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ECG BASICS
CHAMBER ENLARGEMENT AND
ELECTROLYTES ABNORMALITIES
DR MEHUL RATHOD (R3 Medicine)
• The 12 conventional ECG leads record the difference in potential
between electrodes placed on the surface of the body.
• These leads are divided into two groups:
• Six limb (extremity) leads and six chest (precordial) leads.
• The limb leads record potentials transmitted onto the frontal plane,
and the chest leads record potentials transmitted onto the horizontal
plane.
• The six chest leads are unipolar recordings obtained by electrodes in the
following positions;
• lead V1, fourth intercostal space, just to the right of the sternum;
• lead V2, fourth intercostal space, just to the left of the sternum;
• lead V3, midway between V2 and V4:
• Lead V4, midclavicular line, fifth intercostal space;
• and lead V5, anterior axillary line, same level as V4;
• and lead V6, midaxillary line, same level as V4 and V5.
• smallest (1 mm) horizontal divisons correspond to 0.04 (40 ms), with
heavier lines at intervals of 0.20 s (200 ms).
READING 12-LEAD ECGS
• The best way to read 12-lead ECGs is to develop a step-by-step approach
(just as we did for analyzing a rhythm strip). In these modules, we present
a seven-step approach:
• Calculate RATE
• Determine RHYTHM
• Determine QRS AXIS
• Check individual WAVES
• Calculate INTERVALS
• Assess for HYPERTROPHY
• Look for evidence of infarction/dyselectrolytemia.
Step 1: Determining the Heart Rate
• Rule of 300
Count the number of “big boxes” between two QRS complexes, and
divide this into 300 for regular rhythms.
• Second Rule ECGs record 6 seconds [30 boxes] of rhythm per page
Count the number of beats present on the ECG in 6 seconds Multiply
by 10 This is useful for irregular rhythms
300/ 3 = 100 beats/min
Step 2: Determine Regularity
• Look at the R-R distances (using a caliper or markings on a pen or
paper). Regular (are they equidistant apart)? Occasionally irregular?
Regularly irregular? Irregularly irregular?
Step 3: Determining the Axis
• Normal QRS axis from −30° to +110°.
• −30° to −90° is referred to as a left axis deviation (LAD).
• +110° to +180° is referred to as a right axis deviation (RAD)
• −180° to −90° is referred as north-west axis/extreme axis/axis in no man’s land
Step 4: Check Individual Waves
P WAVE
• Always positive in lead I and II
• Always negative in lead aVR
• <2.5 small squares in duration
• <2.5 small squares in amplitude
• Commonly biphasic in lead V1
• Best seen in leads II
• Tall (>2.5 mm), pointed P waves (P pulmonale)—suggests right atrial
enlargement
• Seen in chronic obstructive pulmonary disease (COPD), atrial septal
defect (ASD), TS, Ebstein anomaly (Himalayan P waves)
• Notched/bifid (“M” shaped) P wave (P “mitrale”) in limb leads—
suggests left atrial enlargement Seen in MS, MR, and systemic
hypertension
• Absent P waves—atrial fibrillation/flutter
QRS-Complex
• Normal characteristics:
• Duration: 0.04–0.11 seconds.
Broad/wide QRS (>0.12 s)- 3 small boxes
• Ventricular hypertrophy
• Intraventricular conduction disturbance
• Aberrant ventricular conduction
• Ventricular pre-excitation
• Ventricular ectopic or escape pacemaker
• Ventricular pacing by cardiac pacemaker
Decreased—low voltage QRS (<5 mV in limb
leads/<10 mV in chest leads)
• Obese patient
• Restrictive cardiomyopathy
• Pericardial effusion
• Hypothyroidism
• Hypothermia
• Myocarditis
Q Waves
• The normal Q wave in lead I is due to septal depolarization
• It is small in amplitude—less than 25% of the succeeding R wave, or
less than 3 mm
• Its duration is <0.04 sec or one small box
• THE PATHOLOGICAL Q WAVE
• It is deep in amplitude—more than 25% of the succeeding R wave, or
more than 4 mm. Its
• duration is >0.04 sec or >1 small box
• Pathological Q waves may be seen in Infarction, cardiomyopathies—
hypertrophic obstructive cardiomyopathy (HOCM), infiltrative
myocardial disease
T Wave
U Waves
• The U wave is not always seen. It is typically small, and, by definition,
follows the T wave. U waves are thought to represent repolarization
of the papillary muscles or Purkinje fibers
Prominent U waves are most often seen in
• Hypokalemia
• Hypercalcemia
• thyrotoxicosis, or exposure to digitalis, epinephrine
• in intracranial hemorrhage.
Assess for Hypertrophy
Right Ventricular Hypertrophy (RVH)
• Criteria of RVH
• Tall R in V1 with R >S, or R/S ratio >1
• Deep S waves in V4, V5, and V6
• Associated right axis deviation, right atrial enlargement (RAE)
Cause of RVH
• Long-standing mitral stenosis
• Pulmonary hypertension of any cause
• Ventricular septal defect (VSD) or atrial septal defect (ASD) with initial
L to R shunt
• Congenital heart with RV over load
• tricuspid regurgitation, pulmonary stenosis.
Left Ventricular Hypertrophy (LVH)
• Criteria of LVH
• High QRS voltages in limb leads:
• Sokolow and Lyon criteria: S (V1) + R (V5 or V6) >35 mm
• Cornell criteria: S (V3) + R (aVL) >28 mm (men) or >20 mm (women)
• Others: R (aVL) >13 mm.
Causes of LVH
• Pressure overload—systemic hypertension and aortic stenosis
• Volume overload—AR or MR-dilated cardiomyopathy
• Ventricular septal defect—cause both right and left ventricular
volume overload
• Hypertrophic cardiomyopathy.
ECGs
QRS < 5mm in all limb leads
R in V6 (3)+ S in V1 (5) = 8
Prominent U wave
P wave >2.5 mm
Tall peaked T wave
• Reaching pattern
Tall T wave
THANK YOU

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Presentation on basics of ECG............

  • 1. ECG BASICS CHAMBER ENLARGEMENT AND ELECTROLYTES ABNORMALITIES DR MEHUL RATHOD (R3 Medicine)
  • 2. • The 12 conventional ECG leads record the difference in potential between electrodes placed on the surface of the body. • These leads are divided into two groups: • Six limb (extremity) leads and six chest (precordial) leads. • The limb leads record potentials transmitted onto the frontal plane, and the chest leads record potentials transmitted onto the horizontal plane.
  • 3. • The six chest leads are unipolar recordings obtained by electrodes in the following positions; • lead V1, fourth intercostal space, just to the right of the sternum; • lead V2, fourth intercostal space, just to the left of the sternum; • lead V3, midway between V2 and V4: • Lead V4, midclavicular line, fifth intercostal space; • and lead V5, anterior axillary line, same level as V4; • and lead V6, midaxillary line, same level as V4 and V5.
  • 4.
  • 5.
  • 6. • smallest (1 mm) horizontal divisons correspond to 0.04 (40 ms), with heavier lines at intervals of 0.20 s (200 ms).
  • 7.
  • 8. READING 12-LEAD ECGS • The best way to read 12-lead ECGs is to develop a step-by-step approach (just as we did for analyzing a rhythm strip). In these modules, we present a seven-step approach: • Calculate RATE • Determine RHYTHM • Determine QRS AXIS • Check individual WAVES • Calculate INTERVALS • Assess for HYPERTROPHY • Look for evidence of infarction/dyselectrolytemia.
  • 9. Step 1: Determining the Heart Rate • Rule of 300 Count the number of “big boxes” between two QRS complexes, and divide this into 300 for regular rhythms. • Second Rule ECGs record 6 seconds [30 boxes] of rhythm per page Count the number of beats present on the ECG in 6 seconds Multiply by 10 This is useful for irregular rhythms
  • 10. 300/ 3 = 100 beats/min
  • 11.
  • 12. Step 2: Determine Regularity • Look at the R-R distances (using a caliper or markings on a pen or paper). Regular (are they equidistant apart)? Occasionally irregular? Regularly irregular? Irregularly irregular?
  • 13. Step 3: Determining the Axis • Normal QRS axis from −30° to +110°. • −30° to −90° is referred to as a left axis deviation (LAD). • +110° to +180° is referred to as a right axis deviation (RAD) • −180° to −90° is referred as north-west axis/extreme axis/axis in no man’s land
  • 14.
  • 15.
  • 16. Step 4: Check Individual Waves P WAVE • Always positive in lead I and II • Always negative in lead aVR • <2.5 small squares in duration • <2.5 small squares in amplitude • Commonly biphasic in lead V1
  • 17. • Best seen in leads II • Tall (>2.5 mm), pointed P waves (P pulmonale)—suggests right atrial enlargement • Seen in chronic obstructive pulmonary disease (COPD), atrial septal defect (ASD), TS, Ebstein anomaly (Himalayan P waves) • Notched/bifid (“M” shaped) P wave (P “mitrale”) in limb leads— suggests left atrial enlargement Seen in MS, MR, and systemic hypertension • Absent P waves—atrial fibrillation/flutter
  • 18.
  • 19. QRS-Complex • Normal characteristics: • Duration: 0.04–0.11 seconds. Broad/wide QRS (>0.12 s)- 3 small boxes • Ventricular hypertrophy • Intraventricular conduction disturbance • Aberrant ventricular conduction • Ventricular pre-excitation • Ventricular ectopic or escape pacemaker • Ventricular pacing by cardiac pacemaker
  • 20. Decreased—low voltage QRS (<5 mV in limb leads/<10 mV in chest leads) • Obese patient • Restrictive cardiomyopathy • Pericardial effusion • Hypothyroidism • Hypothermia • Myocarditis
  • 21. Q Waves • The normal Q wave in lead I is due to septal depolarization • It is small in amplitude—less than 25% of the succeeding R wave, or less than 3 mm • Its duration is <0.04 sec or one small box
  • 22. • THE PATHOLOGICAL Q WAVE • It is deep in amplitude—more than 25% of the succeeding R wave, or more than 4 mm. Its • duration is >0.04 sec or >1 small box • Pathological Q waves may be seen in Infarction, cardiomyopathies— hypertrophic obstructive cardiomyopathy (HOCM), infiltrative myocardial disease
  • 24. U Waves • The U wave is not always seen. It is typically small, and, by definition, follows the T wave. U waves are thought to represent repolarization of the papillary muscles or Purkinje fibers Prominent U waves are most often seen in • Hypokalemia • Hypercalcemia • thyrotoxicosis, or exposure to digitalis, epinephrine • in intracranial hemorrhage.
  • 25.
  • 26. Assess for Hypertrophy Right Ventricular Hypertrophy (RVH) • Criteria of RVH • Tall R in V1 with R >S, or R/S ratio >1 • Deep S waves in V4, V5, and V6 • Associated right axis deviation, right atrial enlargement (RAE)
  • 27. Cause of RVH • Long-standing mitral stenosis • Pulmonary hypertension of any cause • Ventricular septal defect (VSD) or atrial septal defect (ASD) with initial L to R shunt • Congenital heart with RV over load • tricuspid regurgitation, pulmonary stenosis.
  • 28. Left Ventricular Hypertrophy (LVH) • Criteria of LVH • High QRS voltages in limb leads: • Sokolow and Lyon criteria: S (V1) + R (V5 or V6) >35 mm • Cornell criteria: S (V3) + R (aVL) >28 mm (men) or >20 mm (women) • Others: R (aVL) >13 mm.
  • 29. Causes of LVH • Pressure overload—systemic hypertension and aortic stenosis • Volume overload—AR or MR-dilated cardiomyopathy • Ventricular septal defect—cause both right and left ventricular volume overload • Hypertrophic cardiomyopathy.
  • 30. ECGs
  • 31.
  • 32.
  • 33. QRS < 5mm in all limb leads
  • 34.
  • 35. R in V6 (3)+ S in V1 (5) = 8
  • 36.
  • 38.
  • 40.
  • 42.
  • 44.
  • 46.