2. Chief complaint
⢠A 27 year old woman presented to the eye
OPD clinic with weakness in left side of
face for 2 weeks duration
She is a monk from Punakha dzongkhag
3. History of chief complaint
⢠2 weeks back she had a sudden onset of
left side facial weakness and pain in the
left ear which lasted for first three days
⢠She had difficulty in closing her left eye
and had on and off watery discharge. She
complains of mild drooling.
⢠No previous episode
⢠There was no history of trauma
4. History cont.
⢠Systemic review
⢠Past ocular history/ocular
medications/systemic medications/
comorbidities/allergies/family history
5. Examination
⢠Asymmetry of the face
⢠Absent wrinkling of left forehead
⢠Inability to close left eye(lagophthalmus)
⢠Mouth deviated to the right side
⢠Unable to puff out left cheek
6.
7.
8. Right eye Left eye
Visual acuity 6/6 6/6
With pinhole
Color vision Normal Normal
Extraocular movements Normal Normal
Bellâs phenomenon Present
Lids and adnexa Normal Normal
Schirmerâs test 15mm 15mm
Conjunctiva and sclera normal normal
Cornea clear Clear( sensation intact)
Anterior chamber Normal depth and quiet Normal depth and quiet
Iris and lens Normal Normal
Pupil Round regular and
reactive
Round regular and
reactive
Dilated fundus Vessel sheathing and
healed scars
Normal
9. Case summary
⢠A 27 year old female presented with
weakness in left side of face for 2 weeks
with difficulty in closing the left eye and left
earache for initial 3 days.neurological
examination showed left lower motor
neuron seventh nerve palsy. Bellâs
phenomenon and corneal sensation was
intact. Dilated funduscopy showed vessel
sheathing and healed scars in the left
fundus.
10. Problems
⢠Left lower motor neuron seventh nerve
palsy
⢠Healed right retinal vasculitis
17. Management
Corticosteroids
â˘Oral prednisolone 60mg daily for 7 days and tapered until 5mg daily
â˘Anti acid medications
â˘Early treatment is recommended especially within 3 days of symptoms of onset.
â˘Significantly reduced mild and moderate sequelae.
Facial physiotherapy
â˘To help in recovery of facial nerve function
â˘Prevents muscle atrophy and aids in full recovery if prognosis is good
18. Eye lubricants
â˘To prevent exposure keratopathy
â˘Depending upon the severity of keratopathy the frequency of lubricants is prescribed
ENT review
â˘Assessement was normal
â˘To rule out any pathology causing facial nerve palsy
22. Anatomy
⢠Motor fibers that innervate the facial
muscles
⢠Parasympathetic fibers innervating
lacrimal, submandibular, and
sublingual salivary glands
⢠Afferent fibers from taste receptors
from the anterior two thirds of the
tongue
⢠Somatic afferents from the external
auditory canal and pinna
⢠The nerve arises from two roots from
the pontomedullary junction and
enters the internal auditory meatus
23. â˘The facial (fallopian) canal= 33 mm
â˘labyrinthine, tympanic, and mastoid
â˘Narrowest in the labyrinthine segment
(average 0.68 mm in diameter)
â˘Facial nerve emerge at the stylomastoid
foramen and pass through the parotid
gland
â˘These fibers divide into five groups of
nerves between the deep and superficial
lobes of the gland
26. Bellâs palsy
⢠Acute peripheral facial nerve palsy of unknown cause
⢠Diagnosis of exclusion
Epidemiology
â˘The annual incidence rate =13 and 34 cases per 100,000 population
â˘Age=15-45 years age group
â˘No race, geographic, or gender predilection
â˘Risk is three times greater during pregnancy
27. Pathophysiology
⢠Herpes simplex virus activation is the
likely cause of Bell's palsy in most cases
⢠Inflammatory and possibly infectious
cause
⢠Nerve damage is maximal in the
labyrinthine part of the facial canal
28.
29. Trauma
⢠Second most common cause
⢠Most common is temporal bone
fractures(blunt and penetrating)
⢠Iatrogenic
48. Take home message
⢠Facial paralysis can be difficult to manage
⢠Should exclude other causes before
labelling it as idiopathic
⢠Multidisciplinary approach
⢠Ophthalmologist role: eye protection and
aesthetic improvement