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RESPIRATORY MEDICINE
History taking in respiratory system
Dr .suresh kumar yogi
PG student
Dr. S. N. Medical Collage
Jodhpur
 History is a record of medical events that have taken
palace in patient’s life.
 Importance of good history lies in the fact that it provide
an insight into the exact nature of the patients problems
and the attitude of patient towards the illness.
 The examiner should first allow the patient to state the
nature of his complaints and narrate his problems in his
own language.
 Listening o the patient account helps not only in
establishing a rapport but also helps the examiner for
further enquiries into the history
 Leading questions must be avoided .
 End of history ,elicit and record negative history .
 In case the patient is unable to speak ,unconscious ,in
shock or a child ,the history should be taken from the
nearst relative/friend who is known as informant
 Examiners approch should be gentle ,
sympathetic and caring.
Particulars of the patients
 Name
 Age
 Sex
 Address
 Occupation
 Contect number
 Date of admission and date of examination
Presenting complaints;
 Complaints for which the patient has come
to the doctor.
 Define and record the main complaints and
the duration of the presence of complaints.
 Duration of complaints may be recorded in
days, weeks ,months or years.
 Complaints should be recorded In
chronological order.
History of presenting complaints;-
 Recording of details of each complaint
separately
 Mode of onset
 duration
 Location / site
 Severity
 Character
 Diurnal variation
 Aggravating and relieving factors
 Exacerbation or remission
The main respiratory symptoms are:
 Cough and sputum
 Haemoptysis
 Dyspnoea .
 Chest pain
 Wheeze .
Other associate symptums
 Fever ;-the persistent elevation of body
temperature above the normal levels
 Normal -37.0 to 37.5 c
 Type of fever-
 Continuous Fever-The temperature
remains above normal throughout the day
and does not fluctuate more than 1 degree
Celsius in 24 hours. This type of fever occurs
in lobar pneumonia, typhoid, urinary tract
infection, infective endocarditis, brucellosis,
typhus etc
Remittent Fever-The temperature remains above
normal throughout the day and fluctuates more than
2 degree Celsius in 24 hours. This type of fever is
usually seen patients of typhoid infection
and infective endocarditis. This type of fever is most
common in practice.
 Intermittent Fever-The temperature is present only
for some hours in a day and remains to normal for
the remaining hours. When the spike occurs daily, it
is quotidian, when every alternate day, it tertian and
when every third day, it is quartan. Intermittent fever
is seen in malaria, kala-azar, pyemia, septicemia etc.
 Anorexia –TB/Bronchogenic ca
 Headace-co2 narcosis
 Edema -corpulmonale
 Loss of appetite is a common feature
whenever people are unwell. It suggests
that the disease is having a significant
effect on well-being.
 Significant loss of weight may well be
indicative of serious illness - eg,
malignancy or tuberculosis.
 Upper gastrointestinal symptoms: gastro-
oesophageal reflux is a common cause of chronic
cough.
 Heart disease may cause respiratory symptoms.
Establish whether there are any indications of heart
failure or coronary heart disease.
 Severe anaemia may cause breathlessness.
 Rheumatoid arthritis and other connective tissue
diseases may cause respiratory symptoms.
 Neuromuscular diseases may cause respiratory
symptoms, particularly dyspnoea.
Cough
 Definition-forced expiration against
closed glottis ,which clears the tracheo-
bronchial tree of any foreign material or
secretion.
it is protective reflex
it can be voluntary or involuntary being
provoked by physical, chemical,or
mechanical stimuli irritating of any part
of the respiratory tract and even from
irritation of the pleura.
Analysis of cough
 Was the onset of the cough sudden or
insidious? What was its initiating event?
Did it start as an isolated symptom or occur
with or follow other symptoms?
 How long has the cough been present? Is it
persistent or episodic? Seasonal or
perennial?
 To determine the severity of the cough,
ask: How frequent is a coughing spell?
How long does each coughing spell last?
What is its effect on daily activity or rest?
 Is the cough productive or dry? Lack of
expectoration does not necessarily
indicate that the cough is dry, as many
patients, particularly children and
women, tend to swallow their sputum
raised to the level of the pharynx. The
sound of the cough would help in
determining its productive or dry nature.
 Is the cough the only symptom, or is it
associated with other respiratory or
nonrespiratory symptoms? Inquiry should
be specifically made about conditions
known to cause the cough, particularly
when it is chronic and persistent.
 In addition to an accurate smoking history ,
ask: To what respiratory irritants is the
patient exposed at home or at work? Is
exposure accidental or intentional?
 What are the precipitating or aggravating
factors? What time of the day or night is
the cough or sputum production worse?
Does it happen in supine position, upon
arising in the morning, with drinking or
eating, with exercise, or with breathing cold
or dry air? Does the cough awaken the
patient from sleep?
 Is there a past history (recent or old) of
foreign body aspiration?
 Has the pattern of the cough and the
amount or other characteristics of the
sputum changed recently?
 Can the patient locate the site of origin of
the cough or the sputum, such as from the
throat or deeper in the chest?
 Has the patient had a similar problem with
coughing in the past?
 Does the cough have easily recognizable
characteristics, as in croup or whooping
cough?
Clinical Significance
 Onset;-
sudden –acute asthama ,massive pulmonary
embolism, Pneumothorax
Gradual-in chronic bronchitis
 Nature;-
( a) Dry cough- in pharyngitis ,allergic bronchitis
( b ) productive cough-in lower respiratory tract
infection like pneumonia , Bronchitis
,Bronchictasis, PTB
 Duration;-
short –in acute Bronchitis, pharyngitis
Long –COPD,PTB ,allergic
bronchitis,Fibrosing alveolits,
 Diurnal variation;
Early morning & night- chronic bronchitis,
Bronchictasis.
Cough disturbing patient sleep in night-
pulmonary oedema, asthma
 Postural variatons;-
Cough In bronchiectasis and lung absess
when the patient lies in the opposite side
of lesion ,this becouse the postural
drainge of the mucus into the healthy
bronchi stimulats cough reflex
sputum
 Sputum is mucus produced from
respiratory tract
 Normal lung produces about 100 ml of
clear sputum each day
 Enqury - amount , Appearance /colour,
type of sputum
 Patients with sputum production should be
asked about
 its frequency and description of the
physical characteristics of the sputum
including
Amount (with each coughing spell and
daily total),
color,
consistency,
ease of its expectoration,
taste, and smell.
Clinical Significance
 Bronchiectasis causes large volumes of
purulent sputum ,which varies with posture.
 Suddenly coughing up large amount of
purulent sputum on a single occasion
suggests rupture of a lung abscess or
empyema in to the bronchial tree.
 Large volumes of watery sputum with a
pink tinge in acutely breathless patient
suggest pulmonary oedema,
 But if occurring over weeks ,suggests
alveolar cell carcinoma
type appearance causes
serous Frothy,pink
Clear,watery/rarely
copious(bronchorrhea)
Acute pulmonary
oedema
Bronchioloalveolar
cancer
Mucoid Clear , grey
White,viscid
Chronic
bronchitis/COPD
asthma
purulent Yellow
Green
Acute
bronchopulmonary
infection
Asthma (eosinophil)
Longer –standing
infection
Pnemonia
Bronchiectasis,cystic
fibrosis,lung abscess
Rusty Rusty red Pnemococcal
pneumonia
Type of sputum
Appearance /colour;-
 Clear or mucoid sputum –in chronic bronchitis
and COPD with no infection
 Yellow sputum- in acute lower respiratory tract
infection(live neutrophils) and in
asthma(eosinophils).
 Green purulent –indicates chronic
infection(dead neutophils)e.g.in COPD or
bronchiectasis
 Rusty red sputum –early pneumococcal
pneumonia,as pneumonic inflammation
causes lysis of red cells.
Haemoptysis
 Coughing up blood ,ranging from blood
streak sputum to gross blood that
originates from the tracheobronchial tree
 Haemoptysis immediattely preceded by
cough indicates origin of bleeding at a
level lower than the larynx
 Most common cause of haemoptysis in
india is TB
Causes of haemoptysis;-
 Respiratory – TB, bronchogenic ca, pulmonary
embolism, lung abscess ,bronchiectasis,
bronchial adenoma, trauma
 Cardiac –mitral stenosis,aortic
aneurysm,primary pulmonary hypertension
 Immunological- good pasture’s syndrome,
wegener’s granulomatosis and PAN
 Itarogenic- lung biopsy, bronchoscopy
 Bleeding disorder-thrombocytopenic
purpura,hemophilia
 Spurious –trauma,ulcer in mouth
Amount and appearance
 Blood –streaked clear sputum or clots in
sputum for more than a week suggest lung
cancer
 Haemoptysis with purulent sputum suggests
infection
 Coughing up large amount of pure blood is
rare but potentially life threatening ;
causes –lung cancer, bronchiectasis , TB
,lung abscess ,mycetoma,Cystic fibrosis,
aortobronchial fistula, granulomatosis wth
polyangitis
Duration and frequency
 Single episodes of hemoptysis ,if
associated with symptoms e.g.pleuritic
chest pain and breathlessness, suggest
pulmonary thromboembolism and infarction
and need immediate investigation
 Bronchiectasis causes intermittent
hemoptysis associated with copious
sputum over years
 Daily hemoptysis for a week or more –in
lung cancer ,other TB and lung abscess.
Breathlessness(Dyspnoea)
Defination;-An abnormal, uncomfortable
subject awareness of once breathing
 Dyspnea (also SOB, air hunger)
 normal in heavy exertion
 pathological if it occurs in unexpected
situations.
 Breathlessness is common symtom of
cardiac disease. however it also present
in respiratory disease
Analysis of breathlessness-
 Occurrence
Rest
Exertion (quantify)
 Position
Orthopnea (dyspnea lying flat)
Trepopnea (dyspnea in lateral position)
Platypnea (dyspnea when upright)
Other precipitating factors
 Environment Emotional state
 Chronology
Duration
Progression
Diurnal and seasonal variations
Constant or intermittent
 Relieving factors
Rest
Medications (physician and self-
prescribed)
 Predisposing factors
Cigarette smoking
Occupational and environmental exposures
 Associated medical diseases and
symptoms
Pulmonary
Cardiac
Neuromuscular
Family history
 Breathlessness occurs as a symptom
most frequently in lung cancer, where it
might affect 75% of people with primary
disease of the lung, bronchus and
trachea (Muers & Round 1993).
 For patients with COPD, intractable
breathlessness develops late in the
course of the disease, gradually
increasing in severity over a period of
years in the majority of people.
Onset and durestion
Severity grading-
Clinical Significance
Causes of breathlessnss
1. Physiological ;–exercises, high altitude,etc
2. Pathological;-
(a) Cardiac –
acute-pulmonary edema, pulmonary embolism, CHD
in neonates
chronic-CCF, acyanotic CHD, chronic
thromoembolism.
(b) Respiratory –
acute- pneumothorax, acute severe asthma, inhaled
foreign body, acute exacerbation of COPD,
pneumonic consolidation, laryngeal edema etc
chronic-COPD, Asthma, pleural effusion(subacute),
bronchogenic Ca, PTB, ILD eg fibrosing alveolitis,
etc
3.Pharmacological ; drugs beta-blockers,
busulfan etc
4.Psycological - hypervantilation
5.Miscellaneous ;-
acute- metabolic acidosis & respiratory
muscle paralysis
chronic-anemia ,ankylosing spondylitis
Wheeze and
stridor
 High pitched whistling sound
 Mainly expiratory
 Usually indicative of bronchospasm
 Wheeze on exercise is common in
asthma and COPD
 Night wakening with wheeze suggestive
of asthma or PND
 But wheeze after wakening in the
morning suggests COPD
Stridor ;-high pitched ,often harsh noise
produced by airflow turbulence through a
partial obstruction of the upper airway.
 Most commonly on inspiration but also on
expiration or biphasically
 Inspiratory-narrowing at vocal cords
 Biphasic –s/o tracheal obstraction
 Expiratory-trachiobronchial obstruction
 Usually accompanied by dyspnoea
 Stridor is serious condition always need
investigation
Analysis of wheeze
 Onset
 Duration and periodicity of wheeze
 Diurnal and seasonal variations
 Duration ;-short duration suggests obstraction
by foreign,functional disturbance of larynx or
inflammatory disease
 Onset ;-
(a) instantaneous onset of dypnoea and stridor
suggests foreigne body impaction or
neuromuscular disturbance of larynx
(b)Acute onset is typical of laryngitis, laryngismus
stridulus, measles, whooping cough or
diphtheria
(c)Slow and gradual onset suggests papilloma or
recurrnt laryngeal nerve
Chest pain
if pain is a symptom, clarify the details of the pain
using SOCRATES
 Site – where is the pain ?
To differentiate from cardic,gastrointestinal. Respiratory
chest pain is usually laterlized
 Onset – when did it start?
sudden -spontaneous pneumothorax
gradual-pluerisy
 Character/type –
sharp /stabing-related to deep breathing and coughing in
pleuritis ,usually subsides after pleural effusion develops
dull ache –lateral/low down in spontaneous pneumothorax
 Radiation – does the pain move anywhere else?
 Associations – other symptoms associated with the
pain
 Time course – worsening / improving / fluctuating /
time of day dependent
 Exacerbating / Relieving factors – does anything
make the pain worse or better?
Costo chondial pain is aggravated by movement
To differentiate from cardiac causes angina is
aggravated by stress and relieved by rest while
pleural pain is aggravated on deep breathing and
coughing.
 Severity – on a scale of 0-10, how severe is the
pain?
 Common respiratory causes of chest pain-
Pleurisy
Pneumothorax
Acute pulmonary embolism
Pneumonia
Massive collapse
Lung absess
Ca lung
Tracheitis
Pneumomediastinum
Past history;-
 H/O PTB and treatment ATT ,DM, HTN, Asthama,blood
transfusion,
 H/o previous hospital admission
 In case of COPD /ASTHMA number of episode of acute
exacerbation
 Use of inhalers (assess compliance and technique).
 Use of steroids (some measure of severity in asthma).
 Other drugs which may have relevance in respiratory
disease - eg, angiotensin-converting enzyme (ACE)
inhibitors (cough).
 Allergies
 Ask about all allergies including, for example, food,
inhaled allergens and drugs.
Occupational and social history
 An occupational history may be very important in
respiratory disease. Occupational Asthma , Industrial
Dust Diseases,Asbestos-related Diseases,Extrinsic
Allergic Alveolitis and Sick Building Syndrome
 Hobbies and pets may also be responsible for respiratory
disease like extrinsic allergic alveolitis .
 Lifestyle and alcohol consumption are also very relevant
to respiratory diseases. Ask about illicit drugs.
 Smoking history should detail, for example, the type and
number of cigarettes smoked currently and in the past.
Ask also about passive smoking.
 Sexual history may be relevant to risk of HIV and AIDS.
Family history;-
 Respiratory diseases with a genetic
component - eg, cystic fibrosis,
emphysema (alpha-1-antitrypsin
deficiency).
 Infectious diseases such as tuberculosis
(remember high-risk groups).
 Atopic diseases such as asthma, hay
fever and eczema.
Thank
you

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History taking in respiratory system

  • 1. RESPIRATORY MEDICINE History taking in respiratory system Dr .suresh kumar yogi PG student Dr. S. N. Medical Collage Jodhpur
  • 2.  History is a record of medical events that have taken palace in patient’s life.  Importance of good history lies in the fact that it provide an insight into the exact nature of the patients problems and the attitude of patient towards the illness.  The examiner should first allow the patient to state the nature of his complaints and narrate his problems in his own language.  Listening o the patient account helps not only in establishing a rapport but also helps the examiner for further enquiries into the history  Leading questions must be avoided .  End of history ,elicit and record negative history .  In case the patient is unable to speak ,unconscious ,in shock or a child ,the history should be taken from the nearst relative/friend who is known as informant
  • 3.  Examiners approch should be gentle , sympathetic and caring. Particulars of the patients  Name  Age  Sex  Address  Occupation  Contect number  Date of admission and date of examination
  • 4. Presenting complaints;  Complaints for which the patient has come to the doctor.  Define and record the main complaints and the duration of the presence of complaints.  Duration of complaints may be recorded in days, weeks ,months or years.  Complaints should be recorded In chronological order.
  • 5. History of presenting complaints;-  Recording of details of each complaint separately  Mode of onset  duration  Location / site  Severity  Character  Diurnal variation  Aggravating and relieving factors  Exacerbation or remission
  • 6. The main respiratory symptoms are:  Cough and sputum  Haemoptysis  Dyspnoea .  Chest pain  Wheeze .
  • 7. Other associate symptums  Fever ;-the persistent elevation of body temperature above the normal levels  Normal -37.0 to 37.5 c  Type of fever-  Continuous Fever-The temperature remains above normal throughout the day and does not fluctuate more than 1 degree Celsius in 24 hours. This type of fever occurs in lobar pneumonia, typhoid, urinary tract infection, infective endocarditis, brucellosis, typhus etc
  • 8. Remittent Fever-The temperature remains above normal throughout the day and fluctuates more than 2 degree Celsius in 24 hours. This type of fever is usually seen patients of typhoid infection and infective endocarditis. This type of fever is most common in practice.  Intermittent Fever-The temperature is present only for some hours in a day and remains to normal for the remaining hours. When the spike occurs daily, it is quotidian, when every alternate day, it tertian and when every third day, it is quartan. Intermittent fever is seen in malaria, kala-azar, pyemia, septicemia etc.
  • 9.  Anorexia –TB/Bronchogenic ca  Headace-co2 narcosis  Edema -corpulmonale  Loss of appetite is a common feature whenever people are unwell. It suggests that the disease is having a significant effect on well-being.  Significant loss of weight may well be indicative of serious illness - eg, malignancy or tuberculosis.
  • 10.  Upper gastrointestinal symptoms: gastro- oesophageal reflux is a common cause of chronic cough.  Heart disease may cause respiratory symptoms. Establish whether there are any indications of heart failure or coronary heart disease.  Severe anaemia may cause breathlessness.  Rheumatoid arthritis and other connective tissue diseases may cause respiratory symptoms.  Neuromuscular diseases may cause respiratory symptoms, particularly dyspnoea.
  • 11. Cough  Definition-forced expiration against closed glottis ,which clears the tracheo- bronchial tree of any foreign material or secretion. it is protective reflex it can be voluntary or involuntary being provoked by physical, chemical,or mechanical stimuli irritating of any part of the respiratory tract and even from irritation of the pleura.
  • 12. Analysis of cough  Was the onset of the cough sudden or insidious? What was its initiating event? Did it start as an isolated symptom or occur with or follow other symptoms?  How long has the cough been present? Is it persistent or episodic? Seasonal or perennial?  To determine the severity of the cough, ask: How frequent is a coughing spell? How long does each coughing spell last? What is its effect on daily activity or rest?
  • 13.  Is the cough productive or dry? Lack of expectoration does not necessarily indicate that the cough is dry, as many patients, particularly children and women, tend to swallow their sputum raised to the level of the pharynx. The sound of the cough would help in determining its productive or dry nature.
  • 14.  Is the cough the only symptom, or is it associated with other respiratory or nonrespiratory symptoms? Inquiry should be specifically made about conditions known to cause the cough, particularly when it is chronic and persistent.  In addition to an accurate smoking history , ask: To what respiratory irritants is the patient exposed at home or at work? Is exposure accidental or intentional?
  • 15.  What are the precipitating or aggravating factors? What time of the day or night is the cough or sputum production worse? Does it happen in supine position, upon arising in the morning, with drinking or eating, with exercise, or with breathing cold or dry air? Does the cough awaken the patient from sleep?  Is there a past history (recent or old) of foreign body aspiration?
  • 16.  Has the pattern of the cough and the amount or other characteristics of the sputum changed recently?  Can the patient locate the site of origin of the cough or the sputum, such as from the throat or deeper in the chest?  Has the patient had a similar problem with coughing in the past?  Does the cough have easily recognizable characteristics, as in croup or whooping cough?
  • 17. Clinical Significance  Onset;- sudden –acute asthama ,massive pulmonary embolism, Pneumothorax Gradual-in chronic bronchitis  Nature;- ( a) Dry cough- in pharyngitis ,allergic bronchitis ( b ) productive cough-in lower respiratory tract infection like pneumonia , Bronchitis ,Bronchictasis, PTB
  • 18.  Duration;- short –in acute Bronchitis, pharyngitis Long –COPD,PTB ,allergic bronchitis,Fibrosing alveolits,  Diurnal variation; Early morning & night- chronic bronchitis, Bronchictasis. Cough disturbing patient sleep in night- pulmonary oedema, asthma
  • 19.  Postural variatons;- Cough In bronchiectasis and lung absess when the patient lies in the opposite side of lesion ,this becouse the postural drainge of the mucus into the healthy bronchi stimulats cough reflex
  • 20. sputum  Sputum is mucus produced from respiratory tract  Normal lung produces about 100 ml of clear sputum each day  Enqury - amount , Appearance /colour, type of sputum
  • 21.  Patients with sputum production should be asked about  its frequency and description of the physical characteristics of the sputum including Amount (with each coughing spell and daily total), color, consistency, ease of its expectoration, taste, and smell.
  • 22. Clinical Significance  Bronchiectasis causes large volumes of purulent sputum ,which varies with posture.  Suddenly coughing up large amount of purulent sputum on a single occasion suggests rupture of a lung abscess or empyema in to the bronchial tree.  Large volumes of watery sputum with a pink tinge in acutely breathless patient suggest pulmonary oedema,  But if occurring over weeks ,suggests alveolar cell carcinoma
  • 23. type appearance causes serous Frothy,pink Clear,watery/rarely copious(bronchorrhea) Acute pulmonary oedema Bronchioloalveolar cancer Mucoid Clear , grey White,viscid Chronic bronchitis/COPD asthma purulent Yellow Green Acute bronchopulmonary infection Asthma (eosinophil) Longer –standing infection Pnemonia Bronchiectasis,cystic fibrosis,lung abscess Rusty Rusty red Pnemococcal pneumonia Type of sputum
  • 24. Appearance /colour;-  Clear or mucoid sputum –in chronic bronchitis and COPD with no infection  Yellow sputum- in acute lower respiratory tract infection(live neutrophils) and in asthma(eosinophils).  Green purulent –indicates chronic infection(dead neutophils)e.g.in COPD or bronchiectasis  Rusty red sputum –early pneumococcal pneumonia,as pneumonic inflammation causes lysis of red cells.
  • 25. Haemoptysis  Coughing up blood ,ranging from blood streak sputum to gross blood that originates from the tracheobronchial tree  Haemoptysis immediattely preceded by cough indicates origin of bleeding at a level lower than the larynx  Most common cause of haemoptysis in india is TB
  • 26. Causes of haemoptysis;-  Respiratory – TB, bronchogenic ca, pulmonary embolism, lung abscess ,bronchiectasis, bronchial adenoma, trauma  Cardiac –mitral stenosis,aortic aneurysm,primary pulmonary hypertension  Immunological- good pasture’s syndrome, wegener’s granulomatosis and PAN  Itarogenic- lung biopsy, bronchoscopy  Bleeding disorder-thrombocytopenic purpura,hemophilia  Spurious –trauma,ulcer in mouth
  • 27. Amount and appearance  Blood –streaked clear sputum or clots in sputum for more than a week suggest lung cancer  Haemoptysis with purulent sputum suggests infection  Coughing up large amount of pure blood is rare but potentially life threatening ; causes –lung cancer, bronchiectasis , TB ,lung abscess ,mycetoma,Cystic fibrosis, aortobronchial fistula, granulomatosis wth polyangitis
  • 28. Duration and frequency  Single episodes of hemoptysis ,if associated with symptoms e.g.pleuritic chest pain and breathlessness, suggest pulmonary thromboembolism and infarction and need immediate investigation  Bronchiectasis causes intermittent hemoptysis associated with copious sputum over years  Daily hemoptysis for a week or more –in lung cancer ,other TB and lung abscess.
  • 29. Breathlessness(Dyspnoea) Defination;-An abnormal, uncomfortable subject awareness of once breathing  Dyspnea (also SOB, air hunger)  normal in heavy exertion  pathological if it occurs in unexpected situations.  Breathlessness is common symtom of cardiac disease. however it also present in respiratory disease
  • 30. Analysis of breathlessness-  Occurrence Rest Exertion (quantify)  Position Orthopnea (dyspnea lying flat) Trepopnea (dyspnea in lateral position) Platypnea (dyspnea when upright) Other precipitating factors  Environment Emotional state
  • 31.  Chronology Duration Progression Diurnal and seasonal variations Constant or intermittent  Relieving factors Rest Medications (physician and self- prescribed)
  • 32.  Predisposing factors Cigarette smoking Occupational and environmental exposures  Associated medical diseases and symptoms Pulmonary Cardiac Neuromuscular Family history
  • 33.  Breathlessness occurs as a symptom most frequently in lung cancer, where it might affect 75% of people with primary disease of the lung, bronchus and trachea (Muers & Round 1993).  For patients with COPD, intractable breathlessness develops late in the course of the disease, gradually increasing in severity over a period of years in the majority of people.
  • 35.
  • 37. Clinical Significance Causes of breathlessnss 1. Physiological ;–exercises, high altitude,etc 2. Pathological;- (a) Cardiac – acute-pulmonary edema, pulmonary embolism, CHD in neonates chronic-CCF, acyanotic CHD, chronic thromoembolism. (b) Respiratory – acute- pneumothorax, acute severe asthma, inhaled foreign body, acute exacerbation of COPD, pneumonic consolidation, laryngeal edema etc chronic-COPD, Asthma, pleural effusion(subacute), bronchogenic Ca, PTB, ILD eg fibrosing alveolitis, etc
  • 38. 3.Pharmacological ; drugs beta-blockers, busulfan etc 4.Psycological - hypervantilation 5.Miscellaneous ;- acute- metabolic acidosis & respiratory muscle paralysis chronic-anemia ,ankylosing spondylitis
  • 39. Wheeze and stridor  High pitched whistling sound  Mainly expiratory  Usually indicative of bronchospasm  Wheeze on exercise is common in asthma and COPD  Night wakening with wheeze suggestive of asthma or PND  But wheeze after wakening in the morning suggests COPD
  • 40. Stridor ;-high pitched ,often harsh noise produced by airflow turbulence through a partial obstruction of the upper airway.  Most commonly on inspiration but also on expiration or biphasically  Inspiratory-narrowing at vocal cords  Biphasic –s/o tracheal obstraction  Expiratory-trachiobronchial obstruction  Usually accompanied by dyspnoea  Stridor is serious condition always need investigation
  • 41. Analysis of wheeze  Onset  Duration and periodicity of wheeze  Diurnal and seasonal variations
  • 42.  Duration ;-short duration suggests obstraction by foreign,functional disturbance of larynx or inflammatory disease  Onset ;- (a) instantaneous onset of dypnoea and stridor suggests foreigne body impaction or neuromuscular disturbance of larynx (b)Acute onset is typical of laryngitis, laryngismus stridulus, measles, whooping cough or diphtheria (c)Slow and gradual onset suggests papilloma or recurrnt laryngeal nerve
  • 43. Chest pain if pain is a symptom, clarify the details of the pain using SOCRATES  Site – where is the pain ? To differentiate from cardic,gastrointestinal. Respiratory chest pain is usually laterlized  Onset – when did it start? sudden -spontaneous pneumothorax gradual-pluerisy  Character/type – sharp /stabing-related to deep breathing and coughing in pleuritis ,usually subsides after pleural effusion develops dull ache –lateral/low down in spontaneous pneumothorax  Radiation – does the pain move anywhere else?
  • 44.  Associations – other symptoms associated with the pain  Time course – worsening / improving / fluctuating / time of day dependent  Exacerbating / Relieving factors – does anything make the pain worse or better? Costo chondial pain is aggravated by movement To differentiate from cardiac causes angina is aggravated by stress and relieved by rest while pleural pain is aggravated on deep breathing and coughing.  Severity – on a scale of 0-10, how severe is the pain?
  • 45.  Common respiratory causes of chest pain- Pleurisy Pneumothorax Acute pulmonary embolism Pneumonia Massive collapse Lung absess Ca lung Tracheitis Pneumomediastinum
  • 46. Past history;-  H/O PTB and treatment ATT ,DM, HTN, Asthama,blood transfusion,  H/o previous hospital admission  In case of COPD /ASTHMA number of episode of acute exacerbation  Use of inhalers (assess compliance and technique).  Use of steroids (some measure of severity in asthma).  Other drugs which may have relevance in respiratory disease - eg, angiotensin-converting enzyme (ACE) inhibitors (cough).  Allergies  Ask about all allergies including, for example, food, inhaled allergens and drugs.
  • 47. Occupational and social history  An occupational history may be very important in respiratory disease. Occupational Asthma , Industrial Dust Diseases,Asbestos-related Diseases,Extrinsic Allergic Alveolitis and Sick Building Syndrome  Hobbies and pets may also be responsible for respiratory disease like extrinsic allergic alveolitis .  Lifestyle and alcohol consumption are also very relevant to respiratory diseases. Ask about illicit drugs.  Smoking history should detail, for example, the type and number of cigarettes smoked currently and in the past. Ask also about passive smoking.  Sexual history may be relevant to risk of HIV and AIDS.
  • 48. Family history;-  Respiratory diseases with a genetic component - eg, cystic fibrosis, emphysema (alpha-1-antitrypsin deficiency).  Infectious diseases such as tuberculosis (remember high-risk groups).  Atopic diseases such as asthma, hay fever and eczema.