regimen Activity tolerance is increased Reduction or modification of risk factors
  • 91. CONGESTIVE HEART FAILURE inability of the heart to pump sufficient blood to meet the needs of the tissue for oxygen and nutrient.
  • 93. ASSESSMENT Subjective data: Shortness of breath Orthopnea (sleeps on two or more pillows) Paroxysmal nocturnal dyspnea (sudden breathlessness during sleep) Dyspnea on exertion (climbing stairs) Apprehension; anxiety; irritability Fatigue; weakness Reported weight gain; feeling of puffiness
  • 94. ASSESSMENT Objective data: VS: BP: decreasing systolic; narrowing pulse pressure Pulse: pulsusalternans (alternating strong-weak-strong cardiac contraction); increased. Respirations: crackles; Cheyne-Stokes Edema: dependent, pitting (1+ to 4+ mm) Liver: enlarged, tender Distended neck veins Chest X-ray: enlarged heart; dilated pulmonary vessels; lung edema
  • 95. Left Ventricular Compared with Right Ventricular Heart Failure
  • 96. ANALYSIS / NURSING DIAGNOSES Decreased cardiac output related to decreased myocardial contractility Activity intolerance related to generalized body weakness and inadequate oxygenation Fatigue related to edema and poor oxygenation
  • 97. Fluid volume excess related to compensatory mechanisms Impaired gas exchange related to pulmonary congestion Anxiety related to shortness of breath Sleep pattern disturbance related to paroxysmal nocturnal disturbance
  • 98. NURSING CARE PLAN Goal # 1: provide physical rest/ reduce emotional stimuli Position: sitting or semi-Fowler’s until tachycardia, dyspnea, edema resolved; change position frequently; pillows for support Rest: planned periods; limit visitors, activity, noise. Chair and commode privileges Support: stay with client who is anxious; have family member who is supportive present; administer sedatives/tranquilizers as ordered Warm fluids if appropriate
  • 99. NURSING CARE PLAN Goal # 2: provide for relief of respiratory distress; reduce cardiac workload Oxygen: low flow rate; encourage deep breathing (5-10 min q 2H); auscultate breath sounds for congestion, pulmonary edema. Position: elevating head of bed 20-25 cm (8-10 in) alleviates pulmonary congestion Medications – digitalis, ACE inhibitors, inotropic agents, diuretics, tranquilizers, vasodilators
  • 100. NURSING CARE PLAN Goal # 3: provide for special safety needs Skin care: Inspect, massage, lubricate bony prominences Use foot cradle, heel protectors; sheepskin Side rails up if hypoxic (disoriented) Vital signs: monitor for signs of fatigue, pulmonary emboli ROM: active, passive; elastic stockings
  • 101. NURSING CARE PLAN Goal # 4: maintain fluid and electrolyte balance, nutritional status Urine output: 30 cc/hr minimum; estimate insensible loss in client who s diaphoretic. Monitor BUN, serum creatinine, and electrolytes. Daily weight; same time, clothes, scale IV: IV infusion pump to avoid circulatory overload; strict I/O Diet Low sodium Small, frequent feedings Discuss food preferences with client.
  • 102. NURSING CARE PLAN Goal # 5: health teaching Diet restrictions; meal preparation Activity restrictions; planned rest periods Medications: schedule (e.g. diuretics in early morning); purpose; dosage; side effects (pulse taking, daily weights, intake of potassium-containing foods) Refer to available communityresources for dietary assistance, weight reduction, exercise program.
  • 103. EVALUATION Increase in activity level tolerance – fatigue decreased No complications – pulmonary edema, respiratory distress Reduction in dependent edema
  • 104. DAY 3 of Cardiovascular diseases
  • 105. hyperlipidemia means high lipid levels.  High lipid levels can speed up a process called atherosclerosis, or hardening of the arteries.
  • 106. Most hyperlipidemia is caused by lifestyle habits or treatable medical conditions. Lifestyle contributors include obesity, not exercising, and smoking. Conditions that cause hyperlipidemia include diabetes, kidney disease, pregnancy, and an underactive thyroid gland.
  • 107. You can also inherit hyperlipidemia. The cause may be genetic if you have a normal body weight and other members of your family have hyperlipidemia.
  • 108. You have a greater chance of developing hyperlipidemia if you are a man older than age 45 or a woman older than age 55. If a close relative had early heart disease (father or brother affected before age 55, mother or sister affected before age 65), you also have an increased risk.
  • 109. Treatment of Hyperlipidemia It is necessary to first identify and treat any potential underlying medical problems, such as diabetes or hypothyroidism, that may contribute to hyperlipidemia. Treatment of hyperlipidemia itself includes dietary changes, weight reduction and exercise. If lifestyle modifications cannot bring about optimal lipid levels, then medications may be necessary.
  • 110. Medications most commonly used to treat high LDL cholesterol levels are statins, such as atorvastatin (Lipitor) or simvastatin (Mevacor). These medications work by reducing the production of cholesterol within the body. 
  • 111. CARDIOMYOPATHIESHeart muscle disease associated with cardiac dysfunction CARDIOMYOPATHIES 1. Dilated Cardiomyopathy 2. Hypertrophic Cardiomyopathy 3. Restrictive cardiomyopathy
  • 112. DILATED CARDIOMYOPATHY ASSOCIATED FACTORS 1. Heavy alcohol intake 2. Pregnancy 3. Viral infection 4. Idiopathic
  • 113. DILATED CARDIOMYOPATHYPATHOPHYSIOLOGY Diminished contractile proteins - poor contraction -decreased blood ejection -increased blood remaining in the ventricle -ventricular stretching and dilatation.- SYSTOLIC DYSFUNCTION
  • 114. HYPERTROPHIC CARDIOMYOPATHY Associated factors: 1. Genetic 2. Idiopathic
  • 115. HYPERTROPHIC CARDIOMYOPATHY Pathophysiology Increased size of myocardium - reduced ventricular volume - increased resistance to ventricular filling - diastolic dysfunction
  • 116. RESTRICTIVE CARDIOMYOPATHY Associated factors 1. Infiltrative diseases like AMYLOIDOSIS 2. Idiopathic
  • 117. RESTRICTIVE CARDIOMYOPATHYPathophysiology Rigid ventricular wall -impaired stretch and diastolic filling -decreased output - Diastolic dysfunction
  • 118. CARDIOMYOPATHIES Assessment findings 1. PND 2. Orthopnea 3. Edema 4. Chest pain 5. Palpitations 6. dizziness 7. Syncope with exertion
  • 119. CARDIOMYOPATHIES Laboratory Findings 1. CXR- may reveal cardiomegaly 2. ECHOCARDIOGRAM 3. ECG 4. Myocardial Biopsy
  • 120. CARDIOMYOPATHIES Medical Management 1. Surgery 2. pacemaker insertion 3. Pharmacological drugs for symptom relief
  • 121. CARDIOMYOPATHIES Nursing Management 1.Improve cardiac output Adequate rest Oxygen therapy Low sodium diet
  • 122. CARDIOMYOPATHIES Nursing Management 2. Increase patient tolerance Schedule activities with rest periods in between
  • 123. CARDIOMYOPATHIES Nursing Management 3. Reduce patient anxiety Support Offer information about transplantations Support family in anticipatory grieving
  • 124. Infective endocarditis Infection of the heart valves and the endothelial surface of the heart Can be acute or chronic
  • 125. Infective endocarditis Etiologic factors 1. Bacteria- Organism depends on several factors 2. Fungi
  • 126. Infective endocarditis Risk factors 1. Prosthetic valves 2. Congenital malformation 3. Cardiomyopathy 4. IV drug users 5. Valvular dysfunctions
  • 127. Infective endocarditis Pathophysiology Direct invasion of microbes  microbes adhere to damaged valve surface and proliferate  damage attracts platelets causing clot formation  erosion of valvular leaflets and vegetation can embolize
  • 128. Infective endocarditis Assessment findings 1. Intermittent HIGH fever 2. anorexia, weight loss 3. cough, back pain and joint pain 4. splinter hemorrhages under nails
  • 129. Infective endocarditis Assessment findings 5. Osler’s nodes- painful nodules on fingerpads 6. Roth’s spots- pale hemorrhages in the retina
  • 130. Infective endocarditis Assessment findings 7. Heart murmurs 8. Heart failure
  • 131. Infective endocarditis Prevention Antibiotic prophylaxis if patient is undergoing procedures like dental extractions, bronchoscopy, surgery, etc.
  • 132. Infective endocarditis LABORATORY EXAM Blood Cultures to determine the exact organism
  • 133. Infective endocarditis Nursing management 1. regular monitoring of temperature, heart sounds 2. manage infection 3. long-term antibiotic therapy
  • 134. Infective endocarditis Medical management 1. Pharmacotherapy IV antibiotic for 2-6 weeks Antifungal agents are given – amphotericin B
  • 135.
  • 137.
  • 143.
  • 144. CARDIOGENIC SHOCK LABORATORY FINDINGSIncreased CVP Normal is 4-10 cmH2O
  • 145.
  • 146. 1. Place patient in a modified Trendelenburg (shock ) position
  • 147. 2. Administer IVF, vasopressors and inotropics such as DOPAMINE and DOBUTAMINE
  • 149.
  • 150. 6. Monitor urinary output, BP and pulses
  • 151.
  • 152.
  • 153.
  • 155. 2. Complication of Myocardial infarction
  • 157.
  • 158. 1. BECK’s Triad- Jugular vein distention, hypotension and distant/muffled heart sound
  • 161.
  • 165.
  • 167.
  • 168. 1. Assist in PERICARDIOCENTESIS
  • 170. 3. Monitor ECG, urine output and BP
  • 171.
  • 174. Elevate head of bed 45-60 degrees
  • 175.
  • 176.
  • 177. 1. Primary or ESSENTIAL
  • 180.
  • 181.
  • 182.
  • 183. Alterations inBlood Flow in the Systemic Circulation
  • 184. Buerger’s Disease Also known as Thromboangiitisobliterans Usually a disease of heavy cigarette smoker/tobacco user men, 25-40y/o Inflammatory arterial disorder that causes thrombus formation often extends to adjacent veins & nerves
  • 185. Affects medium-sized arteries (usually plantar & digital vessels in the foot or lower legs) unknown pathogenesis but it had been suggested that: tobacco may trigger an immune response or unmask a clotting defect; -> these 2 can incite an inflammatory reaction of the vessel wall
  • 186.
  • 187. Intermittent claudication in the arch of foot & digits
  • 188. Increased sensitivity to cold (due to impaired circulation
  • 189.
  • 190. Thick malformed nails (chronic ischemia)
  • 191.
  • 192.
  • 195. Precipitated by exposure to cold & strong emotions
  • 196.
  • 197. Return to normal color Note: although all of the fingers are affected symmetrically, only 1-2digits may be involved Severe cases: arthritis may arise (due to nutritional impairment) Brittle nails Thickening of the skin of fingertips Ulceration & superficial gangrene of fingers (rare occasions)
  • 198.
  • 199. Immersion of hand in cold water to initiate attack aids in the Dx
  • 200. Doppler flow velocimetry – used to quantify blood flow during temperature changes
  • 201.
  • 202. PRIORITIES: Abstinence in smoking & protection from cold
  • 203. Avoidance of emotional stress (anxiety & stress may precipitate vascular spasm)
  • 204. Meds: avoid vasoconstrictors (i.e.. Decongestants) -Calcium channel blockers (Diltiazem, Nifedipine, Nicardipine) – decrease episodes of attacks
  • 205. Care Plan for Clients with Altered Cardiovascular Oxygenation Goals: Relief of pain & symptoms Prevention of further cardiac damage Nursing Interventions: Pain control Proper medications Decrease client’s anxiety Health teachings (meds, activities, diet, exercise, etc) Assessment: Hx of symptoms (pain, esp. chest pain; palpitations; dyspnea) v/s Nursing Dx: ineffective tissue perfusion (cardiopulmonary) Impaired gas exchange Anxiety due to fear of death (clients with MI or Angina)