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Dr Wan Zuraini Bt Mahrawi, MD, GCFM
MSc in Wound Healing & Tissue Repair (Cardiff, UK)
LUMUT WOUND CARE CONFERENCE 2018
CHRONIC WOUND
MANAGEMENT
WOUND IN PRIMARY CARE
C H R O N I C W O U N D
INTRODUCTION
01
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LUMUT WOUND CARE CONFERENCE
2018
CONFERENCE PORTFOLIO
DIABETIC
FOOT
ULCER
VENOUS
LEG ULCER
PRESSURE
INJURY
ARTERIAL
ISCHEMIC
ULCER
SURGICAL
SITE
INFECTION
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CHRONIC WOUND
wounds do not progress normally through the stages
of healing (often getting ‘stalled’ in one phase) and do
not show evidence of healing within four weeks
– Laurie Swezey
Disclaimer : This presentation contain intermediate level of information of wound medicine
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CHRONIC WOUND MOLECULAR ENVIRONMENT
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WRONG DIAGNOSIS
INADEQUATE WBP /
INFECTION / INFLAMMATION
IMPROPER TREATMENT
LACK OF PATIENT
CENTERED MANAGEMENT
ErratumCHRONIC WOUND MANAGEMENT
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Answer 3
From healthcare
personnel involve directly
in wound clinic (HEALTH
CLINIC)
Answer 4
Answer from the speaker
Answer 1
From healthcare
personnel not involve
directly in wound clinic
(HOSPITAL)
Answer 2
From healthcare personnel
involve directly in wound
clinic (HOSPITAL)
ENGAGEMENT TESTQUESTION: PLEASE OUTLINE THIS WOUND MANAGEMENT
PLAN
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01 03
02 04
CONFIRM AETIOLOGY
IDENTIFY SYSTEMIC FACTOR
EXECUTE BASIC TREATMENT
ADD ON SPECIFIC
TREATMENT
PATIENT’S EDUCATION &
FOLLOW UP
Management Strategy
CHRONIC WOUND MANAGEMENT
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SPECIFIC
SYSTEMIC
BASIC
01
03
3 Steps of StrategyCHRONIC WOUND MANAGEMENT
C H R O N I C W O U N D M A N A G E M E N T
SYSTEMIC FACTORS
02
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DIABETES
INFECTION
SYSTEMIC FACTORSTHE GENESIS OF DELAY HEALING
Intracellular
hyperglycemia
Prolonged
inflammatory
cytokines
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DRUGS
NUTRITION
HYPOXIA
SMOKING
Glucocorticoid,
NSAIDs,
Chemo drugs
Vitamin CEA, PUFAs, Protein
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OBESITY
ANEMIA
STRESS
DIMINISHED
CARDIAC
OUTPUT
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MALIGNANCY
NON-COMPLIANCE
C H R O N I C W O U N D
BASIC MANAGEMENT
03
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Treatment
Dressing selection
Wound Bed Preparation
Assessment and DIME Approach
Establish Goal Of Treatment
Prognosis of healing
Education
To the patient and carer
02
01
04
03
Basic Management
CHRONIC WOUND
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Sufficient resources?
Treatable underlying cause?
Ability of healing?
Palliative
wound?
01 Establish Goal of TreatmentCHRONIC WOUND
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Review behavior that
works against
desired outcome
Motivation for
healing potential /
modify feelings
Overview signs and
symptoms of
complications & how
to report
Reason for chosen
plan of care
Patient’s role in this
plan of care
Discuss plan of care
5 64
2 31
02 Educate Patient & CaretakerCHRONIC WOUND
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DEBRIDEMENT
MOISTURE BALANCEINFECTION / INFLAMMATION
EDGE EFFECT
03 Wound Bed PreparationCHRONIC WOUND – DIME APPROACH
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NON-SELECTIVE
Large wounds
Extensive necrotic tissue
Non-surgical candidate
SELECTIVE
Minor to moderate debridement
Painful wound
Non-surgical candidate
EXCISIONAL
Wounds of varying size and depth
Unhealthy wound edges
Heavily necrotic wound
DebridementCHRONIC WOUND
(MECHANICAL) (AUTOLYTIC, ENZYMATIC, BIOLOGICAL)
(SHARP, SURGICAL)
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Infection vs InflammationCHRONIC WOUND
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01 02 03 04 05
Ischemia with
ABI < 0.4,
TcPo2 < 30
mmHg
Inability to
comply with
treatment
Significant
change over 24
– 48hr
Severe change
in wound pain
Foot infection
with T 38C or <
36C, HR > 90,
RR > 20, SBC >
12000 or < 4000
CRITERIA FOR REFERRALCHRONIC WOUND
06 07 08 09 10
Exposed organ
not previously
exposed
Wound bed
discoloration to
black
Worsening
redness around
the wound
Deteriorating
flap or graft
Worsening foul
smell wound
Stimulate inflammation
2
No establish link between
slough amount and biofilm3
Extracellular Polymeric matrix
1
About BiofilmCHRONIC WOUND
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DECREASE HEALING
TIME
COST EFFECTIVE
IMPROVE PATIENT’S QUALITY OF
LIFE
04 Treatment
DRESSING SELECTION
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C H R O N I C W O U N D
SPECIFIC MANAGEMENT
04
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DIABETIC FOOT
ULCER
VENOUS LEG
ULCER
PRESSURE
ULCER
ARTERIAL
ULCER
Specific ManagementCHRONIC WOUND
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Offloading footwear
Callous
removal
Educate patient on
footwear
Regular
assessment
Control
diabetes
Recommendations for follow-up care :
DIABETIC FOOT ULCERSPECIFIC MANAGEMENT
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Long term application of
compression stockings
Consider referral for
surgical ablation / injection
Reduce pressure in the
venous system of the LL
Control obesity and
exercise
VENOUS LEG ULCERSPECIFIC MANAGEMENT
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Compression TherapyVenous Leg Ulcer
ABI Bandage Sub-
bandage
pressure
> 0.8 4-layer 35 – 40
0.7 2-layer 17 – 25
0.6 2-layer 17 – 25
< 0.5 Only with
medical
supervision
-
ABI: Ankle brachial index
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Repositioning Support surfaces
Heel offloading
devices
Silicone foam and
dressing to prevent
pressure and shear
Increase protein
intake and calories
Prevent and treat
incontinence
associated dermatitis
PRESSURE INJURYSPECIFIC MANAGEMENT
Pressure Ulcer Risk Assessment Scale
(PURAS)
NORTON SCALE
(England 1962, 5
parameters) – physical
condition, mental state,
activity, mobility,
incontinence
WATERLOW SCALE
(England 1984, 8
parameters) – BMI, skin
visual, sex, age,
continence, mobility,
appetite, medication
BRADEN SCALE (USA
1987, 6 subscales) –
sensory, perception,
mobility, activity,
nutrition, friction & shear
GOSNELL SCALE
(USA 1973, 5
parameters) – mental
status, continence,
mobility, activity,
nutrition
RAMSTADIUS TOOL
(AUSTRALIA 2000, 2
questions) – skin
integrity, mobility
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01
02
03
04
Early referral to vascular interventionist
(ABSI < 0.8, TcPO2 < 30mmHg)
Lower extremity protection and
assessment
Exercise and smoking
cessation
Ensure adequate flow to the
extremity
ARTERIAL ULCERSPECIFIC MANAGEMENT
C H R O N I C W O U N D
WOUND MANAGEMENT
PRIMARY CARE05
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22897
wounds
49%
ACUTE
51%
CHRONIC
Wound Outcome Data 2017PKD KUALA LANGAT, SELANGOR
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19%
81%
INFECTED
NON-INFECTED
Wound Outcome Data 2017PKD KUALA LANGAT, SELANGOR
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73% 7% 6% 4%
DIABETIC RELATED
WOUND
VENOUS LEG
ULCER
PRESSURE
INJURY
ATYPICAL
WOUND
Chronic Wound by Aetiology
PKD KUALA LANGAT, SELANGOR
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MANAGEMENT
STRATEGY
Short term and long term goals
KNOWLEDGE
Of different type of wound
SUPPORTIVE
THERAPY
Systemic therapy, topical and
adjunctive treatment in wound care
PRE-ELIMINARY REQUIREMENT
C H R O N I C W O U N D M A N A G E M E N T
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CHLORHEXIDINE
01 HYDROGEN
PEROXIDE02 KMNO4
03
PVP - IODINE
04 ACETIC ACID
05 SILVER
SULFADIAZINE06
THE OLD ARMY – What’s Common in Our
Setting?
S H A H A L A M H O S P I T A L W O U N D & O S T O M Y C O N F E R E N C E
2 0 1 8
4%, 5% 3% 1 : 1000/5000
1%0.25%, 0.5%7 - 10%
ALCOHOL
07 METHYLATED
SPIRIT08 ACRIFLAVINE
SOLUTION09 0.1%96%70%
*Full presentation is accessible @ www.slideshare.net, key word: wan zuraini, the old army
C H R O N I C W O U N D
HOME CARE WOUND CARE
5.1
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SKOP
PERKHIDMA
TAN DCS
• Perkhidmatan perawatan termasuk
latihan teknik mencegah berlakunya kudis
tekanan, pendidikan mengenai rawatan
luka dan penjagaan kebersihan, menukar
tiub nasogastrik, kateter urethral,
pemeriksaan darah ujian gula darah dan
mengambil tekanan darah dan
penyediaan sokongan emosi.
1. Perawatan
• Aktiviti rehabilitasi bagi kes yang
memerlukan termasuk rehabilitasi
pergerakan aktif dan pasif serta latihan
activity of daily living.
2. Rehabilitasi
• Perkhidmatan ini merangkumi perawatan
yang melibatkan penjagaan paliatif.
3. Paliatif
DOMICILIARY CARE
SERVICES
C H R O N I C W O U N D
CONCLUSION
06
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Focus Specific Treatment
Knowledge
Construction
Determine
Aetiology
Checking Systemic
Factor, Execute Basic
Treatment
Educate
Follow-up
Plan
“A kind gesture can reach a wound that only compassion can heal….”
CONCLUSIONCHRONIC WOUND MANAGEMENT
THIS PRESENTATION IS
ACCESSIBLE FREELY
THROUGH:
C H R O N I C W O U N D M A N A G E M E N T
www.slideshare.ne
t
Key term: wan Zuraini, wound, wound
wound, wound dressing
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THANK YOU
Kuala Langat Menerajui Transformasi Perubatan Luka
D R W A N Z U R A I N I | m r s . w a n z u @ g m a i l . c o m

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Chronic Wound Management Strategy

Editor's Notes

  1. 1. Minta list nama 2. chill as a teacher….3. isi kan soalan pada kertas 4. introduce yourself
  2. 1. Combine 2 in 1 presentation 2. dilemma question
  3. Clump all management into 1 ..regardless any etiology and to reduce clumsiness
  4. To change the image behind the Mock up. Select the layer - > Right Click -> Send to Back -> Delete the image -> Drag & Drop your Own Picture -> Send to Back (again)
  5. Intrcellular Hyperglycemia – aldose reductase – increase sorbitol from glucose – abnormal management of matrix protein (collagen) bacteria and endotoxins can lead to the prolonged elevation of pro-inflammatory cytokines such as interleukin-1 (IL-1) and TNF-α and elongate the inflammatory phase. increased level of matrix metalloproteases (MMPs), degrade ECM
  6. Supression fibroblast proliferation and collagen synthesis – poor granulation, hypoxia-inducible factor-1 (HIF-1) Chemo drugs decrease RNA DNA – fibroplasia neutropenia, anemia, and thrombocytopenia, - impede inflammation
  7. If you want to change the picture background. 1) Select the shape (in front of the image) and send it to back 2) Delete the image and drag and drop you own // 3) Send to back the new image